2023 - Research.com Biology and Biochemistry in Chile Leader Award
2023 - Research.com Neuroscience in Chile Leader Award
2022 - Research.com Biology and Biochemistry in Chile Leader Award
2022 - Research.com Neuroscience in Chile Leader Award
1999 - Fellow of John Simon Guggenheim Memorial Foundation
1997 - Fellow, The World Academy of Sciences
Nibaldo C. Inestrosa mostly deals with Neuroscience, Wnt signaling pathway, Cell biology, Biochemistry and Acetylcholinesterase. His Neuroscience research is multidisciplinary, incorporating perspectives in Alzheimer's disease, Disease and Synaptic plasticity. Nibaldo C. Inestrosa has researched Wnt signaling pathway in several fields, including Neurotoxicity, Neurodegeneration and Neuroprotection.
His studies in Cell biology integrate themes in fields like Hippocampal formation, Endocrinology, Internal medicine, Neurotransmission and Synapse. His Biochemistry research focuses on Amyloid and how it connects with P3 peptide, Protein structure, Fibril and Ligand. The Acetylcholinesterase study combines topics in areas such as Cell culture, Basal lamina, Peptide sequence, Intracellular and Pharmacology.
His main research concerns Cell biology, Wnt signaling pathway, Neuroscience, Biochemistry and Acetylcholinesterase. His study in Cell biology is interdisciplinary in nature, drawing from both Hippocampal formation, Synapse, Postsynaptic potential and Neuroprotection. His Wnt signaling pathway study incorporates themes from Endocrinology and Internal medicine.
The study incorporates disciplines such as Synaptic plasticity, Disease, Neurodegeneration and Neurotransmission in addition to Neuroscience. His studies deal with areas such as Amyloid precursor protein and Amyloid as well as Biochemistry. He interconnects Basal lamina, Neuromuscular junction, Cholinergic and Senile plaques in the investigation of issues within Acetylcholinesterase.
Nibaldo C. Inestrosa spends much of his time researching Cell biology, Wnt signaling pathway, Neuroscience, Neuroprotection and Hippocampal formation. His Cell biology study integrates concerns from other disciplines, such as Synaptic plasticity, Synapse, Postsynaptic potential and Neurotransmission. His Wnt signaling pathway research includes elements of Neurodegeneration and Disease.
His study in Neuroscience is interdisciplinary in nature, drawing from both Identification and Social isolation. In his study, Endoplasmic reticulum, Neurotoxicity and Mitochondrial permeability transition pore is strongly linked to Mitochondrion, which falls under the umbrella field of Neuroprotection. His research in Hippocampal formation intersects with topics in Genetically modified mouse, Transgene, Hippocampus and Toxicity.
His primary scientific interests are in Wnt signaling pathway, Cell biology, Neuroprotection, Neurodegeneration and Disease. His work deals with themes such as Carbohydrate metabolism, Central nervous system, Neuroscience and Function, which intersect with Wnt signaling pathway. The various areas that Nibaldo C. Inestrosa examines in his Cell biology study include Synaptic plasticity, Postsynaptic potential, Neurotransmission and Amyloid precursor protein.
His Neuroprotection study integrates concerns from other disciplines, such as Cell growth, PORCN, Hippocampal formation, Alzheimer's disease and Hippocampus. His work investigates the relationship between Hippocampus and topics such as Cathepsin D that intersect with problems in Internal medicine and Endocrinology. His research in Neurodegeneration focuses on subjects like Neurotoxicity, which are connected to Bioinformatics, Parkinson's disease and Paraquat.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Acetylcholinesterase Accelerates Assembly of Amyloid-β-Peptides into Alzheimer's Fibrils: Possible Role of the Peripheral Site of the Enzyme
Nibaldo C. Inestrosa;Alejandra Alvarez;Cristián A. Pérez;Ricardo D. Moreno.
Metalloenzyme-like Activity of Alzheimer's Disease β-Amyloid Cu-DEPENDENT CATALYTIC CONVERSION OF DOPAMINE, CHOLESTEROL, AND BIOLOGICAL REDUCING AGENTS TO NEUROTOXIC H2O2
Carlos Opazo;Xudong Huang;Robert A. Cherny;Robert D. Moir.
Journal of Biological Chemistry (2002)
Emerging roles of Wnts in the adult nervous system
Nibaldo C. Inestrosa;Ernest Arenas.
Nature Reviews Neuroscience (2010)
Settlement of benthic marine invertebrates
SR Rodriguez;FP Ojeda;NC Inestrosa.
Marine Ecology Progress Series (1993)
The role of oxidative stress in the toxicity induced by amyloid β-peptide in Alzheimer’s disease
Soledad Miranda;Carlos Opazo;Luis F Larrondo;Francisco J Muñoz.
Progress in Neurobiology (2000)
A structural motif of acetylcholinesterase that promotes amyloid β-peptide fibril formation
G. V. De Ferrari;M. A. Canales;I. Shin;L. M. Weiner.
The alpha-helical to beta-strand transition in the amino-terminal fragment of the amyloid beta-peptide modulates amyloid formation.
Claudio Soto;Eduardo M. Castaño;Blas Frangione;Nibaldo C. Inestrosa.
Journal of Biological Chemistry (1995)
Activation of Wnt signaling rescues neurodegeneration and behavioral impairments induced by β-amyloid fibrils
G V De Ferrari;M A Chacón;M I Barría;J L Garrido.
Molecular Psychiatry (2003)
Stable Complexes Involving Acetylcholinesterase and Amyloid-β Peptide Change the Biochemical Properties of the Enzyme and Increase the Neurotoxicity of Alzheimer’s Fibrils
Alejandra Alvarez;Rodrigo Alarcón;Carlos Opazo;Eliseo O. Campos.
The Journal of Neuroscience (1998)
Wnt signaling function in Alzheimer's disease.
Giancarlo V. De Ferrari and;Nibaldo C. Inestrosa.
Brain Research Reviews (2000)
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