2022 - Research.com Best Scientist Award
2022 - Research.com Biology and Biochemistry in Canada Leader Award
2011 - Canada Gairdner Wightman Award
2011 - Izaak Walton Killam Memorial Prize, Canada Council
1995 - Fellow of the Royal Society of Canada Academy of Science
Huntingtin, Internal medicine, Huntington's disease, Huntingtin Protein and Cell biology are his primary areas of study. His Huntingtin research incorporates themes from Excitotoxicity, Molecular biology and Neurodegeneration. His studies deal with areas such as Endocrinology and Oncology as well as Internal medicine.
Michael R. Hayden focuses mostly in the field of Endocrinology, narrowing it down to matters related to ABCA1 and, in some cases, In vivo. The Huntington's disease study combines topics in areas such as Predictive testing, Genetics, Neuroscience and Trinucleotide repeat expansion. His studies in Cell biology integrate themes in fields like Apoptosis, Biochemistry, Huntingtin-interacting protein 1 and Caspase, Caspase 8.
Michael R. Hayden focuses on Internal medicine, Genetics, Huntingtin, Endocrinology and Huntington's disease. His research on Internal medicine frequently connects to adjacent areas such as Oncology. His Huntingtin research includes elements of Molecular biology, Neuroscience and Cell biology.
His research integrates issues of Caspase, Neurodegeneration and Palmitoylation in his study of Cell biology. His research on Endocrinology often connects related areas such as ABCA1. Disease and Pathology are the two main areas of interest in his Huntington's disease studies.
Michael R. Hayden mostly deals with Huntingtin, Cell biology, Internal medicine, Pharmacology and Neuroscience. His biological study focuses on Huntingtin Protein. His research in Cell biology intersects with topics in Oxidative stress, Palmitoylation, Neurodegeneration, Caspase 6 and Programmed cell death.
His Internal medicine research incorporates themes from Endocrinology, Oncology, Bioinformatics, Gastroenterology and Placebo. His Neuroscience study integrates concerns from other disciplines, such as White matter and Huntington's disease, Disease. His Mutant study deals with Molecular biology intersecting with Gene silencing.
His main research concerns Huntingtin, Neuroscience, Internal medicine, Cell biology and Pharmacology. Particularly relevant to Huntingtin Protein is his body of work in Huntingtin. His biological study spans a wide range of topics, including Neuroinflammation, Disease, Monoamine neurotransmitter and Monoamine oxidase.
His Disease research integrates issues from Penetrance and Trinucleotide repeat expansion. His research investigates the link between Internal medicine and topics such as Endocrinology that cross with problems in Bioinformatics. Michael R. Hayden has researched Cell biology in several fields, including Glutamate receptor, Neurotrophic factors, Neurodegeneration, Caspase and Huntington's disease.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Mutations in ABC1 in Tangier disease and familial high-density lipoprotein deficiency
A. Brooks-Wilson;M. Marcil;S. M. Clee;L.-H. Zhang.
Nature Genetics (1999)
Loss of Huntingtin-Mediated BDNF Gene Transcription in Huntington's Disease
Chiara Zuccato;Andrea Ciammola;Dorotea Rigamonti;Blair R. Leavitt.
Mutations in HFE2 cause iron overload in chromosome 1q-linked juvenile hemochromatosis.
George Papanikolaou;Mark E Samuels;Erwin H Ludwig;Marcia L E MacDonald.
Nature Genetics (2004)
Early mitochondrial calcium defects in Huntington's disease are a direct effect of polyglutamines
Alexander V. Panov;Claire-Anne Gutekunst;Blair R. Leavitt;Michael R. Hayden.
Nature Neuroscience (2002)
Huntingtin interacts with REST/NRSF to modulate the transcription of NRSE-controlled neuronal genes.
Chiara Zuccato;Marzia Tartari;Andrea Crotti;Donato Goffredo.
Nature Genetics (2003)
Targeted disruption of the Huntington's disease gene results in embryonic lethality and behavioral and morphological changes in heterozygotes.
Jamal Nasir;Stan B Floresco;John R O'Kusky;Virginia M Diewert.
A YAC mouse model for Huntington's disease with full-length mutant huntingtin, cytoplasmic toxicity, and selective striatal neurodegeneration.
J.Graeme Hodgson;Nadia Agopyan;Claire-Anne Gutekunst;Blair R Leavitt.
Aspirin for the Primary Prevention of Cardiovascular Events: A Summary of the Evidence for the U.S. Preventive Services Task Force
Michael Hayden;Michael Pignone;Christopher Phillips;Cynthia Mulrow.
Annals of Internal Medicine (2002)
Selective striatal neuronal loss in a YAC128 mouse model of Huntington disease
Elizabeth J. Slow;Jeremy van Raamsdonk;Daniel Rogers;Sarah H. Coleman.
Human Molecular Genetics (2003)
A gene encoding a putative GTPase regulator is mutated in familial amyotrophic lateral sclerosis 2.
Shinji Hadano;Collette K. Hand;Hitoshi Osuga;Yoshiko Yanagisawa.
Nature Genetics (2001)
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