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D-Index & Metrics

Biology and Biochemistry

D-Index
72
Citations
19908
World Ranking
6263
National Ranking
2942

Overview

James G. Tidball is affiliated with the University of California, Los Angeles in the United States. Their research primarily revolves around muscle physiology and disorders, with a significant focus on the interactions between immune cells and muscle regeneration.

Their recent research papers include:

  • Skeletal muscle regeneration via the chemical induction and expansion of myogenic stem cells in situ or in vitro (2021, Nature Biomedical Engineering)
  • Aging of the immune system and impaired muscle regeneration: A failure of immunomodulation of adult myogenesis (2020, Experimental Gerontology)
  • Muscle inflammation is regulated by NF-κB from multiple cells to control distinct states of wasting in cancer cachexia (2024, Cell Reports)
  • Differential Effects of Myeloid Cell PPARδ and IL-10 in Regulating Macrophage Recruitment, Phenotype, and Regeneration following Acute Muscle Injury (2020, The Journal of Immunology)
  • Myeloid cell-specific mutation of Spi1 selectively reduces M2-biased macrophage numbers in skeletal muscle, reduces age-related muscle fibrosis and prevents sarcopenia (2022, Aging Cell)

The scientist collaborates frequently with other researchers, including:

  • Michelle Wehling-Henricks (10 coauthored papers)
  • Steven S. Welc (6 coauthored papers)
  • Makoto Kuro-o (4 coauthored papers)
  • Iván Flores (3 coauthored papers)
  • Denis C. Guttridge (3 coauthored papers)

Publications by James G. Tidball often appear in specialized venues such as:

  • Faculty Opinions - Post-Publication Peer Review of the Biomedical Literature (5 publications)
  • American Journal of Pathology (2 publications)
  • Nature Biomedical Engineering (1 publication)
  • Experimental Gerontology (1 publication)
  • Cell Reports (1 publication)

Their work covers multiple fields and subfields of study, notably:

  • Biochemistry, Genetics and Molecular Biology
  • Medicine
  • Molecular Biology
  • Genetics
  • Surgery
  • Physiology
  • Immunology

James G. Tidball's research addresses several main topics, including:

  • Muscle Physiology and Disorders
  • Nutrition and Health in Aging
  • Immune cells in cancer
  • Genetic Syndromes and Imprinting
  • Parathyroid Disorders and Treatments
  • Tissue Engineering and Regenerative Medicine
  • Exercise and Physiological Responses

Best Publications

  • Inflammatory processes in muscle injury and repair.

    James G. Tidball

  • Regulatory interactions between muscle and the immune system during muscle regeneration

    James G. Tidball;S. Armando Villalta

  • Inflammatory cell response to acute muscle injury.

    James G. Tidball

  • A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx mice

    Michelle Wehling;Melissa J. Spencer;James G. Tidball

  • Regulation of muscle growth and regeneration by the immune system

    James G. Tidball

  • Shifts in macrophage phenotypes and macrophage competition for arginine metabolism affect the severity of muscle pathology in muscular dystrophy

    S. Armando Villalta;Hal X. Nguyen;Bo Deng;Tomomi Gotoh

  • Interplay of IKK/NF-κB signaling in macrophages and myofibers promotes muscle degeneration in Duchenne muscular dystrophy

    Swarnali Acharyya;S. Armando Villalta;Nadine Bakkar;Tepmanas Bupha-Intr

  • IL-10 Triggers Changes in Macrophage Phenotype That Promote Muscle Growth and Regeneration

    Bo Deng;Michelle Wehling-Henricks;S. Armando Villalta;Ying Wang

  • Mechanisms of Muscle Injury, Repair, and Regeneration

    James G. Tidball

  • Macrophages promote muscle membrane repair and muscle fibre growth and regeneration during modified muscle loading in mice in vivo.

    James G. Tidball;Michelle Wehling-Henricks

  • Neuronal nitric oxide synthase and dystrophin-deficient muscular dystrophy

    Wen Jinn Chang;Susan T. Iannaccone;Kim S. Lau;Bettie Sue S Masters

  • Modulation of myostatin expression during modified muscle use

    Michelle Wehling;Baiyuan Cai;James G. Tidball

  • Interleukin-10 reduces the pathology of mdx muscular dystrophy by deactivating M1 macrophages and modulating macrophage phenotype

    S. Armando Villalta;Chiara Rinaldi;Bo Deng;Grace Liu

  • Differential response of macrophage subpopulations to soleus muscle reloading after rat hindlimb suspension

    B. A. St Pierre;J. G. Tidball

  • Helper (CD4(+)) and cytotoxic (CD8(+)) T cells promote the pathology of dystrophin-deficient muscle.

    Melissa J. Spencer;Encarnacion Montecino-Rodriguez;Kenneth Dorshkind;James G. Tidball

  • Dominant negative myostatin produces hypertrophy without hyperplasia in muscle.

    Xiaolei Zhu;Michele Hadhazy;Michelle Wehling;James G. Tidball

  • Mechanical loading regulates NOS expression and activity in developing and adult skeletal muscle

    James G. Tidball;Eliane Lavergne;Kim S. Lau;Melissa J. Spencer

  • Apoptosis precedes necrosis of dystrophin-deficient muscle

    J.G. Tidball;D.E. Albrecht;B.E. Lokensgard;M.J. Spencer

  • CALPAINS ARE ACTIVATED IN NECROTIC FIBERS FROM MDX DYSTROPHIC MICE

    Melissa J. Spencer;Dorothy E. Croall;James G. Tidball

  • The role of free radicals in the pathophysiology of muscular dystrophy

    James G. Tidball;Michelle Wehling-Henricks

Frequent Co-Authors

Melissa J. Spencer
Melissa J. Spencer University of California, Los Angeles
Makoto Kuro-o
Makoto Kuro-o Jichi Medical University
James T. Stull
James T. Stull The University of Texas Southwestern Medical Center
Tim Sparwasser
Tim Sparwasser Johannes Gutenberg University of Mainz
Jeffrey A. Bluestone
Jeffrey A. Bluestone University of California, San Francisco
Stanley C. Froehner
Stanley C. Froehner University of Washington
Louis M. Kunkel
Louis M. Kunkel Boston Children's Hospital
Sankar Ghosh
Sankar Ghosh Columbia University
Robert W. Taylor
Robert W. Taylor Newcastle University
Aldons J. Lusis
Aldons J. Lusis University of California, Los Angeles

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