D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 105 Citations 47,733 368 World Ranking 550 National Ranking 9
Medicine D-index 111 Citations 48,957 354 World Ranking 2280 National Ranking 58

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Internal medicine

His primary areas of investigation include AMPK, AMP-activated protein kinase, Internal medicine, Endocrinology and Protein kinase A. His AMPK research incorporates themes from Metformin, Cell growth and Skeletal muscle. His AMP-activated protein kinase study incorporates themes from Glycolysis, mTORC1, Lipogenesis and Oxidative phosphorylation.

His Endocrinology research is multidisciplinary, incorporating perspectives in Gene expression and Mitochondrial biogenesis. His Protein kinase A research is multidisciplinary, incorporating elements of Adenylate kinase, Neuroprotection and Adenosine monophosphate. His Cell biology research integrates issues from Proinflammatory cytokine and Energy homeostasis.

His most cited work include:

  • AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1 (3674 citations)
  • Phosphorylation of ULK1 (hATG1) by AMP-Activated Protein Kinase Connects Energy Sensing to Mitophagy (1638 citations)
  • Cellular and molecular mechanisms of metformin: an overview (1062 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are AMPK, AMP-activated protein kinase, Protein kinase A, Internal medicine and Endocrinology. His research on AMPK often connects related areas such as Skeletal muscle. His study in AMP-activated protein kinase is interdisciplinary in nature, drawing from both Glycolysis, Enzyme activator, Signal transduction, Oxidative phosphorylation and Mitochondrion.

In his research, Carcinogenesis is intimately related to Cancer research, which falls under the overarching field of Protein kinase A. His studies in Metformin, Lipid metabolism, Insulin receptor, Glucose transporter and Beta oxidation are all subfields of Internal medicine research. His Cell biology research focuses on Transcription factor and how it connects with Gene expression.

He most often published in these fields:

  • AMPK (166.93%)
  • AMP-activated protein kinase (98.58%)
  • Protein kinase A (98.89%)

What were the highlights of his more recent work (between 2016-2021)?

  • AMPK (166.93%)
  • Cell biology (84.34%)
  • Protein kinase A (98.89%)

In recent papers he was focusing on the following fields of study:

Benoit Viollet mainly investigates AMPK, Cell biology, Protein kinase A, Internal medicine and Endocrinology. A large part of his AMPK studies is devoted to AMP-activated protein kinase. His Cell biology study incorporates themes from Autophagy, Inflammation and Gene knockdown.

Benoit Viollet has researched Protein kinase A in several fields, including Beta oxidation, Lipogenesis, Activator and Energy homeostasis. His study looks at the relationship between Internal medicine and fields such as Lipid oxidation, as well as how they intersect with chemical problems. His research in Endocrinology tackles topics such as Knockout mouse which are related to areas like NADPH oxidase, Integrin alpha M, Downregulation and upregulation and Endothelial dysfunction.

Between 2016 and 2021, his most popular works were:

  • AMPK in skeletal muscle function and metabolism. (128 citations)
  • AMPK in skeletal muscle function and metabolism. (128 citations)
  • AMPK in skeletal muscle function and metabolism. (128 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Internal medicine

Benoit Viollet mostly deals with AMPK, Cell biology, Protein kinase A, Metformin and Endocrinology. His studies in AMPK integrate themes in fields like Diabetes mellitus, Carbohydrate metabolism, Glucose uptake and Skeletal muscle. The study incorporates disciplines such as Inflammation, Dendritic cell and Autophagy in addition to Cell biology.

Benoit Viollet specializes in Protein kinase A, namely AMP-activated protein kinase. His Metformin research includes themes of Cancer, Methylation and Pharmacology. His Endocrinology study frequently links to related topics such as Internal medicine.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1

Joungmok Kim;Mondira Kundu;Benoit Viollet;Kun-Liang Guan.
Nature Cell Biology (2011)

4712 Citations

Phosphorylation of ULK1 (hATG1) by AMP-Activated Protein Kinase Connects Energy Sensing to Mitophagy

Daniel F. Egan;David B. Shackelford;Maria M. Mihaylova;Sara R. Gelino.
Science (2011)

2060 Citations

Cellular and molecular mechanisms of metformin: an overview

Benoit Viollet;Benoit Viollet;Benoit Viollet;Bruno Guigas;Nieves Sanz Garcia;Nieves Sanz Garcia;Nieves Sanz Garcia;Jocelyne Leclerc;Jocelyne Leclerc;Jocelyne Leclerc.
Clinical Science (2012)

1629 Citations

Metformin inhibits hepatic gluconeogenesis in mice independently of the LKB1/AMPK pathway via a decrease in hepatic energy state.

Marc Foretz;Sophie Hébrard;Jocelyne Leclerc;Elham Zarrinpashneh.
Journal of Clinical Investigation (2010)

1177 Citations

Systemic Treatment with the Antidiabetic Drug Metformin Selectively Impairs p53-Deficient Tumor Cell Growth

Monica Buzzai;Russell G. Jones;Ravi K. Amaravadi;Julian J. Lum.
Cancer Research (2007)

998 Citations

Metformin: From Mechanisms of Action to Therapies

Marc Foretz;Marc Foretz;Marc Foretz;Bruno Guigas;Luc Bertrand;Michael Pollak.
Cell Metabolism (2014)

795 Citations

AMP-Activated Protein Kinase–Deficient Mice Are Resistant to the Metabolic Effects of Resveratrol

Jee Hyun Um;Sung-Jun Park;Hyeog Kang;Shutong Yang.
Diabetes (2010)

778 Citations

Knockout of the α2 but Not α1 5′-AMP-activated Protein Kinase Isoform Abolishes 5-Aminoimidazole-4-carboxamide-1-β-4-ribofuranosidebut Not Contraction-induced Glucose Uptake in Skeletal Muscle

Sebastian B. Jørgensen;Benoit Viollet;Fabrizio Andreelli;Christian Frøsig.
Journal of Biological Chemistry (2004)

773 Citations

Metformin, Independent of AMPK, Inhibits mTORC1 in a Rag GTPase-Dependent Manner

Adem Kalender;Anand Selvaraj;Anand Selvaraj;So Young Kim;So Young Kim;Pawan Gulati.
Cell Metabolism (2010)

766 Citations

Biguanides suppress hepatic glucagon signalling by decreasing production of cyclic AMP

Russell A. Miller;Qingwei Chu;Jianxin Xie;Marc Foretz;Marc Foretz;Marc Foretz.
Nature (2013)

744 Citations

Best Scientists Citing Benoit Viollet

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Russell G. Jones

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Michael Pollak

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Profile was last updated on December 6th, 2021.
Research.com Ranking is based on data retrieved from the Microsoft Academic Graph (MAG).
The ranking d-index is inferred from publications deemed to belong to the considered discipline.

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