D-Index & Metrics Best Publications
Evelina Gatti

Evelina Gatti

Aix-Marseille University
France

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Genetics
  • Gene expression

Her scientific interests lie mostly in Cell biology, Autophagy, Antigen processing, Antigen presentation and BECN1. The various areas that she examines in her Cell biology study include Molecular biology and Ubiquitin ligase. Her research integrates issues of Transport protein, Endocytic cycle, Pleckstrin homology domain and Endosome in her study of Autophagy.

Her studies examine the connections between Antigen presentation and genetics, as well as such issues in MHC class II, with regards to Endocytosis. Evelina Gatti combines subjects such as Autophagosome, Autolysosome, MAP1LC3B, Chaperone-mediated autophagy and Physiology with her study of BECN1. Her MAP1LC3B research incorporates themes from Bioinformatics, Programmed cell death, Autophagy-Related Protein 7, Sequestosome 1 and Computational biology.

Her most cited work include:

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)

What are the main themes of her work throughout her whole career to date?

The scientist’s investigation covers issues in Cell biology, Immune system, Innate immune system, Antigen presentation and Major histocompatibility complex. Her Cell biology study incorporates themes from Antigen processing, Dendritic cell, Proinflammatory cytokine, Endocytic cycle and Endosome. In Endosome, Evelina Gatti works on issues like Autophagy, which are connected to Signal transducing adaptor protein, FYVE domain, Interleukin 4 and Physiology.

The concepts of her Immune system study are interwoven with issues in Cell, Cell type, Signal transduction, Unfolded protein response and Computational biology. Evelina Gatti interconnects Autolysosome, Chaperone-mediated autophagy, MAP1LC3B, Sequestosome 1 and Programmed cell death in the investigation of issues within Computational biology. Her Innate immune system research integrates issues from Interferon, Virology and Protein biosynthesis.

She most often published in these fields:

  • Cell biology (109.17%)
  • Immune system (50.46%)
  • Innate immune system (46.79%)

What were the highlights of her more recent work (between 2018-2021)?

  • Immune system (50.46%)
  • Cell (22.02%)
  • Cell biology (109.17%)

In recent papers she was focusing on the following fields of study:

Her primary areas of study are Immune system, Cell, Cell biology, Flow cytometry and Computational biology. Her Cell research is multidisciplinary, incorporating elements of MHC class I, Major histocompatibility complex, Ubiquitin ligase and Antigen presentation. Her Cell biology research includes elements of Ubiquitin, Immunoprecipitation and Transmembrane protein.

She interconnects Phenotype, Reprogramming, Blood drawing and Effector in the investigation of issues within Flow cytometry. Her Computational biology research incorporates elements of Whole blood and Cell type. Her research in Proteostasis intersects with topics in Cytokine, Inflammation, Proinflammatory cytokine, Signal transduction and Innate immune system.

Between 2018 and 2021, her most popular works were:

  • At the crossway of ER‐stress and proinflammatory responses (34 citations)
  • At the crossway of ER‐stress and proinflammatory responses (34 citations)
  • At the crossway of ER‐stress and proinflammatory responses (34 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Autophagy (2012)

8302 Citations

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

7788 Citations

Developmental regulation of MHC class II transport in mouse dendritic cells

Philippe Pierre;Shannon J. Turley;Evelina Gatti;Michael Hull.
Nature (1997)

960 Citations

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Parasites & Vectors (2016)

895 Citations

Suppression of eIF2α kinases alleviates Alzheimer's disease–related plasticity and memory deficits

Tao Ma;Mimi A Trinh;Alyse J Wexler;Clarisse Bourbon;Clarisse Bourbon.
Nature Neuroscience (2013)

476 Citations

Defective IL-12 production in mitogen-activated protein (MAP) kinase kinase 3 (Mkk3)-deficient mice.

Hong-Tao Lu;Derek D. Yang;Mark Allen Wysk;Evelina Gatti.
The EMBO Journal (1999)

466 Citations

PLEKHM1 Regulates Autophagosome-Lysosome Fusion through HOPS Complex and LC3/GABARAP Proteins.

David G. McEwan;Doris Popovic;Andrea Gubas;Seigo Terawaki;Seigo Terawaki;Seigo Terawaki.
Molecular Cell (2015)

340 Citations

A rat brain Sec1 homologue related to Rop and UNC18 interacts with syntaxin.

E P Garcia;E Gatti;M Butler;J Burton.
Proceedings of the National Academy of Sciences of the United States of America (1994)

302 Citations

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

279 Citations

MHC class II stabilization at the surface of human dendritic cells is the result of maturation-dependent MARCH I down-regulation

Aude De Gassart;Voahirana Camosseto;Jacques Thibodeau;Maurizio Ceppi.
Proceedings of the National Academy of Sciences of the United States of America (2008)

264 Citations

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