2019 - Fellow of the American Association for the Advancement of Science (AAAS)
Nissim Hay mainly focuses on Cell biology, Protein kinase B, PI3K/AKT/mTOR pathway, Cancer cell and Kinase. His Cell biology research is multidisciplinary, relying on both Hexokinase, Programmed cell death and Carcinogenesis. His Protein kinase B study frequently draws connections between adjacent fields such as Mitochondrion.
His PI3K/AKT/mTOR pathway study frequently draws connections between related disciplines such as Phosphorylation. The study incorporates disciplines such as Oxidative stress, Carbohydrate metabolism, Pentose phosphate pathway and Cancer research in addition to Cancer cell. The various areas that Nissim Hay examines in his Cancer research study include Lung cancer, Breast cancer and Tumor initiation.
His primary scientific interests are in Protein kinase B, Cancer research, Cell biology, PI3K/AKT/mTOR pathway and AKT1. Nissim Hay interconnects Molecular biology and Kinase in the investigation of issues within Protein kinase B. His work deals with themes such as Cancer cell, Cancer, Carcinogenesis, Apoptosis and PTEN, which intersect with Cancer research.
His Cell biology research incorporates elements of Biochemistry and Programmed cell death. The concepts of his PI3K/AKT/mTOR pathway study are interwoven with issues in Platelet activation and Effector. His research integrates issues of Platelet, Internal medicine, Endocrinology and Cyclin D1 in his study of AKT1.
His primary areas of investigation include Cancer research, Protein kinase B, Glycolysis, Metastasis and Cell biology. Nissim Hay combines subjects such as Cancer, Cancer metabolism and Programmed cell death with his study of Cancer research. His work in Cancer addresses issues such as Phosphorylation, which are connected to fields such as Kinase.
Nissim Hay specializes in Protein kinase B, namely AKT2. His Glycolysis research is multidisciplinary, incorporating perspectives in Oxidative phosphorylation and Glucokinase. His work on Serine as part of his general Cell biology study is frequently connected to Glutaminase, thereby bridging the divide between different branches of science.
Cell biology, Cancer research, Hexokinase, Oxidative phosphorylation and Glycolysis are his primary areas of study. His work on AKT2, Protein kinase B and Metabolic Stress as part of general Cell biology study is frequently linked to Nutrient stress, bridging the gap between disciplines. His biological study focuses on AKT1.
His Cancer research study combines topics from a wide range of disciplines, such as Inflammation, Transfection and Arthritis. His Hexokinase research is multidisciplinary, incorporating elements of mTORC1, Gene silencing, Glucokinase and Programmed cell death. His work carried out in the field of Glycolysis brings together such families of science as Glycine, Citric acid cycle, Serine and Biosynthesis.
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Upstream and downstream of mTOR
Nissim Hay;Nahum Sonenberg.
Genes & Development (2004)
The PI 3-kinase/Akt signaling pathway delivers an anti-apoptotic signal.
Scott G. Kennedy;Andrew J. Wagner;Suzanne D. Conzen;Joaquin Jordan.
Genes & Development (1997)
Growth retardation and increased apoptosis in mice with homozygous disruption of the akt1 gene
William S. Chen;Pei Zhang Xu;Kathrin Gottlob;Mei Ling Chen.
Genes & Development (2001)
The regulation and activities of the multifunctional serine/threonine kinase Akt/PKB.
Eugene S. Kandel;Nissim Hay.
Experimental Cell Research (1999)
4E-BP1, a repressor of mRNA translation, is phosphorylated and inactivated by the Akt(PKB) signaling pathway
Anne Claude Gingras;Scott G. Kennedy;Maura A. O'Leary;Nahum Sonenberg.
Genes & Development (1998)
Inhibition of early apoptotic events by Akt/PKB is dependent on the first committed step of glycolysis and mitochondrial hexokinase
Kathrin Gottlob;Nathan Majewski;Scott Kennedy;Scott Kennedy;Eugene Kandel.
Genes & Development (2001)
Dwarfism, impaired skin development, skeletal muscle atrophy, delayed bone development, and impeded adipogenesis in mice lacking Akt1 and Akt2
Xiao ding Peng;Pei Zhang Xu;Mei Ling Chen;Annett Hahn-Windgassen.
Genes & Development (2003)
The two TORCs and Akt.
Prashanth T. Bhaskar;Nissim Hay.
Developmental Cell (2007)
AMPK regulates NADPH homeostasis to promote tumour cell survival during energy stress
Sang Min Jeon;Navdeep S. Chandel;Nissim Hay.
The Akt-mTOR tango and its relevance to cancer.
Cancer Cell (2005)
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