1986 - Member of the National Academy of Sciences
Harold Weintraub mainly investigates Molecular biology, Myogenesis, MyoD, Cell biology and MyoD Protein. He has researched Molecular biology in several fields, including Complementary DNA, Gene, Mutant and Cell fate determination. His study in Myogenesis is interdisciplinary in nature, drawing from both Nuclear localization sequence, Gene expression and Cellular differentiation.
His MyoD study deals with Enhancer intersecting with DNA-binding protein. His Cell biology study combines topics from a wide range of disciplines, such as Basic helix-loop-helix, Genetics and Proneural genes. His studies deal with areas such as Gastrulation, Enhancer binding, Binding site and Mesoderm as well as MyoD Protein.
Harold Weintraub focuses on Molecular biology, DNA, Cell biology, MyoD and Myogenesis. Harold Weintraub interconnects RNA, Chromatin, Enhancer, Gene and Transcription in the investigation of issues within Molecular biology. His study with DNA involves better knowledge in Biochemistry.
In his research, NEUROD1 is intimately related to NeuroD, which falls under the overarching field of Cell biology. His work on MyoD Protein as part of general MyoD study is frequently connected to PITX2, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. Harold Weintraub has included themes like Nuclear localization sequence, Gene expression and Cellular differentiation in his Myogenesis study.
The scientist’s investigation covers issues in MyoD, Cell biology, Molecular biology, Myogenesis and MyoD Protein. Harold Weintraub has researched MyoD in several fields, including Protein structure, Repressor and E-box. The Cell biology study combines topics in areas such as Genetics, Internal medicine, NeuroD and Endocrinology.
His biological study spans a wide range of topics, including Embryonic stem cell, Myoblast fusion, Mutant, Enhancer and C2C12. The study incorporates disciplines such as Nuclear localization sequence and Cellular differentiation in addition to Myogenesis. His MyoD Protein study integrates concerns from other disciplines, such as Gastrulation, DNA, Binding site and Mesoderm.
His scientific interests lie mostly in Cell biology, MyoD, Myogenesis, Molecular biology and MyoD Protein. His Cell biology research incorporates elements of Proneural genes and Neurogenins. His study looks at the relationship between MyoD and fields such as Basic helix-loop-helix, as well as how they intersect with chemical problems.
His Myogenesis research is multidisciplinary, incorporating elements of Nuclear localization sequence and Cellular differentiation. His work deals with themes such as Enhancer, Gene expression, C2C12 and DNA-binding protein, which intersect with Cellular differentiation. Harold Weintraub connects Molecular biology with T cell in his study.
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Expression of a single transfected cDNA converts fibroblasts to myoblasts.
Robert L. Davis;Harold Weintraub;Andrew B. Lassar.
The protein Id: A negative regulator of helix-loop-helix DNA binding proteins
Robert Benezra;Robert L. Davis;Robert L. Davis;Daniel Lockshon;David L. Turner.
Interactions between heterologous helix-loop-helix proteins generate complexes that bind specifically to a common DNA sequence.
Cornelis Murre;Patrick Schonleber McCaw;H. Vaessin;M. Caudy.
Chromosomal subunits in active genes have an altered conformation
Harold Weintraub;Mark Groudine.
The myoD gene family: nodal point during specification of the muscle cell lineage
Harold Weintraub;Robert Davis;Stephen Tapscott;Matthew Thayer.
Expression of achaete-scute homolog 3 in Xenopus embryos converts ectodermal cells to a neural fate.
David L. Turner;Harold Weintraub.
Genes & Development (1994)
Conversion of Xenopus ectoderm into neurons by NeuroD, a basic helix-loop-helix protein
Jacqueline E. Lee;Stanley M. Hollenberg;Stanley M. Hollenberg;Lauren Snider;Lauren Snider;David L. Turner.
The MyoD Family and Myogenesis: Redundancy, Networks, and Thresholds
Activation of muscle-specific genes in pigment, nerve, fat, liver, and fibroblast cell lines by forced expression of MyoD.
H Weintraub;S J Tapscott;R L Davis;M J Thayer.
Proceedings of the National Academy of Sciences of the United States of America (1989)
Differences and similarities in DNA-binding preferences of MyoD and E2A protein complexes revealed by binding site selection
TK Blackwell;H Weintraub.
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