D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 93 Citations 49,879 329 World Ranking 957 National Ranking 587

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Internal medicine

Leonidas C. Platanias mostly deals with Cancer research, Cell biology, Signal transduction, Kinase and Molecular biology. He has included themes like Cell growth, Apoptosis, Immunology, Chronic myelogenous leukemia and MAPK/ERK pathway in his Cancer research study. His study in Cell biology is interdisciplinary in nature, drawing from both Cell culture and Biochemistry.

His work on Protein kinase A as part of general Kinase study is frequently linked to Insulin Receptor Substrate Proteins, therefore connecting diverse disciplines of science. His studies deal with areas such as Autophagy, Chaperone-mediated autophagy, BECN1 and Autolysosome as well as Programmed cell death. His Autophagy research includes themes of Computational biology and Physiology.

His most cited work include:

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Cancer research, Cell biology, Signal transduction, Kinase and PI3K/AKT/mTOR pathway. His Cancer research study deals with Leukemia intersecting with Myeloid. His Cell biology research incorporates elements of Molecular biology and Receptor.

His Signal transduction research is multidisciplinary, incorporating elements of Interferon and Regulation of gene expression. In general Kinase, his work in Protein kinase A, p38 mitogen-activated protein kinases and Mitogen-activated protein kinase is often linked to EIF4E linking many areas of study. C-Raf and Cyclin-dependent kinase 2 is closely connected to MAP kinase kinase kinase in his research, which is encompassed under the umbrella topic of ASK1.

He most often published in these fields:

  • Cancer research (64.31%)
  • Cell biology (37.90%)
  • Signal transduction (34.48%)

What were the highlights of his more recent work (between 2016-2021)?

  • Cancer research (64.31%)
  • Cancer (10.08%)
  • Metastatic breast cancer (5.24%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Cancer research, Cancer, Metastatic breast cancer, Internal medicine and Breast cancer. His Cancer research research incorporates themes from PI3K/AKT/mTOR pathway, Kinase and Cell growth. His research on Kinase concerns the broader Cell biology.

His research in Cell biology intersects with topics in Transcription, Protein subunit and Gene. His work is dedicated to discovering how Internal medicine, Oncology are connected with Stage, Myelodysplastic syndromes, Disease and Chemotherapy and other disciplines. His research investigates the connection between Myeloid leukemia and topics such as Phosphorylation that intersect with issues in Interferon regulatory factors and Computational biology.

Between 2016 and 2021, his most popular works were:

  • IDO1 Inhibition Synergizes with Radiation and PD-1 Blockade to Durably Increase Survival Against Advanced Glioblastoma (66 citations)
  • Concordance of Genomic Alterations by Next-Generation Sequencing in Tumor Tissue versus Circulating Tumor DNA in Breast Cancer. (63 citations)
  • Cell-free DNA and circulating tumor cells: Comprehensive liquid biopsy analysis in advanced breast cancer (60 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Internal medicine

Leonidas C. Platanias focuses on Cancer research, Kinase, Signal transduction, Myeloid leukemia and Cyclin-dependent kinase 9. His work deals with themes such as Cancer cell, Cancer stem cell, PI3K/AKT/mTOR pathway and Cell culture, which intersect with Cancer research. His Kinase study integrates concerns from other disciplines, such as Apoptosis and Cell growth.

His Signal transduction study deals with the bigger picture of Cell biology. His work on STAT1, Tyrosine phosphorylation and Janus kinase as part of general Cell biology research is often related to SIRT2, thus linking different fields of science. His study in Myeloid leukemia is interdisciplinary in nature, drawing from both Haematopoiesis, Extramedullary hematopoiesis, Leukemia and Bone marrow.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

7788 Citations

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Autophagy (2012)

7068 Citations

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

4781 Citations

Mechanisms of type-I- and type-II-interferon-mediated signalling.

Leonidas C. Platanias.
Nature Reviews Immunology (2005)

3445 Citations

Map kinase signaling pathways and hematologic malignancies

Leonidas C. Platanias.
Blood (2003)

600 Citations

DIRECT ASSOCIATION WITH AND DEPHOSPHORYLATION OF JAK2 KINASE BY THE SH2-DOMAIN-CONTAINING PROTEIN TYROSINE PHOSPHATASE SHP-1

Huaiyuan Jiao;Karim Berrada;Wentian Yang;Mina Tabrizi.
Molecular and Cellular Biology (1996)

418 Citations

Signaling pathways activated by interferons.

Leonidas C. Platanias;Eleanor N. Fish.
Experimental Hematology (1999)

373 Citations

Mitochondrial reactive oxygen species activation of p38 mitogen-activated protein kinase is required for hypoxia signaling.

Brooke M. Emerling;Leonidas C. Platanias;Emma Black;Angel R. Nebreda.
Molecular and Cellular Biology (2005)

303 Citations

Activation of the p38 Mitogen-activated Protein Kinase by Type I Interferons

Shahab Uddin;Beata Majchrzak;Joanna Woodson;Pony Arunkumar.
Journal of Biological Chemistry (1999)

292 Citations

Protein kinase C-δ (PKC-δ) is activated by type I interferons and mediates phosphorylation of Stat1 on serine 727

Shahab Uddin;Antonella Sassano;Dilip K. Deb;Amit Verma.
Journal of Biological Chemistry (2002)

286 Citations

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