What is he best known for?
The fields of study he is best known for:
- DNA
- Internal medicine
- Apoptosis
Pura Muñoz-Cánoves mainly focuses on Skeletal muscle, Cell biology, Stem cell, Fibrosis and Myocyte.
He combines subjects such as Inflammation and Duchenne muscular dystrophy with his study of Skeletal muscle.
His work investigates the relationship between Cell biology and topics such as Immunology that intersect with problems in Tissue homeostasis.
The Stem cell study combines topics in areas such as Sarcopenia, Extracellular matrix and Senescence.
Pura Muñoz-Cánoves has researched Fibrosis in several fields, including Muscular dystrophy and ITGA7.
As a member of one scientific family, Pura Muñoz-Cánoves mostly works in the field of Myocyte, focusing on Molecular biology and, on occasion, C2C12, Myogenin and MyoD.
His most cited work include:
- Autophagy maintains stemness by preventing senescence (612 citations)
- Geriatric muscle stem cells switch reversible quiescence into senescence (525 citations)
- Geriatric muscle stem cells switch reversible quiescence into senescence (525 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Cell biology, Skeletal muscle, Stem cell, Regeneration and Myogenesis.
His Cell biology research incorporates themes from Autophagy, Cell, Stem cell theory of aging and Chromatin.
His Skeletal muscle study integrates concerns from other disciplines, such as Fibrosis, Sarcopenia, Duchenne muscular dystrophy and Inflammation.
His Stem cell study combines topics in areas such as Senescence, Tissue homeostasis, Neuroscience and Adult stem cell.
His work carried out in the field of Regeneration brings together such families of science as Homeostasis, Bioinformatics and Pathology.
His work in Myogenesis addresses subjects such as Molecular biology, which are connected to disciplines such as Plasminogen activator and Signal transduction.
He most often published in these fields:
- Cell biology (90.91%)
- Skeletal muscle (85.71%)
- Stem cell (65.58%)
What were the highlights of his more recent work (between 2018-2021)?
- Cell biology (90.91%)
- Skeletal muscle (85.71%)
- Stem cell (65.58%)
In recent papers he was focusing on the following fields of study:
His main research concerns Cell biology, Skeletal muscle, Stem cell, Regeneration and Autophagy.
His research integrates issues of Transcriptome and Ageing in his study of Cell biology.
His study in Skeletal muscle is interdisciplinary in nature, drawing from both Sarcopenia, Progenitor cell, Translation, Five prime untranslated region and Effector.
Pura Muñoz-Cánoves combines subjects such as Niche, Cell sorting and Adult stem cell with his study of Stem cell.
His Regeneration research includes themes of Cripto, Duchenne muscular dystrophy, SMAD and Neuroscience.
His research in Autophagy tackles topics such as Proteostasis which are related to areas like Cell quiescence and Autophagosome.
Between 2018 and 2021, his most popular works were:
- A Network of Macrophages Supports Mitochondrial Homeostasis in the Heart (43 citations)
- A Network of Macrophages Supports Mitochondrial Homeostasis in the Heart (43 citations)
- Sestrin prevents atrophy of disused and aging muscles by integrating anabolic and catabolic signals (39 citations)
In his most recent research, the most cited papers focused on:
- DNA
- Internal medicine
- Apoptosis
Pura Muñoz-Cánoves spends much of his time researching Cell biology, Autophagy, Skeletal muscle, Homeostasis and Sarcopenia.
Many of his studies involve connections with topics such as Ageing and Cell biology.
The various areas that he examines in his Autophagy study include Computational biology and Programmed cell death.
His work deals with themes such as Young age, Tissue repair, Stem cell, Regeneration and Cell Maintenance, which intersect with Skeletal muscle.
His research in Homeostasis intersects with topics in Receptor, Inflammasome, Proteostasis and Mitochondrion.
His studies deal with areas such as Catabolism, mTORC1, Anabolism, Wasting and Atrophy as well as Sarcopenia.
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