His primary areas of investigation include Surgery, Internal medicine, Molecular biology, Cell biology and Pathology. Hikaru Matsuda has researched Surgery in several fields, including Respiratory disease, Carcinoma, Radiology and Cancer. His Internal medicine research is multidisciplinary, relying on both Gastroenterology, Endocrinology and Cardiology.
His Molecular biology study integrates concerns from other disciplines, such as Alternative complement pathway, Complement system, T cell, Immunology and Transfection. The Cell biology study which covers Apoptosis that intersects with Mitochondrion and Cytoplasm. His work investigates the relationship between Pathology and topics such as Neointima that intersect with problems in Myosin.
Internal medicine, Cardiology, Surgery, Anesthesia and Cardiopulmonary bypass are his primary areas of study. His Internal medicine study often links to related topics such as Endocrinology. His study in Ventricle, Stenosis, Tetralogy of Fallot, Artery and Aortic valve replacement falls under the purview of Cardiology.
Surgery is often connected to Radiology in his work.
Hikaru Matsuda mostly deals with Internal medicine, Surgery, Cardiology, Transplantation and Pathology. Hikaru Matsuda regularly ties together related areas like Endocrinology in his Internal medicine studies. The study incorporates disciplines such as Respiratory disease and Radiology in addition to Surgery.
His study in Myocyte extends to Cardiology with its themes. His Transplantation research is multidisciplinary, incorporating perspectives in Genetic transfer and Antigen. The various areas that Hikaru Matsuda examines in his Pathology study include Gene expression, Lung and Immunology.
The scientist’s investigation covers issues in Internal medicine, Surgery, Pathology, Endocrinology and Hepatocyte growth factor. His Internal medicine study incorporates themes from Gastroenterology and Cardiology. Many of his studies on Surgery apply to Radiology as well.
Hikaru Matsuda has included themes like Cancer research and Small intestine in his Pathology study. His Endocrinology research includes elements of Neovascularization, Transplantation and Urology. His biological study spans a wide range of topics, including Critical limb ischemia, Vascular resistance, Growth factor, Endothelium and Tissue culture.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria
Masashi Narita;Shigeomi Shimizu;Toshinori Ito;Thomas Chittenden.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Prevention of hypoxia-induced cell death by Bcl-2 and Bcl-xL
Shigeomi Shimizu;Yutaka Eguchi;Hiroaki Kosaka;Wataru Kamiike.
Nature (1995)
Effect of c-kit mutation on prognosis of gastrointestinal stromal tumors.
Masahiko Taniguchi;Toshirou Nishida;Seiichi Hirota;Koji Isozaki.
Cancer Research (1999)
FAMILIAL GASTROINTESTINAL STROMAL TUMOURS WITH GERMLINE MUTATION OF THE KIT GENE
Toshirou Nishida;Seiichi Hirota;Masahiko Taniguchi;Koji Hashimoto.
Nature Genetics (1998)
Induction of apoptosis as well as necrosis by hypoxia and predominant prevention of apoptosis by Bcl-2 and Bcl-XL.
Shigeomi Shimizu;Yutaka Eguchi;Wataru Kamiike;Yuko Itoh.
Cancer Research (1996)
Bcl-2 prevents apoptotic mitochondrial dysfunction by regulating proton flux
Shigeomi Shimizu;Yutaka Eguchi;Wataru Kamiike;Yoshimitsu Funahashi.
Proceedings of the National Academy of Sciences of the United States of America (1998)
New graft-implanting method for thoracic aortic aneurysm or dissection with a stented graft.
Kato M;Ohnishi K;Kaneko M;Ueda T.
Circulation (1996)
Bcl-2 blocks loss of mitochondrial membrane potential while ICE inhibitors act at a different step during inhibition of death induced by respiratory chain inhibitors.
Shimizu S;Eguchi Y;Kamiike W;Waguri S.
Oncogene (1996)
14-3-3 Interacts Directly with and Negatively Regulates Pro-apoptotic Bax
Masaya Nomura;Shigeomi Shimizu;Tomoyasu Sugiyama;Masashi Narita.
Journal of Biological Chemistry (2003)
A Truncated β-Catenin Disrupts the Interaction between E-Cadherin and α-Catenin: A Cause of Loss of Intercellular Adhesiveness in Human Cancer Cell Lines
Tsukasa Oyama;Yae Kanai;Atsushi Ochiai;Shingo Akimoto.
Cancer Research (1994)
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