2023 - Research.com Molecular Biology in United States Leader Award
2022 - Research.com Best Scientist Award
2018 - Member of the National Academy of Sciences
2012 - Fellow of the American Association for the Advancement of Science (AAAS)
2012 - Fellow, National Academy of Inventors
2002 - Fellow of the Royal Society, United Kingdom
Roger J. Davis mainly investigates Cell biology, MAP kinase kinase kinase, MAP2K7, Mitogen-activated protein kinase kinase and Signal transduction. Cell biology is frequently linked to Molecular biology in his study. His study in Molecular biology is interdisciplinary in nature, drawing from both c-Jun N-terminal kinases and Regulation of gene expression.
His MAP2K7 study combines topics from a wide range of disciplines, such as c-Raf and ASK1. His work in Mitogen-activated protein kinase kinase addresses issues such as JUN kinase, which are connected to fields such as Transactivation. The study incorporates disciplines such as Inflammation, Apoptosis, Transcription factor, NFAT and Kinase in addition to Signal transduction.
His primary areas of investigation include Cell biology, Kinase, Signal transduction, Molecular biology and Mitogen-activated protein kinase kinase. His Cell biology study integrates concerns from other disciplines, such as Apoptosis and Biochemistry. His studies in Kinase integrate themes in fields like Internal medicine, c-jun and Endocrinology.
He works mostly in the field of Signal transduction, limiting it down to topics relating to Cancer research and, in certain cases, Carcinogenesis. His Mitogen-activated protein kinase kinase research integrates issues from MAP2K7 and MAP kinase kinase kinase. Roger J. Davis works mostly in the field of MAP2K7, limiting it down to concerns involving MAPK14 and, occasionally, MAPK7.
Roger J. Davis mostly deals with Cell biology, Kinase, Signal transduction, Internal medicine and Cancer research. His Cell biology course of study focuses on Adipose tissue and Alternative splicing. His Kinase research incorporates themes from Transcription factor, Homeostasis and Downregulation and upregulation.
The concepts of his Signal transduction study are interwoven with issues in Glutamate receptor and NMDA receptor. His Internal medicine research is multidisciplinary, incorporating perspectives in Endocrinology, Programmed cell death, MAP kinase kinase kinase and Cardiology. The Protein kinase A study combines topics in areas such as Molecular biology and STAT protein.
His scientific interests lie mostly in Cell biology, Internal medicine, Endocrinology, Signal transduction and Kinase. His Cell biology study combines topics in areas such as Adipose tissue, Transcriptome and Proinflammatory cytokine. His Signal transduction research includes themes of NMDA receptor, Glutamate receptor, Postsynaptic density and Cancer research.
He has included themes like Downregulation and upregulation and Pancreas in his Kinase study. His study looks at the relationship between Cell signaling and fields such as Myostatin, as well as how they intersect with chemical problems. His MAP kinase kinase kinase research is multidisciplinary, incorporating elements of MAP2K7 and ASK1.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Opposing Effects of ERK and JNK-p38 MAP Kinases on Apoptosis
Zhengui Xia;Martin Dickens;Joël Raingeaud;Roger J. Davis.
Signal transduction by the JNK group of MAP kinases.
Roger J Davis.
JNK1: A protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain
Benoit Dérijard;Benoit Dérijard;Masahiko Hibi;I-Huan Wu;I-Huan Wu;Tamera Barrett.
The mitogen-activated protein kinase signal transduction pathway.
Roger J. Davis.
Journal of Biological Chemistry (1993)
Pro-inflammatory Cytokines and Environmental Stress Cause p38 Mitogen-activated Protein Kinase Activation by Dual Phosphorylation on Tyrosine and Threonine (∗)
Joel Raingeaud;Shashi Gupta;Jeffrey Scott Rogers;Martin Dickens.
Journal of Biological Chemistry (1995)
Carbon monoxide has anti-inflammatory effects involving the mitogen-activated protein kinase pathway
Leo E. Otterbein;Leo E. Otterbein;Fritz H. Bach;Jawed Alam;Miguel P. Soares.
Nature Medicine (2000)
cPLA2 is phosphorylated and activated by MAP kinase.
Lih-Ling Lin;Markus Wartmann;Alice Y. Lin;John L. Knopf.
The JNK signal transduction pathway.
Claire R Weston;Roger J Davis.
Current Opinion in Cell Biology (2002)
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