D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Genetics and Molecular Biology D-index 107 Citations 39,956 234 World Ranking 337 National Ranking 11

Research.com Recognitions

Awards & Achievements

2020 - Fellow of the Royal Society, United Kingdom

2006 - Fellow of the Royal Society of Canada Academy of Science

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • DNA

His primary areas of study are Cell biology, Myogenesis, Stem cell, Cellular differentiation and Skeletal muscle. His Cell biology study combines topics in areas such as Cell, Satellite and Anatomy. His Myogenesis study deals with the bigger picture of Genetics.

His studies in Stem cell integrate themes in fields like Immunology, Induced stem cells, Adult stem cell and Cell fate determination. His Cellular differentiation research is multidisciplinary, incorporating perspectives in Progenitor cell and Regeneration. In his study, which falls under the umbrella issue of Skeletal muscle, Molecular biology and Cell growth is strongly linked to Myocyte.

His most cited work include:

  • Cellular and Molecular Regulation of Muscle Regeneration (1993 citations)
  • Pax7 Is Required for the Specification of Myogenic Satellite Cells (1701 citations)
  • PRDM16 controls a brown fat/skeletal muscle switch (1697 citations)

What are the main themes of his work throughout his whole career to date?

Michael A. Rudnicki mainly focuses on Cell biology, Stem cell, Skeletal muscle, Myogenesis and Myocyte. His Cell biology research integrates issues from Cellular differentiation, Adult stem cell and Satellite. The various areas that Michael A. Rudnicki examines in his Stem cell study include Endothelial stem cell, Cell, Cell type, Immunology and Induced stem cells.

His Skeletal muscle research includes elements of Asymmetric cell division and Cell growth. His Myogenesis study is concerned with Genetics in general. His work carried out in the field of Myocyte brings together such families of science as Duchenne muscular dystrophy and Dystrophin.

He most often published in these fields:

  • Cell biology (73.62%)
  • Stem cell (50.75%)
  • Skeletal muscle (45.48%)

What were the highlights of his more recent work (between 2015-2021)?

  • Cell biology (73.62%)
  • Stem cell (50.75%)
  • Skeletal muscle (45.48%)

In recent papers he was focusing on the following fields of study:

Cell biology, Stem cell, Skeletal muscle, Myocyte and Progenitor cell are his primary areas of study. Michael A. Rudnicki has researched Cell biology in several fields, including Cellular differentiation, Adult stem cell and Transplantation. The Stem cell study combines topics in areas such as Cell, Satellite, Transcription factor and Duchenne muscular dystrophy, Dystrophin.

His Skeletal muscle study incorporates themes from Cell activation, Regulation of gene expression, Asymmetric cell division and Regeneration. His Myocyte study deals with Cell type intersecting with RSPO1. The study incorporates disciplines such as PAX7 and Myogenin in addition to MyoD.

Between 2015 and 2021, his most popular works were:

  • The myogenic regulatory factors, determinants of muscle development, cell identity and regeneration. (125 citations)
  • The myogenic regulatory factors, determinants of muscle development, cell identity and regeneration. (125 citations)
  • Loss of fibronectin from the aged stem cell niche affects the regenerative capacity of skeletal muscle in mice. (121 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • DNA

Michael A. Rudnicki spends much of his time researching Cell biology, Stem cell, Skeletal muscle, Duchenne muscular dystrophy and Regeneration. His research in Cell biology intersects with topics in Chromatin and Adult stem cell. His study in Stem cell is interdisciplinary in nature, drawing from both Cytokine receptor, STAT3, STAT3 Transcription Factor, Myocyte and Neuroscience.

His research in the fields of MYF5 overlaps with other disciplines such as Growth differentiation factor. His Myogenesis research includes themes of Adipogenesis and Cellular differentiation. His Cellular differentiation research incorporates themes from Epigenetic regulation of neurogenesis and Histone deacetylase 5.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Cellular and Molecular Regulation of Muscle Regeneration

Sophie B. P. Chargé;Michael A. Rudnicki.
Physiological Reviews (2004)

2892 Citations

Pax7 Is Required for the Specification of Myogenic Satellite Cells

Patrick Seale;Luc A Sabourin;Adele Girgis-Gabardo;Ahmed Mansouri.
Cell (2000)

2226 Citations

PRDM16 controls a brown fat/skeletal muscle switch

Patrick Seale;Bryan Bjork;Wenli Yang;Shingo Kajimura.
Nature (2008)

2204 Citations

MyoD or Myf-5 is required for the formation of skeletal muscle

Michael A. Rudnicki;Patrick N.J. Schnegelsberg;Ronald H. Stead;Thomas Braun.
Cell (1993)

1894 Citations

Simplified mammalian DNA isolation procedure

Peter W. Laird;Alice Zijderveld;Koert Linders;Michael A. Rudnicki.
Nucleic Acids Research (1991)

1730 Citations

Satellite Cells and the Muscle Stem Cell Niche

Hang Yin;Feodor Price;Michael A. Rudnicki.
Physiological Reviews (2013)

1355 Citations

Asymmetric Self-Renewal and Commitment of Satellite Stem Cells in Muscle

Shihuan Kuang;Kazuki Kuroda;Fabien Le Grand;Michael A. Rudnicki.
Cell (2007)

1276 Citations

Mutations in T-cell antigen receptor genes α and β block thymocyte development at different stages

Peter Mombaerts;Alan R. Clarke;Michael A. Rudnicki;John Iacomini.
Nature (1992)

1208 Citations

Inactivation of MyoD in mice leads to up-regulation of the myogenic HLH gene Myf-5 and results in apparently normal muscle development.

Michael A. Rudnicki;Thomas Braun;Shuji Hinuma;Rudolf Jaenisch.
Cell (1992)

1151 Citations

Muscle injury activates resident fibro/adipogenic progenitors that facilitate myogenesis

Aaron W. B. Joe;Lin Yi;Anuradha Natarajan;Fabien Le Grand.
Nature Cell Biology (2010)

1074 Citations

Editorial Boards

Skeletal Muscle
(Impact Factor: 5.063)

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