2023 - Research.com Molecular Biology in United States Leader Award
His primary areas of study are DNA repair, Molecular biology, DNA damage, DNA and Mitochondrion. His research in DNA repair intersects with topics in Mitochondrial DNA and Cell biology. His work carried out in the field of Cell biology brings together such families of science as Mitophagy and NAD+ kinase.
His Molecular biology research is multidisciplinary, incorporating perspectives in Base excision repair, DNA glycosylase, DNA mismatch repair, Nucleotide excision repair and Gene. In his research, Mutagenesis is intimately related to Oxidative stress, which falls under the overarching field of DNA damage. He interconnects Autophagy, Neurodegeneration and Neuroscience in the investigation of issues within Mitochondrion.
The scientist’s investigation covers issues in DNA repair, Molecular biology, DNA damage, DNA and Cell biology. His study in the field of Nucleotide excision repair and Base excision repair is also linked to topics like Premature aging. The concepts of his Molecular biology study are interwoven with issues in Cell culture, Chinese hamster ovary cell, AP site, Pyrimidine dimer and Gene.
His DNA damage research incorporates elements of Oxidative stress, Cancer research, Poly ADP ribose polymerase and Senescence. His Cell biology study incorporates themes from Neurodegeneration and NAD+ kinase. His Mitochondrial DNA research integrates issues from Mitochondrial DNA repair and Mitochondrion.
Vilhelm A. Bohr mainly investigates Cell biology, DNA repair, Mitochondrion, DNA damage and Mitophagy. His study in Cell biology is interdisciplinary in nature, drawing from both Biochemistry, DNA, Neurodegeneration, Werner syndrome and NAD+ kinase. His work on Base excision repair and Nucleotide excision repair as part of general DNA repair study is frequently linked to Premature aging, therefore connecting diverse disciplines of science.
In his study, Oxidative phosphorylation is strongly linked to Oxidative stress, which falls under the umbrella field of Mitochondrion. The study incorporates disciplines such as Cancer research, Molecular biology, Poly ADP ribose polymerase, Gene and Nuclear DNA in addition to DNA damage. His Mitophagy research includes elements of Caenorhabditis elegans, Genome instability and Disease.
His primary scientific interests are in Cell biology, DNA repair, Mitophagy, Mitochondrion and DNA damage. His work carried out in the field of Cell biology brings together such families of science as Homologous recombination, Exonuclease, Werner syndrome, Telomere and NAD+ kinase. His research integrates issues of Genome instability and Mitochondrial DNA in his study of DNA repair.
His Mitophagy study combines topics from a wide range of disciplines, such as Disease and Caenorhabditis elegans. His research in DNA damage intersects with topics in Apoptosis, Nicotinamide riboside, Gene, SIRT3 and Nuclear DNA. His DNA research is multidisciplinary, relying on both Protein structure, Molecular biology and Nucleic acid secondary structure.
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Metformin improves healthspan and lifespan in mice
Alejandro Martin-Montalvo;Evi M. Mercken;Sarah J. Mitchell;Sarah J. Mitchell;Sarah J. Mitchell;Hector H. Palacios.
Nature Communications (2013)
SIRT6 is a histone H3 lysine 9 deacetylase that modulates telomeric chromatin
Eriko Michishita;Ronald A. McCord;Elisabeth Berber;Mitomu Kioi.
Nutrient-Sensitive Mitochondrial NAD+ Levels Dictate Cell Survival
Hongying Yang;Tianle Yang;Joseph A. Baur;Evelyn Perez.
Ageing as a risk factor for neurodegenerative disease.
Yujun Hou;Xiuli Dan;Mansi Babbar;Yong Wei.
Nature Reviews Neurology (2019)
p53 modulation of TFIIH-associated nucleotide excision repair activity.
Xin Wei Wang;H. Yeh;L. Schaeffer;Richard Roy.
Nature Genetics (1995)
REPAIR OF OXIDATIVE DAMAGE TO NUCLEAR AND MITOCHONDRIAL DNA IN MAMMALIAN CELLS
Deborah L. Croteau;Vilhelm A. Bohr.
Journal of Biological Chemistry (1997)
Base excision repair of oxidative DNA damage and association with cancer and aging.
Scott Maynard;Shepherd H. Schurman;Charlotte Harboe;Nadja C. de Souza-Pinto.
Mitophagy inhibits amyloid-β and tau pathology and reverses cognitive deficits in models of Alzheimer’s disease
E F Fang;Y Hou;K Palikaras;B A Adriaanse.
Nature Neuroscience (2019)
Preferential DNA repair of an active gene in human cells
Isabel Mellon;Vilhelm A. Bohr;Charles Allen Smith;Philip C. Hanawalt.
Proceedings of the National Academy of Sciences of the United States of America (1986)
Defective Mitophagy in XPA via PARP-1 Hyperactivation and NAD+/SIRT1 Reduction
Evandro Fei Fang;Morten Scheibye-Knudsen;Lear E. Brace;Henok Kassahun.
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