What is he best known for?
The fields of study he is best known for:
His scientific interests lie mostly in Skeletal muscle, Myocyte, Myogenesis, Cell biology and Endocrinology.
His study in Skeletal muscle is interdisciplinary in nature, drawing from both Fibrosis, Myosin, Duchenne muscular dystrophy and Decorin.
He interconnects Muscular dystrophy, Connective tissue and CTGF in the investigation of issues within Fibrosis.
His studies deal with areas such as Glypican and Cellular differentiation as well as Myogenesis.
His study in Biglycan and Extracellular matrix is done as part of Cell biology.
His research on Endocrinology often connects related topics like Internal medicine.
His most cited work include:
- Anchorage of collagen-tailed acetylcholinesterase to the extracellular matrix is mediated by heparan sulfate proteoglycans. (142 citations)
- Decorin Core Protein Fragment Leu155-Val260 Interacts with TGF-β but Does Not Compete for Decorin Binding to Type I Collagen (142 citations)
- ECM is required for skeletal muscle differentiation independently of muscle regulatory factor expression (136 citations)
What are the main themes of his work throughout his whole career to date?
Skeletal muscle, Cell biology, Biochemistry, Fibrosis and Internal medicine are his primary areas of study.
His Skeletal muscle research is multidisciplinary, relying on both Myocyte, Extracellular matrix and Myosin.
His Cell biology research incorporates elements of Stromal cell and Cellular differentiation.
The study incorporates disciplines such as mdx mouse, Duchenne muscular dystrophy, Connective tissue and CTGF in addition to Fibrosis.
Enrique Brandan frequently studies issues relating to Endocrinology and Internal medicine.
His Myogenesis study combines topics from a wide range of disciplines, such as Glypican, Molecular biology and Receptor.
He most often published in these fields:
- Skeletal muscle (47.02%)
- Cell biology (37.09%)
- Biochemistry (28.48%)
What were the highlights of his more recent work (between 2016-2021)?
- Skeletal muscle (47.02%)
- Fibrosis (31.79%)
- Cell biology (37.09%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Skeletal muscle, Fibrosis, Cell biology, CTGF and Extracellular matrix.
He combines subjects such as Denervation, Progenitor cell, Connective tissue, Pathology and Muscular dystrophy with his study of Skeletal muscle.
Internal medicine covers Enrique Brandan research in Fibrosis.
In his research, Wnt signaling pathway and Transcription factor is intimately related to Stromal cell, which falls under the overarching field of Cell biology.
The various areas that Enrique Brandan examines in his CTGF study include Skeletal muscle fibrosis, Duchenne muscular dystrophy, Hypoxia and Matricellular protein.
His Myogenesis research includes elements of Tyrosine kinase and Nilotinib.
Between 2016 and 2021, his most popular works were:
- ALS skeletal muscle shows enhanced TGF-β signaling, fibrosis and induction of fibro/adipogenic progenitor markers (55 citations)
- Andrographolide Ameliorates Inflammation and Fibrogenesis and Attenuates Inflammasome Activation in Experimental Non-Alcoholic Steatohepatitis (32 citations)
- Denervation-induced skeletal muscle fibrosis is mediated by CTGF/CCN2 independently of TGF-β. (24 citations)
In his most recent research, the most cited papers focused on:
Enrique Brandan spends much of his time researching Fibrosis, Skeletal muscle, CTGF, Extracellular matrix and Connective tissue.
His Fibrosis study combines topics from a wide range of disciplines, such as Inflammation and Therapeutic effect, Pharmacology.
His Skeletal muscle study results in a more complete grasp of Endocrinology.
His CTGF study is associated with Internal medicine.
His Extracellular matrix research includes themes of Stromal cell, Myofibroblast, Signal transduction and Denervation.
His work investigates the relationship between Connective tissue and topics such as Hypoxia that intersect with problems in Cell biology and Myogenesis.
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