D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Genetics and Molecular Biology D-index 68 Citations 14,160 161 World Ranking 1663 National Ranking 851

Research.com Recognitions

Awards & Achievements

2012 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Enzyme
  • DNA

Nancy H. Colburn mainly focuses on Molecular biology, Cancer research, Carcinogenesis, Cell biology and Tumor promotion. The various areas that Nancy H. Colburn examines in her Molecular biology study include Cell culture, Epidermal growth factor, Reporter gene, Gene expression and Neoplastic transformation. Nancy H. Colburn has researched Cancer research in several fields, including Cancer, Protein kinase B, MAP kinase kinase kinase, Pathology and ASK1.

Nancy H. Colburn is studying Tumor suppressor gene, which is a component of Carcinogenesis. Her Cell biology research includes themes of Eukaryotic translation, EIF4G and Initiation factor. Her Tumor promotion research includes elements of Inflammation, mRNA display, Transcription factor, Transactivation and Molecular cloning.

Her most cited work include:

  • MicroRNA-21 (miR-21) post-transcriptionally downregulates tumor suppressor Pdcd4 and stimulates invasion, intravasation and metastasis in colorectal cancer. (1517 citations)
  • MicroRNA-21 promotes cell transformation by targeting the programmed cell death 4 gene. (577 citations)
  • The Transformation Suppressor Pdcd4 Is a Novel Eukaryotic Translation Initiation Factor 4A Binding Protein That Inhibits Translation (404 citations)

What are the main themes of her work throughout her whole career to date?

The scientist’s investigation covers issues in Molecular biology, Cancer research, Carcinogenesis, Cell culture and Cell biology. Her Molecular biology research integrates issues from Gene expression, Transcription factor, Neoplastic transformation, Transfection and Tumor promotion. Her Cancer research study integrates concerns from other disciplines, such as Cancer, Colorectal cancer, Gene knockdown, Immunology and Tumor suppressor gene.

As part of one scientific family, Nancy H. Colburn deals mainly with the area of Carcinogenesis, narrowing it down to issues related to the Cell growth, and often Apoptosis. Her studies deal with areas such as Cell, Epidermis and Tetradecanoylphorbol Acetate as well as Cell culture. Her work investigates the relationship between Cell biology and topics such as EIF4G that intersect with problems in Eukaryotic translation.

She most often published in these fields:

  • Molecular biology (41.98%)
  • Cancer research (31.30%)
  • Carcinogenesis (25.57%)

What were the highlights of her more recent work (between 2010-2020)?

  • Cancer research (31.30%)
  • Cancer (12.60%)
  • Carcinogenesis (25.57%)

In recent papers she was focusing on the following fields of study:

Nancy H. Colburn mostly deals with Cancer research, Cancer, Carcinogenesis, Biochemistry and Colorectal cancer. Her research in Cancer research intersects with topics in Endocrinology, Cell growth, Gene knockdown, Immunology and Tumor progression. Her work carried out in the field of Cell growth brings together such families of science as Apoptosis, Molecular biology, Kinase, Structure–activity relationship and Chemical synthesis.

Her study explores the link between Molecular biology and topics such as microRNA that cross with problems in Cell biology and Messenger RNA. Her Cancer research is multidisciplinary, relying on both Alternative medicine and Stem cell. Nancy H. Colburn works mostly in the field of Carcinogenesis, limiting it down to topics relating to eIF4A and, in certain cases, Transactivation, Downregulation and upregulation and Suppressor, as a part of the same area of interest.

Between 2010 and 2020, her most popular works were:

  • Sulfiredoxin–Peroxiredoxin IV axis promotes human lung cancer progression through modulation of specific phosphokinase signaling (78 citations)
  • Fluorescence endoscopic detection of murine colitis-associated colon cancer by topically applied enzymatically rapid-activatable probe (74 citations)
  • An integrated understanding of the physiological response to elevated extracellular phosphate (73 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Cancer

Cancer research, Cancer, Cell growth, Carcinogenesis and Biochemistry are her primary areas of study. The study incorporates disciplines such as Tumor necrosis factor alpha, Internal medicine, Endocrinology, Gene knockdown and Kinase in addition to Cancer research. Her Cancer research incorporates themes from Colitis and Cellular differentiation.

Her work deals with themes such as Apoptosis, Transcription factor, IκBα, Molecular biology and microRNA, which intersect with Cell growth. Nancy H. Colburn studied Molecular biology and Resveratrol that intersect with Signal transduction. The concepts of her Carcinogenesis study are interwoven with issues in Ubiquitin, Ubiquitin ligase, Beta-Transducin Repeat-Containing Proteins, Reporter gene and DNA ligase.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

MicroRNA-21 (miR-21) post-transcriptionally downregulates tumor suppressor Pdcd4 and stimulates invasion, intravasation and metastasis in colorectal cancer.

I. A. Asangani;S. A.K. Rasheed;D. A. Nikolova;J. H. Leupold.
Oncogene (2008)

2115 Citations

MicroRNA-21 promotes cell transformation by targeting the programmed cell death 4 gene.

Z. Lu;M. Liu;V. Stribinskis;C. M. Klinge.
Oncogene (2008)

814 Citations

The Transformation Suppressor Pdcd4 Is a Novel Eukaryotic Translation Initiation Factor 4A Binding Protein That Inhibits Translation

Hsin Sheng Yang;Aaron P. Jansen;Anton A. Komar;Xiaojing Zheng.
Molecular and Cellular Biology (2003)

545 Citations

Transgenic mice demonstrate AP-1 (activator protein-1) transactivation is required for tumor promotion.

Matthew R. Young;Jian-Jian Li;Jian-Jian Li;Mercedes Rincón;Richard A. Flavell.
Proceedings of the National Academy of Sciences of the United States of America (1999)

448 Citations

Blocking of tumor promoter-induced AP-1 activity inhibits induced transformation in JB6 mouse epidermal cells.

Zigang Dong;Michael J. Birrer;Rebecca G. Watts;Lynn M. Matrisian.
Proceedings of the National Academy of Sciences of the United States of America (1994)

426 Citations

Tumour promoter induces anchorage independence irreversibly

Nancy H. Colburn;Brigitte F. Former;Katherine A. Nelson;Stuart H. Yuspa.
Nature (1979)

377 Citations

Activator protein 1 (AP-1)– and nuclear factor κB (NF-κB)–dependent transcriptional events in carcinogenesis

Tin-Chen Hsu;Matthew R Young;Joan Cmarik;Nancy H Colburn.
Free Radical Biology and Medicine (2000)

343 Citations

Epidermal expression of the translation inhibitor programmed cell death 4 suppresses tumorigenesis.

Aaron P. Jansen;Corinne E. Camalier;Nancy H. Colburn.
Cancer Research (2005)

338 Citations

Differentially expressed protein Pdcd4 inhibits tumor promoter-induced neoplastic transformation

Joan L. Cmarik;Hongzhong Min;Glenn Hegamyer;Shuning Zhan.
Proceedings of the National Academy of Sciences of the United States of America (1999)

330 Citations

AP1/jun function is differentially induced in promotion-sensitive and resistant JB6 cells

Lori R. Bernstein;Nancy H. Colburn.
Science (1989)

304 Citations

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