Xiao-Jiang Li focuses on Huntingtin, Huntington's disease, Huntingtin Protein, Molecular biology and Neurodegeneration. His Huntingtin research includes themes of Neuron and Cell biology. His Huntington's disease study integrates concerns from other disciplines, such as Neuropathology, Anatomy, Neuropil, Neuroscience and Degenerative disease.
His Huntingtin Protein research is multidisciplinary, incorporating perspectives in Genetically modified mouse, Transgene, Gliosis and Glial fibrillary acidic protein. His studies in Molecular biology integrate themes in fields like Neurotoxicity, Immunoprecipitation, Glutamate receptor, Excitotoxicity and HEK 293 cells. His Mutant research is multidisciplinary, incorporating elements of Mutation and Cell nucleus.
His primary areas of investigation include Huntingtin, Cell biology, Neurodegeneration, Mutant and Molecular biology. As part of his Huntington's disease and Genetics and Huntingtin studies, he is studying Huntingtin. The various areas that he examines in his Huntington's disease study include Neuropil and Neuropathology.
His work deals with themes such as Neurotoxicity, Neurite and Biochemistry, which intersect with Cell biology. Xiao-Jiang Li combines subjects such as Neuroscience, Transgene, Pathogenesis and Proteasome with his study of Neurodegeneration. The concepts of his Molecular biology study are interwoven with issues in Immunoprecipitation, Transcription factor, HEK 293 cells, Promoter and Regulation of gene expression.
His scientific interests lie mostly in Neurodegeneration, Cell biology, Mutant, Neuroscience and CRISPR. His Neurodegeneration research includes themes of Neuropathology and Primate. Xiao-Jiang Li has included themes like Huntingtin and Retinal ganglion cell, Retina in his Cell biology study.
His Huntingtin study necessitates a more in-depth grasp of Huntington's disease. His Mutant research incorporates themes from Neurotoxicity, Transgene, Decussation, Zebrafish and Spinocerebellar ataxia. The study incorporates disciplines such as Autism and Autism spectrum disorder in addition to Neuroscience.
His primary scientific interests are in Neurodegeneration, CRISPR, Neuroscience, Autism spectrum disorder and Autism. The Neurodegeneration study combines topics in areas such as Genome editing, Computational biology, PINK1 and Cell biology. His CRISPR study integrates concerns from other disciplines, such as Mutation, SHANK3 Gene, Scaffold protein, Fluoxetine and Antidepressant.
His study in Neuroscience is interdisciplinary in nature, drawing from both Offspring and Pregnancy. In most of his Autism spectrum disorder studies, his work intersects topics such as Cerebellum.
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Nuclear and Neuropil Aggregates in Huntington’s Disease: Relationship to Neuropathology
Claire-Anne Gutekunst;Shi-Hua Li;Hong Yi;James S. Mulroy.
The Journal of Neuroscience (1999)
A YAC mouse model for Huntington's disease with full-length mutant huntingtin, cytoplasmic toxicity, and selective striatal neurodegeneration.
J.Graeme Hodgson;Nadia Agopyan;Claire-Anne Gutekunst;Blair R Leavitt.
Inhibition of caspase-1 slows disease progression in a mouse model of Huntington's disease
Victor O. Ona;Mingwei Li;Jean Paul G. Vonsattel;L. John Andrews.
Selective striatal neuronal loss in a YAC128 mouse model of Huntington disease
Elizabeth J. Slow;Jeremy van Raamsdonk;Daniel Rogers;Sarah H. Coleman.
Human Molecular Genetics (2003)
Neurological abnormalities in a knock-in mouse model of Huntington’s disease
Chin Hsing Lin;Sara Tallaksen-Greene;Wei Ming Chien;Jamie A. Cearley.
Human Molecular Genetics (2001)
A huntingtin-associated protein enriched in brain with implications for pathology
Xiao-Jiang Li;Shi-Hua Li;Alan H. Sharp;Frederick C. Nucifora.
Huntingtin–protein interactions and the pathogenesis of Huntington's disease
Shi-Hua Li;Xiao-Jiang Li.
Trends in Genetics (2004)
Full-Length Human Mutant Huntingtin with a Stable Polyglutamine Repeat Can Elicit Progressive and Selective Neuropathogenesis in BACHD Mice
Michelle Gray;Dyna I. Shirasaki;Carlos Cepeda;Véronique M. André.
The Journal of Neuroscience (2008)
Widespread expression of Huntington's disease gene (IT15) protein product
Alan H. Sharp;Scott J. Loev;Gabriele Schilling;Shi Hua Li.
The prolyl isomerase Pin1 regulates amyloid precursor protein processing and amyloid-β production
Lucia Pastorino;Anyang Sun;Anyang Sun;Pei-Jung Lu;Xiao Zhen Zhou.
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