World's Best Scientists 2026 revealed!
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Genetics
UK
2024

D-Index & Metrics

Medicine

D-Index
105
Citations
64721
World Ranking
6650
National Ranking
642

Genetics

D-Index
105
Citations
64756
World Ranking
616
National Ranking
92

Gillian P. Bates publication distribution in Genetics in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Genetics in 2026. The highlighted bar marks where Gillian P. Bates sits on this spectrum.

45–54 publications: 6 scientists 55–64 publications: 10 scientists 65–74 publications: 35 scientists 75–84 publications: 84 scientists 85–94 publications: 102 scientists 95–104 publications: 151 scientists 105–114 publications: 175 scientists 115–124 publications: 203 scientists 125–134 publications: 217 scientists 135–144 publications: 205 scientists 145–154 publications: 193 scientists 155–164 publications: 188 scientists 165–174 publications: 170 scientists 175–184 publications: 178 scientists 185–194 publications: 164 scientists 195–204 publications: 173 scientists 205–214 publications: 159 scientists 215–224 publications: 134 scientists 225–234 publications: 143 scientists 235–244 publications: 105 scientists 245–254 publications: 114 scientists 255–264 publications: 92 scientists 265–274 publications: 88 scientists 275–284 publications: 87 scientists 285–294 publications: 80 scientists 295–304 publications: 62 scientists 305–314 publications: 75 scientists 315–324 publications: 67 scientists 325–334 publications: 60 scientists 335–344 publications: 52 scientists 345–354 publications: 40 scientists 355–364 publications: 48 scientists 365–374 publications: 47 scientists 375–384 publications: 46 scientists 385–394 publications: 31 scientists 395–404 publications: 27 scientists 405–414 publications: 40 scientists 415–424 publications: 30 scientists 425–434 publications: 43 scientists 435–444 publications: 29 scientists 445–454 publications: 14 scientists 455–464 publications: 28 scientists 465–474 publications: 21 scientists 475–484 publications: 21 scientists 485–494 publications: 22 scientists 495–504 publications: 17 scientists 505–514 publications: 12 scientists 515–524 publications: 11 scientists 525–534 publications: 8 scientists 535–544 publications: 8 scientists 545–554 publications: 14 scientists 555–564 publications: 4 scientists 565–574 publications: 11 scientists 575–584 publications: 5 scientists 585–594 publications: 11 scientists 595–604 publications: 12 scientists 605–614 publications: 7 scientists 615–624 publications: 6 scientists 625–634 publications: 10 scientists 635–644 publications: 9 scientists 645–654 publications: 10 scientists 655–664 publications: 6 scientists 665–674 publications: 6 scientists 675–684 publications: 6 scientists 685–694 publications: 4 scientists 695–702 publications: 6 scientists 703+ publications: 100 scientists
45 publications 703+

This scientist: 313 publications — 78th percentile

78% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 703 publications or more.

Gillian P. Bates D-index placement in Genetics in 2026

The chart shows the D-index (discipline H-index) distribution of Genetics scientists ranked by Research.com in 2026. The highlighted bar marks where Gillian P. Bates sits on this spectrum.

40–41 D-Index: 24 scientists 42–43 D-Index: 52 scientists 44–45 D-Index: 84 scientists 46–47 D-Index: 112 scientists 48–49 D-Index: 118 scientists 50–51 D-Index: 141 scientists 52–53 D-Index: 143 scientists 54–55 D-Index: 145 scientists 56–57 D-Index: 179 scientists 58–59 D-Index: 162 scientists 60–61 D-Index: 175 scientists 62–63 D-Index: 191 scientists 64–65 D-Index: 172 scientists 66–67 D-Index: 184 scientists 68–69 D-Index: 164 scientists 70–71 D-Index: 158 scientists 72–73 D-Index: 150 scientists 74–75 D-Index: 136 scientists 76–77 D-Index: 127 scientists 78–79 D-Index: 127 scientists 80–81 D-Index: 111 scientists 82–83 D-Index: 110 scientists 84–85 D-Index: 110 scientists 86–87 D-Index: 84 scientists 88–89 D-Index: 102 scientists 90–91 D-Index: 66 scientists 92–93 D-Index: 72 scientists 94–95 D-Index: 70 scientists 96–97 D-Index: 54 scientists 98–99 D-Index: 60 scientists 100–101 D-Index: 49 scientists 102–103 D-Index: 55 scientists 104–105 D-Index: 45 scientists 106–107 D-Index: 42 scientists 108–109 D-Index: 28 scientists 110–111 D-Index: 39 scientists 112–113 D-Index: 25 scientists 114–115 D-Index: 31 scientists 116–117 D-Index: 29 scientists 118–119 D-Index: 34 scientists 120–121 D-Index: 29 scientists 122–123 D-Index: 29 scientists 124–125 D-Index: 18 scientists 126–127 D-Index: 27 scientists 128–129 D-Index: 22 scientists 130–131 D-Index: 16 scientists 132–133 D-Index: 11 scientists 134–135 D-Index: 17 scientists 136–137 D-Index: 12 scientists 138–139 D-Index: 21 scientists 140–141 D-Index: 4 scientists 142–143 D-Index: 9 scientists 144–145 D-Index: 14 scientists 146–147 D-Index: 6 scientists 148–149 D-Index: 10 scientists 150–151 D-Index: 7 scientists 152–153 D-Index: 9 scientists 154–155 D-Index: 8 scientists 156–157 D-Index: 8 scientists 158–159 D-Index: 9 scientists 160+ D-Index: 96 scientists
40 D-Index 160+

This scientist: 105 D-Index — 86th percentile

86% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 160 D-Index or more.

Research.com Recognitions

  • 2024 - Research.com Genetics in United Kingdom Leader Award
  • 2023 - Research.com Genetics in United Kingdom Leader Award

Overview

Gillian P. Bates is affiliated with University College London in the United Kingdom. Their research primarily focuses on molecular and cellular mechanisms underlying neurodegenerative diseases, particularly Huntington's disease.

The main fields of study associated with their work are Biochemistry, Genetics and Molecular Biology, and Neuroscience. Within these broader fields, Bates's work is specialized in subfields including Molecular Biology, Cellular and Molecular Neuroscience, Neurology, Genetics, and Cell Biology.

The key research topics covered in their publications include Genetic Neurodegenerative Diseases, Mitochondrial Function and Pathology, Muscle Physiology and Disorders, Neurological Disorders and Treatments, RNA Research and Splicing, Cardiomyopathy and Myosin Studies, and Endoplasmic Reticulum Stress and Disease.

Some of their recent notable papers are:

  • "FAN1 controls mismatch repair complex assembly via MLH1 retention to stabilize CAG repeat expansion in Huntington's disease" (2021), published in Cell Reports
  • "Subcellular Localization And Formation Of Huntingtin Aggregates Correlates With Symptom Onset And Progression In A Huntington'S Disease Model" (2020), published in Brain Communications
  • "Uninterrupted CAG repeat drives striatum-selective transcriptionopathy and nuclear pathogenesis in human Huntingtin BAC mice" (2022), published in Neuron
  • "TBK1 phosphorylates mutant Huntingtin and suppresses its aggregation and toxicity in Huntington's disease models" (2020), published in The EMBO Journal
  • "A CAG repeat threshold for therapeutics targeting somatic instability in Huntington's disease" (2024), published in Brain

Bates frequently collaborates with other researchers. Their most common coauthors include:

  • Christian Landles (17 coauthored works)
  • Kirupa Sathasivam (13 coauthored works)
  • Georgina F Osborne (10 coauthored works)
  • Edward J. Smith (10 coauthored works)
  • Jemima Phillips (7 coauthored works)

The scientist has published extensively in venues such as bioRxiv (Cold Spring Harbor Laboratory), Brain Communications, Brain, Scientific Reports, and Neuron. These journals represent prominent platforms within their research areas and reflect the scope of their published contributions.

Best Publications

  • A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes

    Marcy E. MacDonald;Christine M. Ambrose;Mabel P. Duyao;Richard H. Myers

  • A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes. The Huntington's Disease Collaborative Research Group.

    M Shah;N Datson;L Srinidhi;VP Stanton

  • Exon 1 of the HD Gene with an Expanded CAG Repeat Is Sufficient to Cause a Progressive Neurological Phenotype in Transgenic Mice

    Laura Mangiarini;Kirupa Sathasivam;Mary Seller;Barbara Cozens

  • Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in Brain

    Marian DiFiglia;Ellen Sapp;Kathryn O. Chase;Stephen W. Davies

  • FORMATION OF NEURONAL INTRANUCLEAR INCLUSIONS UNDERLIES THE NEUROLOGICAL DYSFUNCTION IN MICE TRANSGENIC FOR THE HD MUTATION

    Stephen W Davies;Mark Turmaine;Barbara A Cozens;Marian DiFiglia

  • Huntingtin-Encoded Polyglutamine Expansions Form Amyloid-like Protein Aggregates In Vitro and In Vivo

    Eberhard Scherzinger;Rudi Lurz;Mark Turmaine;Laura Mangiarini

  • Huntington's disease

    Gillian Bates;K P S J Murphy;P Harper;L Jones

  • Characterization of progressive motor deficits in mice transgenic for the human Huntington's disease mutation.

    R. J. Carter;L. A. Lione;Trevor Humby;L. Mangiarini

  • The Huntington's disease protein interacts with p53 and CREB-binding protein and represses transcription.

    Joan S. Steffan;Aleksey Kazantsev;Olivera Spasic-Boskovic;Marilee Greenwald

  • Suberoylanilide hydroxamic acid, a histone deacetylase inhibitor, ameliorates motor deficits in a mouse model of Huntington's disease

    E. Hockly;V.M. Richon;B. Woodman;D.L. Smith

  • Self-assembly of polyglutamine-containing huntingtin fragments into amyloid-like fibrils: Implications for Huntington’s disease pathology

    Eberhard Scherzinger;Annie Sittler;Katja Schweiger;Volker Heiser

  • Altered brain neurotransmitter receptors in transgenic mice expressing a portion of an abnormal human Huntington disease gene

    Jang-Ho J. Cha;Christoph M. Kosinski;Julie A. Kerner;Stephen A. Alsdorf

  • A novel pathogenic pathway of immune activation detectable before clinical onset in Huntington's disease

    Maria Björkqvist;Edward J. Wild;Jenny Thiele;Aurelio Silvestroni

  • Huntingtin aggregation and toxicity in Huntington's disease.

    Gillian Bates

  • A novel gene encoding an integral membrane protein is mutated in nephropathic cystinosis

    M. Town;G. Jean;S. Cherqui;M. Attard

  • Huntingtin and the molecular pathogenesis of Huntington's disease Fourth in Molecular Medicine Review Series

    Christian Landles;Gillian P Bates

  • Nonapoptotic neurodegeneration in a transgenic mouse model of Huntington's disease

    Mark Turmaine;Aysha Raza;Amarbirpal Mahal;Laura Mangiarini

  • Intranuclear Neuronal Inclusions in Huntington's Disease and Dentatorubral and Pallidoluysian Atrophy: Correlation between the Density of Inclusions andIT15CAG Triplet Repeat Length

    Mark W. Becher;Joyce A. Kotzuk;Alan H. Sharp;Stephen W. Davies

  • Global changes to the ubiquitin system in Huntington's disease

    Eric J. Bennett;Thomas A. Shaler;Ben Woodman;Kwon-Yul Ryu

  • Formation of neuronal intranuclear inclusions (NII) underlies the neurological dysfunction in mice transgenic for the HD mutation

    SW Davies;M Turmaine;BA Cozens;L Mangiarini

Frequent Co-Authors

Erich E. Wanker
Erich E. Wanker Max Delbrück Center for Molecular Medicine
Sarah J. Tabrizi
Sarah J. Tabrizi University College London
Marcy E. MacDonald
Marcy E. MacDonald Harvard University
Hans Lehrach
Hans Lehrach Max Planck Society
James F. Gusella
James F. Gusella Harvard University
John J. Wasmuth
John J. Wasmuth University of California, Irvine
Leslie M. Thompson
Leslie M. Thompson University of California, Irvine
Martin Farrall
Martin Farrall University of Oxford
Robert Williamson
Robert Williamson University of Melbourne

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