What is he best known for?
The fields of study he is best known for:
- Gene
- Apoptosis
- Internal medicine
His primary areas of investigation include Cell biology, ASK1, MAP kinase kinase kinase, Signal transduction and Apoptosis.
His Cell biology research includes elements of Thioredoxin and Biochemistry.
Hidenori Ichijo combines subjects such as Cancer research, MAP2K7 and p38 mitogen-activated protein kinases with his study of ASK1.
His research in MAP kinase kinase kinase intersects with topics in Molecular biology, Cyclin-dependent kinase 5 and Kinase activity.
His Signal transduction study incorporates themes from Regulation of gene expression and Response element.
He interconnects Oxidative stress and Phosphorylation in the investigation of issues within Apoptosis.
His most cited work include:
- Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1. (2078 citations)
- Induction of Apoptosis by ASK1, a Mammalian MAPKKK That Activates SAPK/JNK and p38 Signaling Pathways (2000 citations)
- Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018. (1421 citations)
What are the main themes of his work throughout his whole career to date?
Hidenori Ichijo spends much of his time researching Cell biology, ASK1, Kinase, Apoptosis and Signal transduction.
His Cell biology study combines topics in areas such as Molecular biology, Biochemistry and Programmed cell death.
His ASK1 research incorporates themes from MAP2K7, Cancer research, MAP kinase kinase kinase and p38 mitogen-activated protein kinases.
His MAP kinase kinase kinase research includes themes of Cyclin-dependent kinase 9 and Mitogen-activated protein kinase kinase.
His work carried out in the field of Apoptosis brings together such families of science as Reactive oxygen species and Cell growth.
His Signal transduction study integrates concerns from other disciplines, such as Unfolded protein response, Endoplasmic reticulum, Endocrinology and Internal medicine.
He most often published in these fields:
- Cell biology (59.08%)
- ASK1 (51.41%)
- Kinase (30.69%)
What were the highlights of his more recent work (between 2014-2021)?
- Cell biology (59.08%)
- ASK1 (51.41%)
- Kinase (30.69%)
In recent papers he was focusing on the following fields of study:
Hidenori Ichijo mostly deals with Cell biology, ASK1, Kinase, Apoptosis and Osmotic shock.
His biological study spans a wide range of topics, including Regulator, Oxidative stress and Programmed cell death.
His Programmed cell death study combines topics in areas such as Cell and Neuroscience.
He combines subjects such as Cancer research, Signal transduction and p38 mitogen-activated protein kinases with his study of ASK1.
His p38 mitogen-activated protein kinases research is multidisciplinary, incorporating perspectives in Lipid peroxide, Endocrinology and Internal medicine.
He studies Kinase, namely MAP kinase kinase kinase.
Between 2014 and 2021, his most popular works were:
- Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018. (1421 citations)
- Essential versus accessory aspects of cell death: recommendations of the NCCD 2015 (591 citations)
- The Src/c-Abl pathway is a potential therapeutic target in amyotrophic lateral sclerosis (104 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Apoptosis
- Internal medicine
Hidenori Ichijo mainly focuses on Cell biology, ASK1, Kinase, Programmed cell death and Apoptosis.
His Cell biology study incorporates themes from Osmotic shock and Biochemistry.
His study in ASK1 is interdisciplinary in nature, drawing from both Cancer research, Ubiquitin and Genetically modified mouse.
His Kinase study focuses on MAP kinase kinase kinase in particular.
His Programmed cell death study integrates concerns from other disciplines, such as Brown adipose tissue, Neuroscience, Transduction and Cell growth.
The Signal transduction study combines topics in areas such as Thermogenin and Regulation of gene expression.
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