What is she best known for?
The fields of study she is best known for:
The scientist’s investigation covers issues in Huntington's disease, Huntingtin, Huntingtin Protein, Genetics and Neurodegeneration.
Her studies deal with areas such as Genetically modified mouse, Cell biology, Neuroscience and Trinucleotide repeat expansion as well as Huntington's disease.
The Huntingtin study combines topics in areas such as Molecular biology, Gene expression, Protein aggregation and Immunology.
Gillian P. Bates combines subjects such as Atrophin-1, Polyglutamine tract and Dentatorubral-pallidoluysian atrophy with her study of Huntingtin Protein.
Her work on Locus, Allele and Methylation as part of general Genetics study is frequently connected to Lysosomal transport and Cystinosis, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
Her work deals with themes such as SETD2, Histone and Biochemistry, which intersect with Neurodegeneration.
Her most cited work include:
- A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes (6485 citations)
- A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes. The Huntington's Disease Collaborative Research Group. (2979 citations)
- Exon 1 of the HD Gene with an Expanded CAG Repeat Is Sufficient to Cause a Progressive Neurological Phenotype in Transgenic Mice (2680 citations)
What are the main themes of her work throughout her whole career to date?
Gillian P. Bates mainly investigates Huntington's disease, Huntingtin, Genetics, Cell biology and Huntingtin Protein.
Her Huntington's disease study combines topics in areas such as Genetically modified mouse, Neuroscience, Neurodegeneration and Trinucleotide repeat expansion.
Her Genetically modified mouse research focuses on Endocrinology and how it relates to Receptor.
Her study in Huntingtin is interdisciplinary in nature, drawing from both Molecular biology and Exon.
In her work, Heat shock factor is strongly intertwined with Heat shock, which is a subfield of Cell biology.
Her Huntingtin Protein research is multidisciplinary, incorporating elements of Immunology and Dentatorubral-pallidoluysian atrophy.
She most often published in these fields:
- Huntington's disease (50.33%)
- Huntingtin (34.77%)
- Genetics (30.79%)
What were the highlights of her more recent work (between 2013-2021)?
- Cell biology (27.81%)
- Huntingtin (34.77%)
- Huntington's disease (50.33%)
In recent papers she was focusing on the following fields of study:
Gillian P. Bates mainly focuses on Cell biology, Huntingtin, Huntington's disease, Exon and Mutant.
Her work carried out in the field of Cell biology brings together such families of science as Neurodegeneration, HSF1, Pathogenesis and Heat shock.
Her Huntingtin study incorporates themes from Phenotype, Genetically modified mouse, Neural stem cell and Trinucleotide repeat expansion.
Her biological study focuses on Huntingtin Protein.
Her Exon research incorporates themes from Molecular biology, Messenger RNA, RNA splicing and Intron.
Her studies in Mutant integrate themes in fields like Inclusion bodies, Gene knockin, Neuroprotection and Phosphorylation.
Between 2013 and 2021, her most popular works were:
- UBQLN2 mediates autophagy-independent protein aggregate clearance by the proteasome (172 citations)
- HTT-lowering reverses Huntington’s disease immune dysfunction caused by NFκB pathway dysregulation (109 citations)
- Treating the whole body in Huntington's disease (92 citations)
In her most recent research, the most cited papers focused on:
Gillian P. Bates spends much of her time researching Huntington's disease, Huntingtin, Cell biology, Internal medicine and Neurodegeneration.
Gillian P. Bates is interested in Huntingtin Protein, which is a branch of Huntington's disease.
Her research integrates issues of Exon, Genetically modified mouse and Trinucleotide repeat expansion in her study of Huntingtin.
Her research in Cell biology intersects with topics in Inclusion bodies, Biochemistry, Neuroprotection and Downregulation and upregulation.
Her work investigates the relationship between Internal medicine and topics such as Endocrinology that intersect with problems in Atrophy, Heart failure and Cardiomyopathy.
Her biological study spans a wide range of topics, including Protein aggregation, Immunology, Innate immune system, Immune system and Proinflammatory cytokine.
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