2023 - Research.com Biology and Biochemistry in Hungary Leader Award
2022 - Research.com Biology and Biochemistry in Hungary Leader Award
Gábor Juhász spends much of his time researching Autophagy, Cell biology, Programmed cell death, Neuroscience and Endosome. His Autophagy study incorporates themes from Cell growth, Function, Signal transduction, Gene and Kinase. His Cell biology study combines topics from a wide range of disciplines, such as Endocytic cycle, Biochemistry, Autophagosome and BAG3.
His research investigates the link between Programmed cell death and topics such as Effector that cross with problems in PI3K/AKT/mTOR pathway and Ecdysone. His Neuroscience research incorporates elements of Chaperone-mediated autophagy, Phagocytosis, Autophagy database and Compartment. The Chaperone-mediated autophagy study combines topics in areas such as MAP1LC3B, BECN1 and Autolysosome.
His primary scientific interests are in Cell biology, Autophagy, Neuroscience, Biochemistry and Endosome. His Cell biology research includes themes of Endocytic cycle, Endocytosis, Lysosome and Drosophila Protein. His work carried out in the field of Autophagy brings together such families of science as Mutant and Programmed cell death.
His work in Electroencephalography, Electrophysiology, Premovement neuronal activity, Wakefulness and Prefrontal cortex are all subfields of Neuroscience research. His studies in Endosome integrate themes in fields like Phagosome, Small GTPase and UVRAG. His Nucleoside study also includes
Cell biology, Autophagy, Endosome, Lysosome and Neurodegeneration are his primary areas of study. His research integrates issues of Immune system, Mitophagy and Longevity in his study of Cell biology. His work in Autophagosome and Microautophagy is related to Autophagy.
The study incorporates disciplines such as Endocytic cycle, Endocytosis, Salivary gland and Protein subunit in addition to Endosome. His Lysosome research is multidisciplinary, relying on both Regulator, Disease, Small GTPase and Motility. His studies deal with areas such as Synapse, Neuroscience and Synaptosome as well as Neurodegeneration.
His primary areas of investigation include Cell biology, Autophagy, Neurodegeneration, Lysosome and Longevity. His Cell biology research is multidisciplinary, incorporating perspectives in Heat shock protein, Endocytic cycle, Endocytosis and Biogenesis. His study of Microautophagy is a part of Autophagy.
Gábor Juhász has included themes like Cancer, Synapse, Inflammatory bowel disease and Pathogenesis in his Neurodegeneration study. His research in Lysosome intersects with topics in Vesicle tethering, Catabolic Process, Autophagosome formation and Autophagosome lysosome fusion, Autophagosome. His Longevity study integrates concerns from other disciplines, such as Function, Stem cell, Intracellular, Spermidine and Metabolism.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Molecular definitions of autophagy and related processes
Lorenzo Galluzzi;Lorenzo Galluzzi;Eric H. Baehrecke;Andrea Ballabio;Patricia Boya.
The EMBO Journal (2017)
Direct induction of autophagy by Atg1 inhibits cell growth and induces apoptotic cell death
Ryan C. Scott;Gábor Juhász;Thomas P. Neufeld.
Current Biology (2007)
Programmed Autophagy in the Drosophila Fat Body Is Induced by Ecdysone through Regulation of the PI3K Pathway
Tor Erik Rusten;Karine Lindmo;Gábor Juhász;Miklós Sass.
Developmental Cell (2004)
Atg7-dependent autophagy promotes neuronal health, stress tolerance, and longevity but is dispensable for metamorphosis in Drosophila
Gábor Juhász;Balázs Érdi;Miklós Sass;Thomas P. Neufeld.
Genes & Development (2007)
The class III PI(3)K Vps34 promotes autophagy and endocytosis but not TOR signaling in Drosophila
Gábor Juhász;Jahda H. Hill;Ying Yan;Miklós Sass.
Journal of Cell Biology (2008)
Autophagosome-lysosome fusion is independent of V-ATPase-mediated acidification.
Caroline C Mauvezin;Péter Nagy;Gábor Juhász;Thomas P Neufeld.
Nature Communications (2015)
Microenvironmental autophagy promotes tumour growth.
Nadja S. Katheder;Nadja S. Katheder;Rojyar Khezri;Rojyar Khezri;Fergal O’Farrell;Fergal O’Farrell;Sebastian W. Schultz;Sebastian W. Schultz.
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