Thomas Braun mostly deals with Cell biology, Molecular biology, Myogenesis, Myocyte and Internal medicine. Thomas Braun is interested in Stem cell, which is a branch of Cell biology. His Molecular biology research integrates issues from Endothelial stem cell, Cellular differentiation, RNA, Progenitor cell and Regulation of gene expression.
MyoD, Myogenin and MYF5 are the core of his Myogenesis study. His Myocyte research includes elements of Inflammation, TBX1, Programmed cell death and PAX3. Thomas Braun has included themes like Endocrinology, Signal transduction, Precursor cell and Downregulation and upregulation in his Internal medicine study.
Thomas Braun spends much of his time researching Cell biology, Internal medicine, Molecular biology, Endocrinology and Myocyte. His Cell biology research is multidisciplinary, incorporating elements of Chromatin, Embryonic stem cell and Skeletal muscle. He studies Skeletal muscle, focusing on Myogenesis in particular.
His Internal medicine study frequently involves adjacent topics like Cardiology. His Molecular biology study integrates concerns from other disciplines, such as Cellular differentiation, Mutant and Gene. His Myocyte study frequently draws connections to adjacent fields such as Cell growth.
His primary scientific interests are in Cell biology, Chromatin, Computational biology, Transcription factor and Stem cell. His Cell biology research incorporates themes from Embryonic stem cell and Cell. He works mostly in the field of Computational biology, limiting it down to topics relating to Genome and, in certain cases, CRISPR and Homology directed repair.
His Transcription factor study typically links adjacent topics like Signal transduction. His Stem cell research incorporates elements of Skeletal muscle, Lung and Adult stem cell. The Sirtuin study combines topics in areas such as Endocrinology and Internal medicine.
Thomas Braun focuses on Cell biology, Lung, Cancer research, Transcriptome and Cell type. His Cell biology research focuses on Stem cell in particular. His Lung study combines topics in areas such as Oxidative stress, Alternative oxidase, Apoptosis and In vivo.
His study in Cancer research is interdisciplinary in nature, drawing from both Cancer, Mitochondrial ROS, microRNA, Gene silencing and Epigenetics. His Transcriptome study deals with Neuroscience intersecting with Neurofilament. As part of one scientific family, Thomas Braun deals mainly with the area of Chromatin, narrowing it down to issues related to the Epigenome, and often Cell fate determination, Pioneer factor, Cellular differentiation and Transcription factor.
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MyoD or Myf-5 is required for the formation of skeletal muscle
Michael A. Rudnicki;Patrick N.J. Schnegelsberg;Ronald H. Stead;Thomas Braun.
Myostatin Mutation Associated with Gross Muscle Hypertrophy in a Child
Markus Schuelke;Kathryn R Wagner;Leslie E Stolz;Christoph Hübner.
The New England Journal of Medicine (2004)
Atheroprotective communication between endothelial cells and smooth muscle cells through miRNAs.
Eduard Hergenreider;Susanne Heydt;Karine Tréguer;Thomas Boettger.
Nature Cell Biology (2012)
Inactivation of MyoD in mice leads to up-regulation of the myogenic HLH gene Myf-5 and results in apparently normal muscle development.
Michael A. Rudnicki;Thomas Braun;Shuji Hinuma;Rudolf Jaenisch.
A novel human muscle factor related to but distinct from MyoD1 induces myogenic conversion in 10T1/2 fibroblasts.
T. Braun;G. Buschhausen‐Denker;E. Bober;E. Tannich.
The EMBO Journal (1989)
Targeted inactivation of the muscle regulatory gene Myf-5 results in abnormal rib development and perinatal death.
Thomas Braun;Michael A. Rudnicki;Hans-Henning Arnold;Rudolf Jaenisch.
Succinate Dehydrogenase Supports Metabolic Repurposing of Mitochondria to Drive Inflammatory Macrophages
Evanna L. Mills;Beth Kelly;Angela Logan;Ana S.H. Costa.
Long Noncoding RNA MALAT1 Regulates Endothelial Cell Function and Vessel Growth
Katharina M. Michalik;Xintian You;Yosif Manavski;Anuradha Doddaballapur.
Circulation Research (2014)
Acquisition of the contractile phenotype by murine arterial smooth muscle cells depends on the Mir143/145 gene cluster
Thomas Boettger;Nadine Beetz;Sawa Kostin;Johanna Schneider.
Journal of Clinical Investigation (2009)
Sirt7 Increases Stress Resistance of Cardiomyocytes and Prevents Apoptosis and Inflammatory Cardiomyopathy in Mice
Olesya Vakhrusheva;Christian Smolka;Praveen Gajawada;Sawa Kostin.
Circulation Research (2008)
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