D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 90 Citations 24,151 509 World Ranking 7776 National Ranking 209

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Gene
  • Cancer

Shin'ichi Takeda focuses on Skeletal muscle, Molecular biology, Muscular dystrophy, Duchenne muscular dystrophy and Dystrophin. His research integrates issues of Laminin, Gene expression and Mesenchymal stem cell, Cell biology in his study of Skeletal muscle. The Cell biology study combines topics in areas such as Inflammation, Immunology and Satellite.

His Muscular dystrophy research is multidisciplinary, relying on both Myogenesis and Anatomy. Shin'ichi Takeda combines subjects such as Endocrinology and Viral vector with his study of Duchenne muscular dystrophy. He interconnects Bioinformatics, Exon skipping, Genetic enhancement, Exon and Genetically modified mouse in the investigation of issues within Dystrophin.

His most cited work include:

  • Mesenchymal progenitors distinct from satellite cells contribute to ectopic fat cell formation in skeletal muscle (719 citations)
  • Bone marrow stromal cells generate muscle cells and repair muscle degeneration. (513 citations)
  • A mutation in the ceruloplasmin gene is associated with systemic hemosiderosis in humans. (386 citations)

What are the main themes of his work throughout his whole career to date?

Shin'ichi Takeda mostly deals with Duchenne muscular dystrophy, Muscular dystrophy, Dystrophin, Internal medicine and Skeletal muscle. The Duchenne muscular dystrophy study which covers Exon that intersects with RNA splicing. His studies in Muscular dystrophy integrate themes in fields like Laminin, Myogenesis, Genetic enhancement and Pathology.

His Dystrophin study combines topics in areas such as Molecular biology, Transgene and Muscle disorder. His work investigates the relationship between Internal medicine and topics such as Surgery that intersect with problems in Lung. Shin'ichi Takeda works mostly in the field of Skeletal muscle, limiting it down to concerns involving Cell biology and, occasionally, Immunology.

He most often published in these fields:

  • Duchenne muscular dystrophy (30.86%)
  • Muscular dystrophy (24.85%)
  • Dystrophin (22.04%)

What were the highlights of his more recent work (between 2015-2021)?

  • Duchenne muscular dystrophy (30.86%)
  • Dystrophin (22.04%)
  • Muscular dystrophy (24.85%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Duchenne muscular dystrophy, Dystrophin, Muscular dystrophy, Cell biology and Skeletal muscle. Duchenne muscular dystrophy is a subfield of Internal medicine that he explores. Shin'ichi Takeda has included themes like Endocrinology and Cardiology in his Internal medicine study.

His research integrates issues of In vivo, Genetic disorder, Transgene and Bioinformatics in his study of Dystrophin. His work in Cell biology addresses subjects such as Regulation of gene expression, which are connected to disciplines such as Transcriptional regulation. His Skeletal muscle research integrates issues from Progenitor cell, Gene expression, Downregulation and upregulation and RNA-binding protein.

Between 2015 and 2021, his most popular works were:

  • C9orf72 and RAB7L1 regulate vesicle trafficking in amyotrophic lateral sclerosis and frontotemporal dementia. (82 citations)
  • Recent advances in innovative therapeutic approaches for Duchenne muscular dystrophy: from discovery to clinical trials. (67 citations)
  • Mitochondria mediate cell membrane repair and contribute to Duchenne muscular dystrophy (65 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Cancer

His main research concerns Duchenne muscular dystrophy, Dystrophin, Muscular dystrophy, Exon skipping and Skeletal muscle. His Duchenne muscular dystrophy research includes elements of Cancer research, Endocrinology, Inflammation, Myogenesis and Exon. His Dystrophin research includes themes of Metabolic myopathy, Oxidative phosphorylation and Gene expression.

His research in Muscular dystrophy focuses on subjects like Immunology, which are connected to Satellite. His Skeletal muscle research is multidisciplinary, incorporating elements of Progenitor cell, Stem cell, Cell biology and Downregulation and upregulation. In the field of Cell biology, his study on Myocyte overlaps with subjects such as Sarcolipin.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Mesenchymal progenitors distinct from satellite cells contribute to ectopic fat cell formation in skeletal muscle.

Akiyoshi Uezumi;So Ichiro Fukada;Naoki Yamamoto;Shin'Ichi Takeda.
Nature Cell Biology (2010)

1009 Citations

Bone marrow stromal cells generate muscle cells and repair muscle degeneration.

Mari Dezawa;Hiroto Ishikawa;Yutaka Itokazu;Tomoyuki Yoshihara.
Science (2005)

776 Citations

A mutation in the ceruloplasmin gene is associated with systemic hemosiderosis in humans.

Kunihiro Yoshida;Kenichi Furihata;Shin'ichi Takeda;Akinori Nakamura.
Nature Genetics (1995)

550 Citations

Fibrosis and adipogenesis originate from a common mesenchymal progenitor in skeletal muscle.

Akiyoshi Uezumi;Takahito Ito;Daisuke Morikawa;Natsuko Shimizu.
Journal of Cell Science (2011)

516 Citations

Efficacy of systemic morpholino exon-skipping in Duchenne dystrophy dogs.

Toshifumi Yokota;Qi‐long Lu;Terence Partridge;Masanori Kobayashi.
Annals of Neurology (2009)

468 Citations

Molecular signature of quiescent satellite cells in adult skeletal muscle.

So-ichiro Fukada;Akiyoshi Uezumi;Madoka Ikemoto;Satoru Masuda.
Stem Cells (2007)

457 Citations

Up-regulation of monocyte chemoattractant protein-1 in tubulointerstitial lesions of human diabetic nephropathy

Takashi Wada;Kengo Furuichi;Norihiko Sakai;Yasunori Iwata.
Kidney International (2000)

448 Citations

Cardiac side population cells have a potential to migrate and differentiate into cardiomyocytes in vitro and in vivo

Tomomi Oyama;Toshio Nagai;Hiroshi Wada;Atsuhiko Thomas Naito.
Journal of Cell Biology (2007)

381 Citations

Benefits of surgery for patients with pulmonary metastases from colorectal carcinoma

Masayoshi Inoue;Mitsunori Ohta;Keiji Iuchi;Akihide Matsumura.
The Annals of Thoracic Surgery (2004)

353 Citations

Laminin α2 chain-null mutant mice by targeted disruption of the Lama2 gene: a new model of merosin (laminin 2)-deficient congenital muscular dystrophy

Yuko Miyagoe;Kazunori Hanaoka;Ikuya Nonaka;Michiko Hayasaka.
FEBS Letters (1997)

341 Citations

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