His primary areas of investigation include Cell biology, MAP kinase kinase kinase, MAP2K7, Mitogen-activated protein kinase kinase and Molecular biology. The Cell biology study which covers Cytotoxic T cell that intersects with Cytokine and Effector. The various areas that Alan J. Whitmarsh examines in his MAP kinase kinase kinase study include Mitogen-activated protein kinase and Transcription factor.
His research investigates the link between MAP2K7 and topics such as ASK1 that cross with problems in Cyclin-dependent kinase 9. His Molecular biology study combines topics from a wide range of disciplines, such as Excitotoxicity and c-Jun N-terminal kinases. His c-Jun N-terminal kinases study combines topics in areas such as Transcription factor complex, Kainic acid, Programmed cell death and Neuroprotection.
His primary areas of study are Cell biology, MAP kinase kinase kinase, Mitogen-activated protein kinase kinase, ASK1 and MAP2K7. His work focuses on many connections between Cell biology and other disciplines, such as Biochemistry, that overlap with his field of interest in Function. His study looks at the relationship between ASK1 and fields such as Cyclin-dependent kinase 9, as well as how they intersect with chemical problems.
His research integrates issues of c-Raf and MAPK14 in his study of MAP2K7. His research in Mitogen-activated protein kinase intersects with topics in MAPK phosphatase, p38 mitogen-activated protein kinases, Gene expression, Transcription factor and Molecular biology. Alan J. Whitmarsh studied Molecular biology and Transcription factor complex that intersect with Kainic acid and Excitotoxicity.
The scientist’s investigation covers issues in Cell biology, Kinase, PI3K/AKT/mTOR pathway, Mitochondrion and Retrograde signaling. Cell biology is closely attributed to Biochemistry in his study. His work on Dephosphorylation, Autophagy-related protein 13, Phosphorylation cascade and Protein phosphorylation as part of general Biochemistry study is frequently connected to Cellular localization, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
Alan J. Whitmarsh has included themes like Regulator and Cell growth in his PI3K/AKT/mTOR pathway study. His work on Respiratory enzyme and Mitochondrial fission as part of his general Mitochondrion study is frequently connected to COQ7, thereby bridging the divide between different branches of science. His MAP kinase kinase kinase research includes themes of Sciatic nerve, Akt/PKB signaling pathway and Streptozotocin.
Alan J. Whitmarsh spends much of his time researching Cell biology, Mitochondrion, Retrograde signaling, Stress granule assembly and Kinase. Cell biology is a component of his Unfolded protein response and Mitochondrial fission studies. His work in the fields of Unfolded protein response, such as Mitochondrial unfolded protein response, intersects with other areas such as COQ7 and DNAJA3.
His biological study spans a wide range of topics, including Cell and Cell nucleus, Nucleus. PI3K/AKT/mTOR pathway, P70-S6 Kinase 1, Effector and Neurodegeneration are fields of study that intersect with his Stress granule assembly study.
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Transcription factor AP-1 regulation by mitogen-activated protein kinase signal transduction pathways
A J Whitmarsh;R J Davis.
Journal of Molecular Medicine (1996)
Selective interaction of JNK protein kinase isoforms with transcription factors.
Shashi Gupta;Tamera Barrett;Alan J. Whitmarsh;Julie Cavanagh.
The EMBO Journal (1996)
MKK3- and MKK6-regulated gene expression is mediated by the p38 mitogen-activated protein kinase signal transduction pathway.
Joel Raingeaud;Alan J. Whitmarsh;Tamera Barrett;Benoit Derijard.
Molecular and Cellular Biology (1996)
Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene
D D Yang;C Y Kuan;A J Whitmarsh;A J Whitmarsh;M Rincón.
Integration of MAP Kinase Signal Transduction Pathways at the Serum Response Element
Alan J. Whitmarsh;Paul Shore;Andrew D. Sharrocks;Roger J. Davis.
A mammalian scaffold complex that selectively mediates MAP kinase activation
Alan J. Whitmarsh;Julie Cavanagh;Cathy Tournier;Jun Yasuda.
Defective T cell differentiation in the absence of Jnk1
Chen Dong;Derek D. Yang;Mark Allen Wysk;Alan J. Whitmarsh.
The JIP group of mitogen-activated protein kinase scaffold proteins
Jun Yasuda;Alan J. Whitmarsh;Julie Cavanagh;Manoj Sharma.
Molecular and Cellular Biology (1999)
Differentiation of CD4+ T Cells to Th1 Cells Requires MAP Kinase JNK2
Derek D Yang;Dietrich Conze;Alan J Whitmarsh;Tamera Barrett.
Transcriptional regulation by the MAP kinase signaling cascades
Shen Hsi Yang;Andrew D. Sharrocks;Alan J. Whitmarsh.
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