The scientist’s investigation covers issues in Programmed cell death, Cell biology, Autophagy, Apoptosis and Cancer research. He interconnects Cerebellum, Neuroscience, DNA methylation, Cancer cell and Molecular biology in the investigation of issues within Programmed cell death. The Cell biology study combines topics in areas such as Statin and Cell growth.
Saeid Ghavami has included themes like Unfolded protein response, PI3K/AKT/mTOR pathway, Lysosome and Virus in his Autophagy study. His Cancer research research is multidisciplinary, relying on both Cancer, Chemotherapy, Immunology and HMG-CoA reductase. His work carried out in the field of Computational biology brings together such families of science as MAP1LC3B, Chaperone-mediated autophagy, BECN1 and Autolysosome.
Saeid Ghavami mostly deals with Cell biology, Autophagy, Internal medicine, Cancer research and Apoptosis. In his work, Cardiac fibrosis is strongly intertwined with Myofibroblast, which is a subfield of Cell biology. Saeid Ghavami mostly deals with ATG5 in his studies of Autophagy.
In his research, Tuberculosis is intimately related to Immunology, which falls under the overarching field of Cancer research. His work on Apoptosis deals in particular with Programmed cell death and Caspase. Saeid Ghavami has researched Programmed cell death in several fields, including Viability assay and Glioma.
Saeid Ghavami focuses on Autophagy, Cell biology, Cancer research, Internal medicine and Apoptosis. Saeid Ghavami has researched Autophagy in several fields, including Cancer cell, Multicellular organism, Wnt signaling pathway and Programmed cell death. Saeid Ghavami combines subjects such as Cell culture and Autolysosome with his study of Programmed cell death.
His Cell biology study integrates concerns from other disciplines, such as Endocytosis and Induced pluripotent stem cell, Embryoid body. Saeid Ghavami studied Internal medicine and Oncology that intersect with Meta-analysis and Allele. His study in the field of Cell cycle also crosses realms of G2/M Arrest.
His main research concerns Autophagy, Programmed cell death, Apoptosis, Pharmacology and Cancer research. His Autophagy research is multidisciplinary, relying on both Computational biology, Mitochondrion and Insulin resistance. His work carried out in the field of Programmed cell death brings together such families of science as Multicellular organism and Autolysosome.
In his research on the topic of Apoptosis, Cancer therapy, Prenylation, Cancer cell, Statin and Cancer is strongly related with Chemotherapy. His work deals with themes such as Cell culture, Cell, Cell therapy, In vitro toxicology and Cytotoxicity, which intersect with Cancer research. As a part of the same scientific family, Saeid Ghavami mostly works in the field of Unfolded protein response, focusing on Endocytosis and, on occasion, Cell biology.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)
Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Autophagy (2012)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Autophagy and apoptosis dysfunction in neurodegenerative disorders.
Saeid Ghavami;Shahla Shojaei;Behzad Yeganeh;Sudharsana R. Ande.
Progress in Neurobiology (2014)
Apoptosis and cancer: mutations within caspase genes
Saeid Ghavami;Mohammad Hashemi;Sudharsana R Ande;Behzad Yeganeh.
Journal of Medical Genetics (2009)
Beclin 1 and autophagy are required for the tumorigenicity of breast cancer stem-like/progenitor cells
C Gong;C Bauvy;C Bauvy;G Tonelli;G Tonelli;W Yue;W Yue.
Oncogene (2013)
S100A8/A9 at low concentration promotes tumor cell growth via RAGE ligation and MAP kinase-dependent pathway
Saeid Ghavami;Iran Rashedi;Brian M. Dattilo;Mehdi Eshraghi.
Journal of Leukocyte Biology (2008)
Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
S100A8/A9 induces autophagy and apoptosis via ROS-mediated cross-talk between mitochondria and lysosomes that involves BNIP3.
Saeid Ghavami;Mehdi Eshragi;Sudharsana R Ande;Walter J Chazin.
Cell Research (2010)
Mechanism of apoptosis induced by S100A8/A9 in colon cancer cell lines: the role of ROS and the effect of metal ions
Saeid Ghavami;Claus Kerkhoff;Marek Los;Mohammad Hashemi.
Journal of Leukocyte Biology (2004)
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