H-Index & Metrics Top Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Medicine H-index 206 Citations 137,351 710 World Ranking 49 National Ranking 36

Research.com Recognitions

Awards & Achievements

2010 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Enzyme

Mark P. Mattson mainly focuses on Internal medicine, Endocrinology, Cell biology, Neuroscience and Oxidative stress. In his research, Growth cone is intimately related to Neuron, which falls under the overarching field of Internal medicine. His work carried out in the field of Endocrinology brings together such families of science as Ryanodine receptor, Genetically modified mouse and Neurodegeneration.

He interconnects Excitotoxicity, Apoptosis and Programmed cell death in the investigation of issues within Cell biology. His Neuroscience research is multidisciplinary, incorporating perspectives in Glutamate receptor, Neurotrophic factors and Alzheimer's disease. His Oxidative stress study incorporates themes from Molecular biology, Ceramide, Homocysteine and Reactive oxygen species.

His most cited work include:

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • Triple-Transgenic Model of Alzheimer's Disease with Plaques and Tangles: Intracellular Aβ and Synaptic Dysfunction (2978 citations)
  • Pathways towards and away from Alzheimer's disease (2339 citations)

What are the main themes of his work throughout his whole career to date?

Mark P. Mattson spends much of his time researching Neuroscience, Internal medicine, Cell biology, Endocrinology and Glutamate receptor. His Neuroscience research is multidisciplinary, incorporating elements of Synaptic plasticity and Neurotrophic factors. His studies in Neurotrophic factors integrate themes in fields like Neurogenesis and Neurotrophin.

His Cell biology research includes themes of Apoptosis, Biochemistry and Programmed cell death. The study incorporates disciplines such as Alzheimer's disease, Calcium, Neuroprotection and Amyloid in addition to Endocrinology. His Glutamate receptor study integrates concerns from other disciplines, such as NMDA receptor, Neurotoxicity and Neuron.

He most often published in these fields:

  • Neuroscience (41.24%)
  • Internal medicine (42.89%)
  • Cell biology (40.91%)

What were the highlights of his more recent work (between 2013-2021)?

  • Neuroscience (41.24%)
  • Internal medicine (42.89%)
  • Endocrinology (42.56%)

In recent papers he was focusing on the following fields of study:

Mark P. Mattson focuses on Neuroscience, Internal medicine, Endocrinology, Cell biology and Disease. His Neuroscience study deals with Synaptic plasticity intersecting with Long-term potentiation. Specifically, his work in Endocrinology is concerned with the study of Oxidative stress.

Mark P. Mattson studies Mitochondrion which is a part of Cell biology. As a member of one scientific family, Mark P. Mattson mostly works in the field of Mitochondrion, focusing on Neurodegeneration and, on occasion, Neuroprotection. As a part of the same scientific study, he usually deals with the Neuroplasticity, concentrating on Neurogenesis and frequently concerns with Neural stem cell and Glutamate receptor.

Between 2013 and 2021, his most popular works were:

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • Fasting: Molecular Mechanisms and Clinical Applications (556 citations)
  • Fasting: Molecular Mechanisms and Clinical Applications (556 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Internal medicine

The scientist’s investigation covers issues in Neuroscience, Cell biology, Disease, Mitophagy and Internal medicine. His Neuroscience research is multidisciplinary, relying on both Synaptic plasticity, Bioenergetics and Neurodegeneration. Mark P. Mattson has included themes like Cell, Neuroinflammation, DNA repair and NAD+ kinase in his Cell biology study.

His Mitophagy study also includes

  • Mitochondrion that connect with fields like Excitotoxicity,
  • Caenorhabditis elegans and related Nicotinamide-nucleotide adenylyltransferase and Drosophila melanogaster. His studies deal with areas such as Endocrinology and Transcription factor as well as Internal medicine. His Neuroplasticity study which covers Neurogenesis that intersects with Glutamate receptor.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Top Publications

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

6803 Citations

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

4781 Citations

Triple-Transgenic Model of Alzheimer's Disease with Plaques and Tangles: Intracellular Aβ and Synaptic Dysfunction

Salvatore Oddo;Antonella Caccamo;Jason D. Shepherd;M. Paul Murphy.
Neuron (2003)

3934 Citations

Pathways towards and away from Alzheimer's disease

Mark P. Mattson.
Nature (2004)

3437 Citations

beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity

Mark P. Mattson;Bin Cheng;Dave Davis;Karin Bryant.
The Journal of Neuroscience (1992)

1996 Citations

Apoptosis in neurodegenerative disorders.

Mark P. Mattson.
Nature Reviews Molecular Cell Biology (2000)

1787 Citations

Self-Propagating, Molecular-Level Polymorphism in Alzheimer's {beta}-Amyloid Fibrils

Aneta T. Petkova;Richard D. Leapman;Zhihong Guo;Wai Ming Yau.
Science (2005)

1729 Citations

A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease.

K Hensley;J M Carney;M P Mattson;M Aksenova.
Proceedings of the National Academy of Sciences of the United States of America (1994)

1429 Citations

Cellular actions of beta-amyloid precursor protein and its soluble and fibrillogenic derivatives

Mark P. Mattson.
Physiological Reviews (1997)

1306 Citations

Mitochondrial Manganese Superoxide Dismutase Prevents Neural Apoptosis and Reduces Ischemic Brain Injury: Suppression of Peroxynitrite Production, Lipid Peroxidation, and Mitochondrial Dysfunction

Jeffrey N. Keller;Mark S. Kindy;Fredrick W. Holtsberg;Daret K. St. Clair.
The Journal of Neuroscience (1998)

1274 Citations

Profile was last updated on December 6th, 2021.
Research.com Ranking is based on data retrieved from the Microsoft Academic Graph (MAG).
The ranking h-index is inferred from publications deemed to belong to the considered discipline.

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