2023 - Research.com Molecular Biology in Canada Leader Award
2020 - Fellow of the Royal Society, United Kingdom
2006 - Fellow of the Royal Society of Canada Academy of Science
His primary areas of study are Cell biology, Myogenesis, Stem cell, Cellular differentiation and Skeletal muscle. His Cell biology study combines topics in areas such as Cell, Satellite and Anatomy. His Myogenesis study deals with the bigger picture of Genetics.
His studies in Stem cell integrate themes in fields like Immunology, Induced stem cells, Adult stem cell and Cell fate determination. His Cellular differentiation research is multidisciplinary, incorporating perspectives in Progenitor cell and Regeneration. In his study, which falls under the umbrella issue of Skeletal muscle, Molecular biology and Cell growth is strongly linked to Myocyte.
Michael A. Rudnicki mainly focuses on Cell biology, Stem cell, Skeletal muscle, Myogenesis and Myocyte. His Cell biology research integrates issues from Cellular differentiation, Adult stem cell and Satellite. The various areas that Michael A. Rudnicki examines in his Stem cell study include Endothelial stem cell, Cell, Cell type, Immunology and Induced stem cells.
His Skeletal muscle research includes elements of Asymmetric cell division and Cell growth. His Myogenesis study is concerned with Genetics in general. His work carried out in the field of Myocyte brings together such families of science as Duchenne muscular dystrophy and Dystrophin.
Cell biology, Stem cell, Skeletal muscle, Myocyte and Progenitor cell are his primary areas of study. Michael A. Rudnicki has researched Cell biology in several fields, including Cellular differentiation, Adult stem cell and Transplantation. The Stem cell study combines topics in areas such as Cell, Satellite, Transcription factor and Duchenne muscular dystrophy, Dystrophin.
His Skeletal muscle study incorporates themes from Cell activation, Regulation of gene expression, Asymmetric cell division and Regeneration. His Myocyte study deals with Cell type intersecting with RSPO1. The study incorporates disciplines such as PAX7 and Myogenin in addition to MyoD.
Michael A. Rudnicki spends much of his time researching Cell biology, Stem cell, Skeletal muscle, Duchenne muscular dystrophy and Regeneration. His research in Cell biology intersects with topics in Chromatin and Adult stem cell. His study in Stem cell is interdisciplinary in nature, drawing from both Cytokine receptor, STAT3, STAT3 Transcription Factor, Myocyte and Neuroscience.
His research in the fields of MYF5 overlaps with other disciplines such as Growth differentiation factor. His Myogenesis research includes themes of Adipogenesis and Cellular differentiation. His Cellular differentiation research incorporates themes from Epigenetic regulation of neurogenesis and Histone deacetylase 5.
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Cellular and Molecular Regulation of Muscle Regeneration
Sophie B. P. Chargé;Michael A. Rudnicki.
Physiological Reviews (2004)
Pax7 is required for the specification of myogenic satellite cells.
Patrick Seale;Luc A Sabourin;Adele Girgis-Gabardo;Ahmed Mansouri.
PRDM16 controls a brown fat/skeletal muscle switch
Patrick Seale;Bryan Bjork;Wenli Yang;Shingo Kajimura.
MyoD or Myf-5 is required for the formation of skeletal muscle
Michael A. Rudnicki;Patrick N.J. Schnegelsberg;Ronald H. Stead;Thomas Braun.
Simplified mammalian DNA isolation procedure.
Peter W. Laird;Alice Zijderveld;Koert Linders;Michael A. Rudnicki.
Nucleic Acids Research (1991)
Satellite Cells and the Muscle Stem Cell Niche
Hang Yin;Feodor Price;Michael A. Rudnicki.
Physiological Reviews (2013)
Asymmetric Self-Renewal and Commitment of Satellite Stem Cells in Muscle
Shihuan Kuang;Kazuki Kuroda;Fabien Le Grand;Michael A. Rudnicki.
Muscle injury activates resident fibro/adipogenic progenitors that facilitate myogenesis
Aaron W. B. Joe;Lin Yi;Anuradha Natarajan;Fabien Le Grand.
Nature Cell Biology (2010)
Mutations in T-cell antigen receptor genes α and β block thymocyte development at different stages
Peter Mombaerts;Alan R. Clarke;Michael A. Rudnicki;John Iacomini.
Inactivation of MyoD in mice leads to up-regulation of the myogenic HLH gene Myf-5 and results in apparently normal muscle development.
Michael A. Rudnicki;Thomas Braun;Shuji Hinuma;Rudolf Jaenisch.
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