World's Best Scientists 2026 revealed!

D-Index & Metrics

Molecular Biology

D-Index
63
Citations
14801
World Ranking
1803
National Ranking
902

Pier Lorenzo Puri publication distribution in Molecular Biology in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Molecular Biology in 2026. The highlighted bar marks where Pier Lorenzo Puri sits on this spectrum.

47–56 publications: 7 scientists 57–66 publications: 17 scientists 67–76 publications: 65 scientists 77–86 publications: 90 scientists 87–96 publications: 125 scientists 97–106 publications: 131 scientists 107–116 publications: 162 scientists 117–126 publications: 177 scientists 127–136 publications: 158 scientists 137–146 publications: 158 scientists 147–156 publications: 146 scientists 157–166 publications: 159 scientists 167–176 publications: 131 scientists 177–186 publications: 110 scientists 187–196 publications: 112 scientists 197–206 publications: 100 scientists 207–216 publications: 89 scientists 217–226 publications: 98 scientists 227–236 publications: 74 scientists 237–246 publications: 72 scientists 247–256 publications: 63 scientists 257–266 publications: 53 scientists 267–276 publications: 54 scientists 277–286 publications: 49 scientists 287–296 publications: 52 scientists 297–306 publications: 43 scientists 307–316 publications: 46 scientists 317–326 publications: 41 scientists 327–336 publications: 42 scientists 337–346 publications: 31 scientists 347–356 publications: 28 scientists 357–366 publications: 29 scientists 367–376 publications: 26 scientists 377–386 publications: 24 scientists 387–396 publications: 24 scientists 397–406 publications: 14 scientists 407–416 publications: 13 scientists 417–426 publications: 20 scientists 427–436 publications: 12 scientists 437–446 publications: 20 scientists 447–456 publications: 11 scientists 457–466 publications: 10 scientists 467–476 publications: 14 scientists 477–486 publications: 14 scientists 487–496 publications: 10 scientists 497–506 publications: 13 scientists 507–516 publications: 13 scientists 517–526 publications: 2 scientists 527–536 publications: 4 scientists 537–546 publications: 6 scientists 547–556 publications: 8 scientists 557–563 publications: 6 scientists 564+ publications: 100 scientists
47 publications 564+

This scientist: 146 publications — 35th percentile

35% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 564 publications or more.

Pier Lorenzo Puri D-index placement in Molecular Biology in 2026

The chart shows the D-index (discipline H-index) distribution of Molecular Biology scientists ranked by Research.com in 2026. The highlighted bar marks where Pier Lorenzo Puri sits on this spectrum.

40–41 D-Index: 36 scientists 42–43 D-Index: 101 scientists 44–45 D-Index: 115 scientists 46–47 D-Index: 121 scientists 48–49 D-Index: 118 scientists 50–51 D-Index: 130 scientists 52–53 D-Index: 106 scientists 54–55 D-Index: 116 scientists 56–57 D-Index: 113 scientists 58–59 D-Index: 129 scientists 60–61 D-Index: 120 scientists 62–63 D-Index: 105 scientists 64–65 D-Index: 131 scientists 66–67 D-Index: 95 scientists 68–69 D-Index: 97 scientists 70–71 D-Index: 106 scientists 72–73 D-Index: 83 scientists 74–75 D-Index: 89 scientists 76–77 D-Index: 77 scientists 78–79 D-Index: 70 scientists 80–81 D-Index: 73 scientists 82–83 D-Index: 60 scientists 84–85 D-Index: 48 scientists 86–87 D-Index: 45 scientists 88–89 D-Index: 50 scientists 90–91 D-Index: 31 scientists 92–93 D-Index: 51 scientists 94–95 D-Index: 43 scientists 96–97 D-Index: 38 scientists 98–99 D-Index: 39 scientists 100–101 D-Index: 41 scientists 102–103 D-Index: 29 scientists 104–105 D-Index: 33 scientists 106–107 D-Index: 35 scientists 108–109 D-Index: 20 scientists 110–111 D-Index: 38 scientists 112–113 D-Index: 19 scientists 114–115 D-Index: 28 scientists 116–117 D-Index: 13 scientists 118–119 D-Index: 23 scientists 120–121 D-Index: 16 scientists 122–123 D-Index: 15 scientists 124–125 D-Index: 11 scientists 126–127 D-Index: 21 scientists 128–129 D-Index: 7 scientists 130–131 D-Index: 13 scientists 132–133 D-Index: 14 scientists 134–135 D-Index: 17 scientists 136–137 D-Index: 9 scientists 138–139 D-Index: 8 scientists 140–141 D-Index: 16 scientists 142–143 D-Index: 7 scientists 144 D-Index: 7 scientists 145+ D-Index: 100 scientists
40 D-Index 145+

This scientist: 63 D-Index — 43rd percentile

43% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 145 D-Index or more.

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Transcription factor

His primary areas of study are MyoD, Molecular biology, Cell biology, Cancer research and Transcription. His study with MyoD involves better knowledge in Myogenesis. In his research on the topic of Molecular biology, Epigenetics is strongly related with Chromatin.

His Cell biology research integrates issues from Cell, Genetics and Skeletal muscle. He studied Transcription and DNA that intersect with Acetyltransferase activity, Acetylation and Cell cycle progression. His PCAF study combines topics in areas such as Acetyltransferase and Acetyltransferases.

His most cited work include:

  • p38 and Extracellular Signal-Regulated Kinases Regulate the Myogenic Program at Multiple Steps (412 citations)
  • Differential roles of p300 and PCAF acetyltransferases in muscle differentiation (377 citations)
  • Differential roles of p300 and PCAF acetyltransferases in muscle differentiation (377 citations)

What are the main themes of his work throughout his whole career to date?

Pier Lorenzo Puri spends much of his time researching Cell biology, MyoD, Molecular biology, Myogenesis and Cellular differentiation. His study in Cell biology is interdisciplinary in nature, drawing from both Chromatin, Genetics and Skeletal muscle. His MyoD research includes elements of Cyclin-dependent kinase, Transcription and Ectopic expression.

The various areas that Pier Lorenzo Puri examines in his Molecular biology study include Cell culture, Transcription factor, Activator and Deacetylase activity, Histone. His study on Myogenin, Myogenic regulatory factors and C2C12 is often connected to PITX2 as part of broader study in Myogenesis. His Cellular differentiation study also includes fields such as

  • Cell cycle which connect with DNA damage,
  • Regulation of gene expression which intersects with area such as Cancer research,
  • Signal transduction that connect with fields like Angiotensin II.

He most often published in these fields:

  • Cell biology (56.49%)
  • MyoD (33.12%)
  • Molecular biology (27.92%)

What were the highlights of his more recent work (between 2018-2021)?

  • Cell biology (56.49%)
  • Skeletal muscle (16.23%)
  • Duchenne muscular dystrophy (12.99%)

In recent papers he was focusing on the following fields of study:

Cell biology, Skeletal muscle, Duchenne muscular dystrophy, Stem cell and Inflammation are his primary areas of study. His MyoD, Myogenesis and Myogenic differentiation study are his primary interests in Cell biology. Pier Lorenzo Puri interconnects Chromatin, Cell cycle, Compartmentalization and DNA damage in the investigation of issues within MyoD.

The concepts of his Skeletal muscle study are interwoven with issues in Myocyte, Lesion, Extracellular matrix and Nerve injury. His research integrates issues of Cancer research, Downregulation and upregulation and Epigenetics in his study of Duchenne muscular dystrophy. His Stem cell study combines topics from a wide range of disciplines, such as Endocrinology, Internal medicine, Cellular differentiation and Cell fate determination.

Between 2018 and 2021, his most popular works were:

  • Transcription Factor-Directed Re-wiring of Chromatin Architecture for Somatic Cell Nuclear Reprogramming toward trans-Differentiation (22 citations)
  • Intergenerational inheritance of high fat diet-induced cardiac lipotoxicity in Drosophila (17 citations)
  • Macrophages fine tune satellite cell fate in dystrophic skeletal muscle of mdx mice. (14 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Cancer

His scientific interests lie mostly in Cell biology, Cellular differentiation, Stem cell, Skeletal muscle and Cell fate determination. Particularly relevant to MyoD is his body of work in Cell biology. His Cellular differentiation research incorporates themes from Secretion, Muscular dystrophy, Duchenne muscular dystrophy, Embryo and Effector.

His work carried out in the field of Stem cell brings together such families of science as Inflammation, STAT3 and Homeostasis.

Best Publications

  • p38 and Extracellular Signal-Regulated Kinases Regulate the Myogenic Program at Multiple Steps

    Zhenguo Wu;Pamela J. Woodring;Kunjan S. Bhakta;Kumiko Tamura

  • Studying arrhythmogenic right ventricular dysplasia with patient-specific iPSCs

    Changsung Kim;Johnson Wong;Jianyan Wen;Shirong Wang

  • Differential roles of p300 and PCAF acetyltransferases in muscle differentiation

    Pier Lorenzo Puri;Pier Lorenzo Puri;Vittorio Sartorelli;Vittorio Sartorelli;Xiang Jiao Yang;Yasuo Hamamori

  • Regulation of Histone Acetyltransferases p300 and PCAF by the bHLH Protein Twist and Adenoviral Oncoprotein E1A

    Yasuo Hamamori;Vittorio Sartorelli;Vasily Ogryzko;Pier Lorenzo Puri;Pier Lorenzo Puri

  • Acetylation of MyoD Directed by PCAF Is Necessary for the Execution of the Muscle Program

    Vittorio Sartorelli;Pier Lorenzo Puri;Pier Lorenzo Puri;Yasuo Hamamori;Vasily Ogryzko

  • p38 pathway targets SWI-SNF chromatin-remodeling complex to muscle-specific loci.

    Cristiano Simone;Sonia Vanina Forcales;David A Hill;Anthony N Imbalzano

  • TNF/p38α/Polycomb Signaling to Pax7 Locus in Satellite Cells Links Inflammation to the Epigenetic Control of Muscle Regeneration

    Daniela Palacios;Chiara Mozzetta;Silvia Consalvi;Giuseppina Caretti;Giuseppina Caretti

  • STAT3 signaling controls satellite cell expansion and skeletal muscle repair

    Matthew Timothy Tierney;Tufan Aydogdu;David Sala;Barbora Malecova

  • Functional and morphological recovery of dystrophic muscles in mice treated with deacetylase inhibitors.

    G C Minetti;C Colussi;R Adami;C Serra

  • Regulation of muscle regulatory factors by DNA-binding, interacting proteins, and post-transcriptional modifications.

    Pier Lorenzo Puri;Vittorio Sartorelli

  • p300 is required for MyoD‐dependent cell cycle arrest and muscle‐specific gene transcription

    Pier Lorenzo Puri;Maria Laura Avantaggiati;Clara Balsano;Nianli Sang

  • Induction of terminal differentiation by constitutive activation of p38 MAP kinase in human rhabdomyosarcoma cells.

    Pier Lorenzo Orenzo Puri;Zhenguo Wu;Peilin Zhang;Lauren D. Wood

  • p38-dependent phosphorylation of the mRNA decay-promoting factor KSRP controls the stability of select myogenic transcripts.

    Paola Briata;Sonia Vanina Forcales;Marco Ponassi;Giorgio Corte

  • HDAC2 blockade by nitric oxide and histone deacetylase inhibitors reveals a common target in Duchenne muscular dystrophy treatment

    Claudia Colussi;Chiara Mozzetta;Aymone Gurtner;Barbara Illi

  • The hepatitis B virus X gene induces p53-mediated programmed cell death

    Paolo Chirillo;Sabrina Pagano;Gioacchino Natoli;Pier Lorenzo Puri

  • Deacetylase inhibitors increase muscle cell size by promoting myoblast recruitment and fusion through induction of follistatin.

    Simona Iezzi;Monica Di Padova;Carlo Serra;Giuseppina Caretti

  • Class I histone deacetylases sequentially interact with MyoD and pRb during skeletal myogenesis

    Pier Lorenzo Puri;Pier Lorenzo Puri;Simona Iezzi;Peter Stiegler;Tung Ti Chen

  • Denervation-activated STAT3–IL-6 signalling in fibro-adipogenic progenitors promotes myofibres atrophy and fibrosis

    Luca Madaro;Magda Passafaro;David Sala;Usue Etxaniz

  • Fibroadipogenic progenitors mediate the ability of HDAC inhibitors to promote regeneration in dystrophic muscles of young, but not old Mdx mice

    Chiara Mozzetta;Silvia Consalvi;Valentina Saccone;Matthew Tierney

  • Signal-dependent incorporation of MyoD-BAF60c into Brg1-based SWI/SNF chromatin-remodelling complex.

    Sonia Vanina Forcales;Sonia Albini;Lorenzo Giordani;Barbora Malecová

Frequent Co-Authors

Vittorio Sartorelli
Vittorio Sartorelli National Institutes of Health
Jean Y. J. Wang
Jean Y. J. Wang University of California, San Diego
Cristiano Simone
Cristiano Simone University of Bari Aldo Moro
Mark Mercola
Mark Mercola Cardiovascular Institute of the South
Gioacchino Natoli
Gioacchino Natoli European Institute of Oncology
Antonio Giordano
Antonio Giordano Temple University
Antonello Mai
Antonello Mai Sapienza University of Rome
Carlo Gaetano
Carlo Gaetano Goethe University Frankfurt
Larry Kedes
Larry Kedes University of Southern California

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