2023 - Research.com Genetics in Denmark Leader Award
2022 - Research.com Genetics and Molecular Biology in Denmark Leader Award
2010 - Fellow of the Royal Society, United Kingdom
Member of the European Molecular Biology Organization (EMBO)
Ian D. Hickson spends much of his time researching Genetics, Helicase, DNA, Molecular biology and RecQ helicase. As part of his studies on Genetics, Ian D. Hickson often connects relevant subjects like Cell biology. His Cell biology study which covers Chromosomal fragile site that intersects with Sister chromatids, Chromatin and Chromosome segregation.
His work in the fields of Bloom syndrome overlaps with other areas such as Premature aging. His work carried out in the field of DNA brings together such families of science as Peptide sequence, Genome, Gene and Phosphorylation. Ian D. Hickson interconnects Base excision repair, AP endonuclease, DNA- lyase, AP site and Biochemistry in the investigation of issues within Molecular biology.
His main research concerns Genetics, Molecular biology, DNA, Helicase and DNA repair. His work on Genetics deals in particular with DNA replication, Homologous recombination, RecQ helicase, Genome instability and Gene. His Molecular biology research includes themes of Cell culture, Cell, Gene expression, DNA damage and Topoisomerase.
His research in DNA tackles topics such as Cell biology which are related to areas like Chromatin. His study in the field of Bloom syndrome and Topoisomerase III is also linked to topics like Premature aging. His DNA repair research is multidisciplinary, incorporating elements of Mutation, Cancer research and Radiosensitivity.
Ian D. Hickson focuses on Cell biology, Genetics, DNA replication, DNA and Mitosis. The various areas that Ian D. Hickson examines in his Cell biology study include DNA damage, Genome instability, Helicase, Molecular biology and DNA repair. His Helicase study deals with Spindle apparatus intersecting with Nucleic acid thermodynamics.
The concepts of his DNA replication study are interwoven with issues in Telomere, Replication protein A and DNA synthesis. His work in DNA tackles topics such as Gene which are related to areas like Structure–activity relationship. His studies in Mitosis integrate themes in fields like Cytokinesis, Chromosome segregation, Chromatin, Anaphase and Sister chromatids.
Ian D. Hickson spends much of his time researching Cell biology, Genetics, Molecular biology, Helicase and Mitosis. His Cell biology research incorporates themes from Non-homologous end joining, Genome, DNA, Genome instability and Protein structure. His Molecular biology research incorporates elements of Replication protein A and Chromosome instability.
His study in Helicase is interdisciplinary in nature, drawing from both Holliday junction, DNA repair and Gene expression. His DNA repair research integrates issues from G-quadruplex, DNA damage and Homologous recombination. His Mitosis study incorporates themes from Chromatid, Sister chromatids, Chromosome segregation and Anaphase.
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The Bloom's syndrome helicase suppresses crossing over during homologous recombination
Leonard Wu;Ian D. Hickson.
Nature (2003)
RecQ helicases: caretakers of the genome.
Ian D. Hickson.
Nature Reviews Cancer (2003)
Cellular Responses to DNA Damage
Chris J Norbury;Ian D Hickson.
Annual Review of Pharmacology and Toxicology (2001)
The Bloom’s and Werner’s syndrome proteins are DNA structure-specific helicases
Payam Mohaghegh;Julia K. Karow;Robert M. Brosh;Vilhelm A. Bohr.
Nucleic Acids Research (2001)
The Bloom’s Syndrome Helicase Unwinds G4 DNA
Hui Sun;Julia K. Karow;Ian D. Hickson;Nancy Maizels.
Journal of Biological Chemistry (1998)
XRCC1 coordinates the initial and late stages of DNA abasic site repair through protein–protein interactions
Antonio E. Vidal;Serge Boiteux;Ian D. Hickson;J. Pablo Radicella.
The EMBO Journal (2001)
Replication stress induces sister-chromatid bridging at fragile site loci in mitosis
Kok Lung Chan;Timea Palmai-Pallag;Songmin Ying;Ian D. Hickson.
Nature Cell Biology (2009)
53BP1 nuclear bodies form around DNA lesions generated by mitotic transmission of chromosomes under replication stress.
Claudia Lukas;Velibor Savic;Simon Bekker-Jensen;Carsten Doil.
Nature Cell Biology (2011)
The Bloom's syndrome gene product promotes branch migration of Holliday junctions
Julia K. Karow;Angelos Constantinou;Ji-Liang Li;Stephen C. West.
Proceedings of the National Academy of Sciences of the United States of America (2000)
Sgs1: A eukaryotic homolog of E. coil RecQ that interacts with topoisomerase II in vivo and is required for faithful chromosome segregation
Paul M Watt;Edward J Louis;Rhona H Borts;Ian D Hickson.
web science (1995)
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