D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 135 Citations 56,504 520 World Ranking 232 National Ranking 166

Overview

What is he best known for?

The fields of study he is best known for:

  • Enzyme
  • Gene
  • Biochemistry

His primary areas of investigation include Oxidative stress, Biochemistry, Alzheimer's disease, Protein oxidation and Lipid peroxidation. D. Allan Butterfield has researched Oxidative stress in several fields, including Neurotoxicity, Proteomics, Neurodegeneration, Neuroscience and Amyloid. Biochemistry is closely attributed to Cell biology in his research.

His study in Alzheimer's disease is interdisciplinary in nature, drawing from both Endocrinology and Pathogenesis. His work carried out in the field of Protein oxidation brings together such families of science as Protein Carbonyl Content, Lipid oxidation and Vitamin E. His research investigates the connection between Lipid peroxidation and topics such as Pharmacology that intersect with issues in Superoxide dismutase.

His most cited work include:

  • Evidence of oxidative damage in Alzheimer's disease brain: central role for amyloid β-peptide (944 citations)
  • Nitric oxide in the central nervous system: neuroprotection versus neurotoxicity (875 citations)
  • Lipid peroxidation and protein oxidation in Alzheimer's disease brain: Potential causes and consequences involving amyloid β-peptide-associated free radical oxidative stress (794 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Oxidative stress, Biochemistry, Alzheimer's disease, Protein oxidation and Internal medicine. D. Allan Butterfield studies Oxidative stress, namely Lipid peroxidation. His study looks at the relationship between Biochemistry and topics such as Neurotoxicity, which overlap with Methionine and Peptide.

His research in Alzheimer's disease focuses on subjects like Neuroscience, which are connected to Disease. His work in Protein oxidation tackles topics such as Pharmacology which are related to areas like Doxorubicin. Within one scientific family, D. Allan Butterfield focuses on topics pertaining to Endocrinology under Internal medicine, and may sometimes address concerns connected to Biliverdin reductase.

He most often published in these fields:

  • Oxidative stress (53.42%)
  • Biochemistry (43.62%)
  • Alzheimer's disease (29.02%)

What were the highlights of his more recent work (between 2015-2021)?

  • Oxidative stress (53.42%)
  • Alzheimer's disease (29.02%)
  • Neuroscience (12.75%)

In recent papers he was focusing on the following fields of study:

D. Allan Butterfield focuses on Oxidative stress, Alzheimer's disease, Neuroscience, Cell biology and Endocrinology. His Oxidative stress study results in a more complete grasp of Biochemistry. The various areas that D. Allan Butterfield examines in his Alzheimer's disease study include Neuropathology, PI3K/AKT/mTOR pathway and Neurodegeneration.

D. Allan Butterfield combines subjects such as Signal transduction, Disease and Pathogenesis with his study of Neuroscience. His Endocrinology research is multidisciplinary, relying on both Internal medicine and Biliverdin reductase. His studies in Protein oxidation integrate themes in fields like Proteome and Gel electrophoresis.

Between 2015 and 2021, his most popular works were:

  • Oxidative stress, dysfunctional glucose metabolism and Alzheimer disease (258 citations)
  • Oxidative stress, protein modification and Alzheimer disease. (142 citations)
  • Oxidative Stress, Amyloid-β Peptide, and Altered Key Molecular Pathways in the Pathogenesis and Progression of Alzheimer's Disease. (102 citations)

In his most recent research, the most cited papers focused on:

  • Enzyme
  • Gene
  • Internal medicine

D. Allan Butterfield mostly deals with Alzheimer's disease, Oxidative stress, Neuroscience, Biochemistry and Senile plaques. His Alzheimer's disease study integrates concerns from other disciplines, such as Neuropathology, PI3K/AKT/mTOR pathway, Neurodegeneration and Cognitive decline. His Oxidative stress research includes elements of Mitochondrion, Cell biology and Pathogenesis.

D. Allan Butterfield interconnects Neurotoxicity, Disease and Cytokine in the investigation of issues within Neuroscience. His research on Biochemistry frequently connects to adjacent areas such as Foot shock. His work deals with themes such as Proteostasis, Proteomics and Oxidative phosphorylation, which intersect with Lipid peroxidation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Evidence of oxidative damage in Alzheimer's disease brain: central role for amyloid β-peptide

D.Allan Butterfield;Jennifer Drake;Chava Pocernich;Alessandra Castegna.
Trends in Molecular Medicine (2001)

1473 Citations

Nitric oxide in the central nervous system: neuroprotection versus neurotoxicity

Vittorio Calabrese;Cesare Mancuso;Menotti Calvani;Enrico Rizzarelli.
Nature Reviews Neuroscience (2007)

1316 Citations

Lipid peroxidation and protein oxidation in Alzheimer's disease brain: Potential causes and consequences involving amyloid β-peptide-associated free radical oxidative stress

D.Allan Butterfield;Christopher M. Lauderback.
Free Radical Biology and Medicine (2002)

1089 Citations

Brain Regional Correspondence Between Alzheimer's Disease Histopathology and Biomarkers of Protein Oxidation

Kenneth Hensley;Nathan Hall;Ramachandran Subramaniam;Pamela Cole.
Journal of Neurochemistry (2002)

1070 Citations

Amyloid β-peptide (1-42)-induced Oxidative Stress and Neurotoxicity: Implications for Neurodegeneration in Alzheimer's Disease Brain. A Review

D Allan Butterfield.
Free Radical Research (2002)

1009 Citations

Review: Alzheimer's amyloid beta-peptide-associated free radical oxidative stress and neurotoxicity.

Sridhar Varadarajan;Servet Yatin;Marina Aksenova;D.Allan Butterfield.
Journal of Structural Biology (2000)

972 Citations

Evidence that amyloid beta-peptide-induced lipid peroxidation and its sequelae in Alzheimer's disease brain contribute to neuronal death

D Allan Butterfield;Alessandra Castegna;Christopher M Lauderback;Jennifer Drake.
Neurobiology of Aging (2002)

880 Citations

Proteomic identification of oxidatively modified proteins in Alzheimer's disease brain. Part I: creatine kinase BB, glutamine synthase, and ubiquitin carboxy-terminal hydrolase L-1.

Alessandra Castegna;Michael Aksenov;Marina Aksenova;Visith Thongboonkerd.
Free Radical Biology and Medicine (2002)

683 Citations

Proteomic identification of nitrated proteins in Alzheimer's disease brain.

Alessandra Castegna;Visith Thongboonkerd;Jon B. Klein;Bert Lynn.
Journal of Neurochemistry (2003)

625 Citations

Roles of amyloid β-peptide-associated oxidative stress and brain protein modifications in the pathogenesis of Alzheimer's disease and mild cognitive impairment

D. Allan Butterfield;Tanea Reed;Shelley F. Newman;Rukhsana Sultana.
Free Radical Biology and Medicine (2007)

613 Citations

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