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Biology and Biochemistry

D-Index
77
Citations
24443
World Ranking
4726
National Ranking
2288

Overview

Mercedes Rincon is affiliated with the University of Vermont in the United States and has a professional focus spanning medicine, biochemistry, genetics, and molecular biology. Their research output includes 43 publications in medicine and 31 in biochemistry, genetics, and molecular biology, with particular attention to molecular biology, oncology, epidemiology, immunology, and physiology.

Their main research topics encompass liver disease diagnosis and treatment, diet and metabolism studies, gut microbiota and health, CAR-T cell therapy research, virus-based gene therapy, drug transport and resistance mechanisms, and epigenetics including DNA methylation.

Frequent publication venues for Mercedes Rincon include Cancer Research and The Journal of Immunology, each with six publications. Other common venues are the Journal of Hepatology with four publications, Nature Communications with three, and Hepatology with two.

Their recent papers demonstrate a range of scientific inquiry, including:

  • Mitochondrial ATP fuels ABC transporter-mediated drug efflux in cancer chemoresistance, 2021, Nature Communications
  • Silencing hepatic MCJ attenuates non-alcoholic fatty liver disease (NAFLD) by increasing mitochondrial fatty acid oxidation, 2020, Nature Communications
  • Manipulating mitochondrial electron flow enhances tumor immunogenicity, 2023, Science
  • Mitochondrial bioenergetics boost macrophage activation, promoting liver regeneration in metabolically compromised animals, 2021, Hepatology
  • Enhanced mitochondrial activity reshapes a gut microbiota profile that delays NASH progression, 2022, Hepatology

Mercedes Rincon collaborates regularly with several researchers, including Felipe Valença-Pereira, Juan Anguíta, Maria Luz Martínez-Chantar, Marta Varela-Rey, and Qian Fang. The frequency of coauthorship ranges from six to ten joint publications, indicating ongoing research partnerships.

Best Publications

  • Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene

    D D Yang;C Y Kuan;A J Whitmarsh;A J Whitmarsh;M Rincón

  • The two faces of IL-6 on Th1/Th2 differentiation

    Sean Diehl;Mercedes Rincón

  • INTERLEUKIN (IL)-6 DIRECTS THE DIFFERENTIATION OF IL-4-PRODUCING CD4+ T CELLS

    Mercedes Rincón;Juan Anguita;Tetsuo Nakamura;Erol Fikrig

  • Role of IL-6 in asthma and other inflammatory pulmonary diseases.

    Mercedes Rincon;Charles G. Irvin

  • A switch from white to brown fat increases energy expenditure in cancer-associated cachexia.

    Michele Petruzzelli;Martina Schweiger;Renate Schreiber;Ramon Campos-Olivas

  • The effects of IL-6 on CD4 T cell responses

    Oliver Dienz;Mercedes Rincon

  • Differentiation of CD4+ T Cells to Th1 Cells Requires MAP Kinase JNK2

    Derek D Yang;Dietrich Conze;Alan J Whitmarsh;Tamera Barrett

  • Phosphorylation by p38 MAPK as an Alternative Pathway for GSK3β Inactivation

    Tina M. Thornton;Gustavo Pedraza-Alva;Bin Deng;C. David Wood

  • Non-classical p38 map kinase functions: cell cycle checkpoints and survival.

    Tina M. Thornton;Mercedes Rincon

  • Interferon‐γ expression by Th1 effector T cells mediated by the p38 MAP kinase signaling pathway

    Mercedes Rincón;Hervé Enslen;Joël Raingeaud;Joël Raingeaud;Michael Recht

  • Inhibition of Th1 Differentiation by IL-6 Is Mediated by SOCS1

    Sean Diehl;Juan Anguita;Angelika Hoffmeyer;Tyler Zapton

  • The induction of antibody production by IL-6 is indirectly mediated by IL-21 produced by CD4+ T cells.

    Oliver Dienz;Sheri M. Eaton;Jeffrey P. Bond;Wendy Neveu

  • Transgenic mice demonstrate AP-1 (activator protein-1) transactivation is required for tumor promotion.

    Matthew R. Young;Jian-Jian Li;Jian-Jian Li;Mercedes Rincón;Richard A. Flavell

  • Nuclear Accumulation of NFAT4 Opposed by the JNK Signal Transduction Pathway

    Chi Wing Chow;Mercedes Rincón;Mercedes Rincón;Julie Cavanagh;Julie Cavanagh;Martin Dickens;Martin Dickens

  • Interleukin-6: from an inflammatory marker to a target for inflammatory diseases

    Mercedes Rincon

  • Triggering of T cell proliferation through AIM, an activation inducer molecule expressed on activated human lymphocytes.

    M Cebrián;E Yagüe;M Rincón;M López-Botet

  • AP-1 transcriptional activity requires both T-cell receptor-mediated and co-stimulatory signals in primary T lymphocytes

    M Rincón;R A Flavell

  • Autocrine Production of Interleukin 6 Causes Multidrug Resistance in Breast Cancer Cells

    Dietrich Conze;Linda Weiss;Patricia Susan Regen;Alok Bhushan

  • NF-κB Activation by the Pre-T Cell Receptor Serves as a Selective Survival Signal in T Lymphocyte Development

    Reinhard E Voll;Eijiro Jimi;Roderick J Phillips;Domingo F Barber

  • Regulation of the immune response by stress‐activated protein kinases

    Mercedes Rincón;Roger J. Davis

Frequent Co-Authors

Richard A. Flavell
Richard A. Flavell Yale University
Juan Anguita
Juan Anguita Ikerbasque
Roger J. Davis
Roger J. Davis University of Massachusetts Chan Medical School
Cory Teuscher
Cory Teuscher University of Vermont
Laura Haynes
Laura Haynes University of Connecticut
Douglas J. Taatjes
Douglas J. Taatjes University of Vermont
Matthew E. Poynter
Matthew E. Poynter University of Vermont
Alan J. Whitmarsh
Alan J. Whitmarsh University of Manchester
Erol Fikrig
Erol Fikrig Yale University
Charles G. Irvin
Charles G. Irvin University of Vermont

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