His main research concerns Oxidative stress, Mitochondrion, Reactive oxygen species, Superoxide dismutase and Molecular biology. His work on Lipid peroxidation and Protein oxidation as part of general Oxidative stress research is frequently linked to PIN1, bridging the gap between disciplines. His research integrates issues of Oxidative phosphorylation, Axotomy, Programmed cell death and Premovement neuronal activity in his study of Mitochondrion.
His study in Programmed cell death is interdisciplinary in nature, drawing from both Autophagy, Autophagosome, Cytochrome c and Central nervous system. His study focuses on the intersection of Reactive oxygen species and fields such as Glutathione with connections in the field of Neurotoxicity. His work deals with themes such as CAAT box, Promoter and Transcription factor, Gene, Intron, which intersect with Molecular biology.
Daret K. St. Clair focuses on Oxidative stress, Molecular biology, Mitochondrion, Reactive oxygen species and Cancer research. In his study, Pharmacology, Cardiotoxicity and Toxicity is strongly linked to Doxorubicin, which falls under the umbrella field of Oxidative stress. His Molecular biology study incorporates themes from Superoxide dismutase, Apoptosis and Transcription factor, Gene, Transfection.
His Mitochondrion study integrates concerns from other disciplines, such as Oxidative phosphorylation, Signal transduction and Chromosomal translocation. He has researched Reactive oxygen species in several fields, including DNA damage, Superoxide and Cytosol. In Cancer research, Daret K. St. Clair works on issues like Prostate cancer, which are connected to RELB.
His primary scientific interests are in Oxidative stress, Cell biology, Reactive oxygen species, Cancer research and Mitochondrion. He combines subjects such as Apoptosis, Glutathione, Immunology and Doxorubicin with his study of Oxidative stress. His research in Cell biology intersects with topics in Autophagy and Cell growth.
His Autophagy research incorporates elements of Computational biology and Programmed cell death. His biological study spans a wide range of topics, including Tumor necrosis factor alpha, Superoxide dismutase and Superoxide. He interconnects MAP1LC3B, BECN1, Sequestosome 1 and Autophagosome in the investigation of issues within Chaperone-mediated autophagy.
Daret K. St. Clair mainly investigates Oxidative stress, Reactive oxygen species, Doxorubicin, Mitochondrion and Cell biology. His Oxidative stress study combines topics from a wide range of disciplines, such as Cancer, Apoptosis and Bioinformatics. His work carried out in the field of Cancer brings together such families of science as Tumor necrosis factor alpha and Cancer research.
His Reactive oxygen species research is multidisciplinary, incorporating perspectives in Superoxide dismutase and Superoxide. His Doxorubicin research includes elements of Pharmacology and Protein oxidation. The study incorporates disciplines such as Oxidative phosphorylation and Antioxidant in addition to Mitochondrion.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Mitochondrial Manganese Superoxide Dismutase Prevents Neural Apoptosis and Reduces Ischemic Brain Injury: Suppression of Peroxynitrite Production, Lipid Peroxidation, and Mitochondrial Dysfunction
Jeffrey N. Keller;Mark S. Kindy;Fredrick W. Holtsberg;Daret K. St. Clair.
The Journal of Neuroscience (1998)
ROS and p53: A versatile partnership
Bin Liu;Yumin Chen;Daret K. St. Clair.
Free Radical Biology and Medicine (2008)
Regulation of superoxide dismutase genes: implications in disease.
Lu Miao;Daret K. St. Clair.
Free Radical Biology and Medicine (2009)
Loss of FBP1 by Snail-mediated repression provides metabolic advantages in basal-like breast cancer.
Chenfang Dong;Tingting Yuan;Yadi Wu;Yifan Wang.
Cancer Cell (2013)
Collateral damage in cancer chemotherapy: oxidative stress in nontargeted tissues.
Yumin Chen;Paiboon Jungsuwadee;Mary Vore;D. Allan Butterfield.
Molecular Interventions (2007)
Redox proteomics identification of oxidatively modified hippocampal proteins in mild cognitive impairment: Insights into the development of Alzheimer's disease
D. Allan Butterfield;H. Fai Poon;Daret St. Clair;Jeffery N. Keller.
Neurobiology of Disease (2006)
Manganese superoxide dismutase, MnSOD and its mimics
Sumitra Miriyala;Ivan Spasojevic;Artak Tovmasyan;Daniela Salvemini.
Biochimica et Biophysica Acta (2012)
Exacerbation of Damage and Altered NF-κB Activation in Mice Lacking Tumor Necrosis Factor Receptors after Traumatic Brain Injury
Patrick G. Sullivan;Annadora J. Bruce-Keller;Alexander G. Rabchevsky;Sylivia Christakos.
The Journal of Neuroscience (1999)
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