What is he best known for?
The fields of study he is best known for:
His primary areas of study are Molecular biology, MyoD, Myogenin, Myogenesis and PITX2.
His studies in Molecular biology integrate themes in fields like Gene expression, Cellular differentiation, Skeletal muscle, Myocyte and Regulation of gene expression.
The subject of his Regulation of gene expression research is within the realm of Genetics.
Stephen J. Tapscott is interested in MyoD Protein, which is a branch of MyoD.
His work focuses on many connections between Myogenesis and other disciplines, such as Complementary DNA, that overlap with his field of interest in Cell culture, Messenger RNA, Nuclear localization sequence and MYOD1 Gene.
The PITX2 study combines topics in areas such as Chromatin, Chromatin remodeling, MYF5 and Myogenic regulatory factors.
His most cited work include:
- Activation of muscle-specific genes in pigment, nerve, fat, liver, and fibroblast cell lines by forced expression of MyoD. (778 citations)
- MyoD1: a nuclear phosphoprotein requiring a Myc homology region to convert fibroblasts to myoblasts (656 citations)
- MyoD and the transcriptional control of myogenesis (652 citations)
What are the main themes of his work throughout his whole career to date?
Stephen J. Tapscott mainly investigates Genetics, Molecular biology, MyoD, Myogenesis and Cell biology.
Stephen J. Tapscott has researched Molecular biology in several fields, including Enhancer, Gene expression, Xenopus, Chromatin immunoprecipitation and Transfection.
His MyoD study combines topics from a wide range of disciplines, such as Regulation of gene expression, PITX2 and Myogenin.
His Myogenesis study is concerned with the larger field of Myocyte.
As a member of one scientific family, he mostly works in the field of Myocyte, focusing on Skeletal muscle and, on occasion, Dystrophy and Homeobox.
His research in Cell biology intersects with topics in Cellular differentiation and Anatomy.
He most often published in these fields:
- Genetics (43.66%)
- Molecular biology (39.44%)
- MyoD (36.62%)
What were the highlights of his more recent work (between 2014-2021)?
- DUX4 (26.41%)
- Facioscapulohumeral muscular dystrophy (21.83%)
- Genetics (43.66%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in DUX4, Facioscapulohumeral muscular dystrophy, Genetics, Muscular dystrophy and Cell biology.
Stephen J. Tapscott combines subjects such as Myocyte, Chromatin and Gene expression with his study of DUX4.
His Facioscapulohumeral muscular dystrophy research includes themes of Clinical trial, Disease, Pathology and Skeletal muscle.
His Muscular dystrophy research is multidisciplinary, incorporating perspectives in Phenotype, Dystrophy, Psychological repression and Gene expression profiling.
The various areas that Stephen J. Tapscott examines in his Cell biology study include Cell, Cellular differentiation, Transcription factor, CAF-1 and Gene silencing.
His Myogenesis research focuses on MyoD Protein and MyoD.
Between 2014 and 2021, his most popular works were:
- Conservation and innovation in the DUX4-family gene network (138 citations)
- Mutations in DNMT3B Modify Epigenetic Repression of the D4Z4 Repeat and the Penetrance of Facioscapulohumeral Dystrophy (102 citations)
- Inter-individual differences in CpG methylation at D4Z4 correlate with clinical variability in FSHD1 and FSHD2 (97 citations)
In his most recent research, the most cited papers focused on:
His main research concerns Genetics, DUX4, Facioscapulohumeral muscular dystrophy, Epigenetics and Myogenesis.
His DUX4 research incorporates themes from Myocyte, Transcriptome, Epigenetic Repression and Derepression.
His Myocyte research includes elements of Cellular differentiation and Skeletal muscle.
His Facioscapulohumeral muscular dystrophy research is multidisciplinary, relying on both Clinical trial and Family medicine.
His MyoD Protein and MyoD investigations are all subjects of Myogenesis research.
His MyoD study combines topics in areas such as PITX2, RNA polymerase II, E-box and Myogenin.
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