World's Best Scientists 2026 revealed!
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Biology and Biochemistry
Qatar
2026

D-Index & Metrics

Biology and Biochemistry

D-Index
81
Citations
19865
World Ranking
3951
National Ranking
3

Research.com Recognitions

  • 2026 - Research.com Biology and Biochemistry in Qatar Leader Award
  • 2025 - Research.com Biology and Biochemistry in Qatar Leader Award

Overview

Shahab Uddin is affiliated with Hamad Medical Corporation in Qatar and has contributed extensively to the fields of biochemistry, genetics, and molecular biology, as well as medicine. Their research spans molecular biology, oncology, cancer research, immunology, and epidemiology, showing a broad interdisciplinary approach.

Their recent publications include the following works:

  • CAR-cell therapy in the era of solid tumor treatment: current challenges and emerging therapeutic advances, 2023, Molecular Cancer
  • Emerging COVID-19 variants and their impact on SARS-CoV-2 diagnosis, therapeutics and vaccines, 2022, Annals of Medicine
  • Targeting cancer signaling pathways by natural products: Exploring promising anti-cancer agents, 2022, Biomedicine & Pharmacotherapy
  • Cytokine- and chemokine-induced inflammatory colorectal tumor microenvironment: Emerging avenue for targeted therapy, 2022, Cancer Communications
  • Inducing Angiogenesis, a Key Step in Cancer Vascularization, and Treatment Approaches, 2020, Cancers

Shahab Uddin has collaborated frequently with several researchers, including:

  • Kirti S. Prabhu
  • Said Dermime
  • Maysaloun Merhi
  • Shilpa Kuttikrishnan
  • Aamir Ahmad

Their work is often published in journals and venues such as:

  • Seminars in Cancer Biology (13 publications)
  • Biomedicine & Pharmacotherapy (12 publications)
  • Cancers (8 publications)
  • International Journal of Molecular Sciences (8 publications)
  • Journal of Translational Medicine (7 publications)

Their research topics cover various areas related to cancer and molecular biology, including:

  • Cancer-related molecular mechanisms research
  • Cancer Immunotherapy and Biomarkers
  • Circular RNAs in diseases
  • Cancer Cells and Metastasis
  • Epigenetics and DNA Methylation
  • MicroRNA in disease regulation
  • RNA modifications and cancer

The subfields they frequently contribute to demonstrate a focus on understanding molecular and cellular functions related to cancer and immunity. Their scope includes molecular signaling, tumor microenvironment, inflammatory mechanisms, and therapeutic approaches in oncology.

Best Publications

  • Extracellular vesicles-mediated intercellular communication: roles in the tumor microenvironment and anti-cancer drug resistance.

    Selma Maacha;Ajaz A. Bhat;Lizandra Jimenez;Afsheen Raza

  • Tight Junction Proteins and Signaling Pathways in Cancer and Inflammation: A Functional Crosstalk.

    Ajaz A Bhat;Srijayaprakash Uppada;Iman W Achkar;Sheema Hashem

  • Protein kinase C-δ (PKC-δ) is activated by type I interferons and mediates phosphorylation of Stat1 on serine 727

    Shahab Uddin;Antonella Sassano;Dilip K. Deb;Amit Verma

  • Role of phosphatidylinositol 3'-kinase/AKT pathway in diffuse large B-cell lymphoma survival.

    Shahab Uddin;Azhar R. Hussain;Abdul K. Siraj;Pulicat S. Manogaran

  • Activation of the p38 Mitogen-activated Protein Kinase by Type I Interferons

    Shahab Uddin;Beata Majchrzak;Joanna Woodson;Pony Arunkumar

  • Interferon-α Engages the Insulin Receptor Substrate-1 to Associate with the Phosphatidylinositol 3′-Kinase (∗)

    Shahab Uddin;Lynne Yenush;Xiao Jian Sun;Michelle E. Sweet

  • Role of miRNA-Regulated Cancer Stem Cells in the Pathogenesis of Human Malignancies.

    Abdul Q Khan;Eiman I Ahmed;Noor R Elareer;Kulsoom Junejo

  • The Rac1/p38 Mitogen-activated Protein Kinase Pathway Is Required for Interferon α-dependent Transcriptional Activation but Not Serine Phosphorylation of Stat Proteins

    Shahab Uddin;Fatima Lekmine;Niti Sharma;Beata Majchrzak

  • Activation of Rac1 and the p38 Mitogen-activated Protein Kinase Pathway in Response to All-trans-retinoic Acid

    Yazan Alsayed;Shahab Uddin;Nadim Mahmud;Fatima Lekmine

  • Interferon-α Resistance in a Cutaneous T-Cell Lymphoma Cell Line Is Associated With Lack of STAT1 Expression

    Wenn H. Sun;Carlos Pabon;Carlos Pabon;Yazan Alsayed;Yazan Alsayed;Paul P. Huang;Paul P. Huang

  • Clinicopathological Analysis of Papillary Thyroid Cancer with PIK3CA Alterations in a Middle Eastern Population

    Jehad Abubaker;Zeenath Jehan;Prashant Bavi;Mehar Sultana

  • Protein kinase B/Akt mediates effects of insulin on hepatic insulin-like growth factor-binding protein-1 gene expression through a conserved insulin response sequence.

    Stephen B. Cichy;Shahab Uddin;Alexey Danilkovich;Shaodong Guo

  • Curcumin induces apoptosis via inhibition of PI3'-kinase/AKT pathway in acute T cell leukemias.

    Azhar Hussain;M. Al-Rasheed;P. S. Manogaran;K. A. Al-Hussein

  • Protein Kinase C-δ Regulates Thrombin-Induced ICAM-1 Gene Expression in Endothelial Cells via Activation of p38 Mitogen-Activated Protein Kinase

    Arshad Rahman;Khandaker N. Anwar;Shahab Uddin;Ning Xu

  • Vascular Endothelial Growth Factor (VEGF) Signaling in Tumour Vascularization: Potential and Challenges

    Kodappully Sivaraman Siveen;Kirti Prabhu;Roopesh Krishnankutty;Shilpa Kuttikrishnan

  • Activation of the p38 Mitogen-activated Protein Kinase Mediates the Suppressive Effects of Type I Interferons and Transforming Growth Factor-β on Normal Hematopoiesis

    Amit K. Verma;Dilip K. Deb;Antonella Sassano;Shahab Uddin

  • The p38 MAPK Pathway Mediates the Growth Inhibitory Effects of Interferon-α in BCR-ABL-expressing Cells

    Ingrid A. Mayer;Amit Verma;Isabella M. Grumbach;Shahab Uddin

  • Differentiation stage-specific activation of p38 mitogen-activated protein kinase isoforms in primary human erythroid cells.

    Shahab Uddin;Jeong Ah-Kang;Jodie Ulaszek;Dolores Mahmud

  • Genome-wide expression analysis of Middle Eastern colorectal cancer reveals FOXM1 as a novel target for cancer therapy.

    Shahab Uddin;Maqbool Ahmed;Azhar Hussain;Jehad Abubaker

  • Cross-talk between NFkB and the PI3-kinase/AKT pathway can be targeted in primary effusion lymphoma (PEL) cell lines for efficient apoptosis.

    Azhar R. Hussain;Saeeda O. Ahmed;Maqbool Ahmed;Omar S. Khan

Frequent Co-Authors

Leonidas C. Platanias
Leonidas C. Platanias Northwestern University
Eleanor N. Fish
Eleanor N. Fish University of Toronto
Martin Steinhoff
Martin Steinhoff Hamad Medical Corporation
Amittha Wickrema
Amittha Wickrema University of Chicago
Amit Verma
Amit Verma Albert Einstein College of Medicine
Brian J. Druker
Brian J. Druker Oregon Health & Science University
Morris F. White
Morris F. White Boston Children's Hospital
Ravinder Reddy
Ravinder Reddy University of Pennsylvania
Wael El-Rifai
Wael El-Rifai University of Miami
Fowzan S. Alkuraya
Fowzan S. Alkuraya Alfaisal University

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