His primary areas of investigation include AMPK, AMP-activated protein kinase, Internal medicine, Endocrinology and Protein kinase A. His AMPK research incorporates themes from Metformin, Cell growth and Skeletal muscle. His AMP-activated protein kinase study incorporates themes from Glycolysis, mTORC1, Lipogenesis and Oxidative phosphorylation.
His Endocrinology research is multidisciplinary, incorporating perspectives in Gene expression and Mitochondrial biogenesis. His Protein kinase A research is multidisciplinary, incorporating elements of Adenylate kinase, Neuroprotection and Adenosine monophosphate. His Cell biology research integrates issues from Proinflammatory cytokine and Energy homeostasis.
His primary areas of study are AMPK, AMP-activated protein kinase, Protein kinase A, Internal medicine and Endocrinology. His research on AMPK often connects related areas such as Skeletal muscle. His study in AMP-activated protein kinase is interdisciplinary in nature, drawing from both Glycolysis, Enzyme activator, Signal transduction, Oxidative phosphorylation and Mitochondrion.
In his research, Carcinogenesis is intimately related to Cancer research, which falls under the overarching field of Protein kinase A. His studies in Metformin, Lipid metabolism, Insulin receptor, Glucose transporter and Beta oxidation are all subfields of Internal medicine research. His Cell biology research focuses on Transcription factor and how it connects with Gene expression.
Benoit Viollet mainly investigates AMPK, Cell biology, Protein kinase A, Internal medicine and Endocrinology. A large part of his AMPK studies is devoted to AMP-activated protein kinase. His Cell biology study incorporates themes from Autophagy, Inflammation and Gene knockdown.
Benoit Viollet has researched Protein kinase A in several fields, including Beta oxidation, Lipogenesis, Activator and Energy homeostasis. His study looks at the relationship between Internal medicine and fields such as Lipid oxidation, as well as how they intersect with chemical problems. His research in Endocrinology tackles topics such as Knockout mouse which are related to areas like NADPH oxidase, Integrin alpha M, Downregulation and upregulation and Endothelial dysfunction.
Benoit Viollet mostly deals with AMPK, Cell biology, Protein kinase A, Metformin and Endocrinology. His studies in AMPK integrate themes in fields like Diabetes mellitus, Carbohydrate metabolism, Glucose uptake and Skeletal muscle. The study incorporates disciplines such as Inflammation, Dendritic cell and Autophagy in addition to Cell biology.
Benoit Viollet specializes in Protein kinase A, namely AMP-activated protein kinase. His Metformin research includes themes of Cancer, Methylation and Pharmacology. His Endocrinology study frequently links to related topics such as Internal medicine.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1
Joungmok Kim;Mondira Kundu;Benoit Viollet;Kun-Liang Guan.
Nature Cell Biology (2011)
Phosphorylation of ULK1 (hATG1) by AMP-Activated Protein Kinase Connects Energy Sensing to Mitophagy
Daniel F. Egan;David B. Shackelford;Maria M. Mihaylova;Sara R. Gelino.
Cellular and molecular mechanisms of metformin: an overview
Benoit Viollet;Benoit Viollet;Benoit Viollet;Bruno Guigas;Nieves Sanz Garcia;Nieves Sanz Garcia;Nieves Sanz Garcia;Jocelyne Leclerc;Jocelyne Leclerc;Jocelyne Leclerc.
Clinical Science (2012)
Metformin inhibits hepatic gluconeogenesis in mice independently of the LKB1/AMPK pathway via a decrease in hepatic energy state.
Marc Foretz;Sophie Hébrard;Jocelyne Leclerc;Elham Zarrinpashneh.
Journal of Clinical Investigation (2010)
Systemic Treatment with the Antidiabetic Drug Metformin Selectively Impairs p53-Deficient Tumor Cell Growth
Monica Buzzai;Russell G. Jones;Ravi K. Amaravadi;Julian J. Lum.
Cancer Research (2007)
Metformin: From Mechanisms of Action to Therapies
Marc Foretz;Marc Foretz;Marc Foretz;Bruno Guigas;Luc Bertrand;Michael Pollak.
Cell Metabolism (2014)
AMP-Activated Protein Kinase–Deficient Mice Are Resistant to the Metabolic Effects of Resveratrol
Jee Hyun Um;Sung-Jun Park;Hyeog Kang;Shutong Yang.
Knockout of the α2 but Not α1 5′-AMP-activated Protein Kinase Isoform Abolishes 5-Aminoimidazole-4-carboxamide-1-β-4-ribofuranosidebut Not Contraction-induced Glucose Uptake in Skeletal Muscle
Sebastian B. Jørgensen;Benoit Viollet;Fabrizio Andreelli;Christian Frøsig.
Journal of Biological Chemistry (2004)
Metformin, Independent of AMPK, Inhibits mTORC1 in a Rag GTPase-Dependent Manner
Adem Kalender;Anand Selvaraj;Anand Selvaraj;So Young Kim;So Young Kim;Pawan Gulati.
Cell Metabolism (2010)
Biguanides suppress hepatic glucagon signalling by decreasing production of cyclic AMP
Russell A. Miller;Qingwei Chu;Jianxin Xie;Marc Foretz;Marc Foretz;Marc Foretz.
Profile was last updated on December 6th, 2021.
Research.com Ranking is based on data retrieved from the Microsoft Academic Graph (MAG).
The ranking d-index is inferred from publications deemed to belong to the considered discipline.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: