Pier Lorenzo Puri

What is he best known for?

The fields of study he is best known for:

• Gene
• DNA
• Transcription factor

His primary areas of study are MyoD, Molecular biology, Cell biology, Cancer research and Transcription. His study with MyoD involves better knowledge in Myogenesis. In his research on the topic of Molecular biology, Epigenetics is strongly related with Chromatin.

His Cell biology research integrates issues from Cell, Genetics and Skeletal muscle. He studied Transcription and DNA that intersect with Acetyltransferase activity, Acetylation and Cell cycle progression. His PCAF study combines topics in areas such as Acetyltransferase and Acetyltransferases.

His most cited work include:

• p38 and Extracellular Signal-Regulated Kinases Regulate the Myogenic Program at Multiple Steps (412 citations)
• Differential roles of p300 and PCAF acetyltransferases in muscle differentiation (377 citations)
• Differential roles of p300 and PCAF acetyltransferases in muscle differentiation (377 citations)

What are the main themes of his work throughout his whole career to date?

Pier Lorenzo Puri spends much of his time researching Cell biology, MyoD, Molecular biology, Myogenesis and Cellular differentiation. His study in Cell biology is interdisciplinary in nature, drawing from both Chromatin, Genetics and Skeletal muscle. His MyoD research includes elements of Cyclin-dependent kinase, Transcription and Ectopic expression.

The various areas that Pier Lorenzo Puri examines in his Molecular biology study include Cell culture, Transcription factor, Activator and Deacetylase activity, Histone. His study on Myogenin, Myogenic regulatory factors and C2C12 is often connected to PITX2 as part of broader study in Myogenesis. His Cellular differentiation study also includes fields such as

• Cell cycle which connect with DNA damage,
• Regulation of gene expression which intersects with area such as Cancer research,
• Signal transduction that connect with fields like Angiotensin II.

He most often published in these fields:

• Cell biology (56.49%)
• MyoD (33.12%)
• Molecular biology (27.92%)

What were the highlights of his more recent work (between 2018-2021)?

• Cell biology (56.49%)
• Skeletal muscle (16.23%)
• Duchenne muscular dystrophy (12.99%)

In recent papers he was focusing on the following fields of study:

Cell biology, Skeletal muscle, Duchenne muscular dystrophy, Stem cell and Inflammation are his primary areas of study. His MyoD, Myogenesis and Myogenic differentiation study are his primary interests in Cell biology. Pier Lorenzo Puri interconnects Chromatin, Cell cycle, Compartmentalization and DNA damage in the investigation of issues within MyoD.

The concepts of his Skeletal muscle study are interwoven with issues in Myocyte, Lesion, Extracellular matrix and Nerve injury. His research integrates issues of Cancer research, Downregulation and upregulation and Epigenetics in his study of Duchenne muscular dystrophy. His Stem cell study combines topics from a wide range of disciplines, such as Endocrinology, Internal medicine, Cellular differentiation and Cell fate determination.

Between 2018 and 2021, his most popular works were:

• Transcription Factor-Directed Re-wiring of Chromatin Architecture for Somatic Cell Nuclear Reprogramming toward trans-Differentiation (22 citations)
• Intergenerational inheritance of high fat diet-induced cardiac lipotoxicity in Drosophila (17 citations)
• Macrophages fine tune satellite cell fate in dystrophic skeletal muscle of mdx mice. (14 citations)

In his most recent research, the most cited papers focused on:

• Gene
• DNA
• Cancer

His scientific interests lie mostly in Cell biology, Cellular differentiation, Stem cell, Skeletal muscle and Cell fate determination. Particularly relevant to MyoD is his body of work in Cell biology. His Cellular differentiation research incorporates themes from Secretion, Muscular dystrophy, Duchenne muscular dystrophy, Embryo and Effector.

His work carried out in the field of Stem cell brings together such families of science as Inflammation, STAT3 and Homeostasis.