Ikuya Nonaka spends much of his time researching Muscular dystrophy, Internal medicine, Endocrinology, Genetics and Molecular biology. His Muscular dystrophy research incorporates themes from Duchenne muscular dystrophy and Skeletal muscle. His Internal medicine study integrates concerns from other disciplines, such as Collagen VI, Bethlem myopathy, Ullrich congenital muscular dystrophy and Respiratory chain.
Basal lamina is closely connected to Laminin in his research, which is encompassed under the umbrella topic of Molecular biology. The various areas that Ikuya Nonaka examines in his Dystrophin study include Immunofluorescence and Pathology. His study in Mitochondrial myopathy is interdisciplinary in nature, drawing from both Encephalopathy and Heteroplasmy.
His primary areas of investigation include Pathology, Internal medicine, Endocrinology, Myopathy and Muscular dystrophy. His research brings together the fields of Anatomy and Pathology. Ikuya Nonaka works mostly in the field of Internal medicine, limiting it down to topics relating to Mitochondrial myopathy and, in certain cases, Cytochrome c oxidase.
As a part of the same scientific family, Ikuya Nonaka mostly works in the field of Myopathy, focusing on Mutation and, on occasion, Mitochondrial DNA. His work deals with themes such as Limb-girdle muscular dystrophy and Laminin, which intersect with Muscular dystrophy. His research investigates the connection between Skeletal muscle and topics such as Molecular biology that intersect with problems in Biochemistry.
Ikuya Nonaka mainly investigates Pathology, Myopathy, Internal medicine, Muscular dystrophy and Endocrinology. In his study, which falls under the umbrella issue of Pathology, Limb-girdle muscular dystrophy, Vacuole and Electromyography is strongly linked to Anatomy. His Myopathy study is associated with Genetics.
The Internal medicine study combines topics in areas such as Surgery, Weakness and Cardiology. The study incorporates disciplines such as Rimmed vacuoles, Pediatrics and Muscle pathology in addition to Muscular dystrophy. His study in Skeletal muscle, Muscle atrophy and Myocyte are all subfields of Endocrinology.
His scientific interests lie mostly in Internal medicine, Myopathy, Pathology, Muscular dystrophy and Endocrinology. His Myopathy study combines topics in areas such as Cytoplasm, Cytoplasmic inclusion, Mutation, Phenotype and Cerebellar ataxia. His Mutation research is included under the broader classification of Genetics.
His Pathology research is multidisciplinary, relying on both Hypotonia, Anatomy and Muscle pathology. His studies examine the connections between Muscular dystrophy and genetics, as well as such issues in Pediatrics, with regards to PTRF. His Endocrinology research integrates issues from Muscle biopsy, Sialic acid and Multiple Acyl-CoA Dehydrogenase Deficiency.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
A mutation in the tRNA Leu(UUR) gene associated with the MELAS subgroup of mitochondrial encephalomyopathies
Yu-Ichi Goto;Ikuya Nonaka;Satoshi Horai.
Myogenin gene disruption results in perinatal lethality because of severe muscle defect
Yoko Nabeshima;Kazunori Hanaoka;Michiko Hayasaka;Eisaku Esuml.
Mitochondrial DNA Deletions in Progressive External Ophthalmoplegia and Kearns-Sayre Syndrome
Carlos T. Moraes;Salvatore Dimauro;Massimo Zeviani;Anne Lombes.
The New England Journal of Medicine (1989)
An ancient retrotransposal insertion causes Fukuyama-type congenital muscular dystrophy
Kazuhiro Kobayashi;Yutaka Nakahori;Masashi Miyake;Kiichiro Matsumura.
Primary LAMP-2 deficiency causes X-linked vacuolar cardiomyopathy and myopathy (Danon disease)
Ichizo Nishino;Jin Fu;Kurenai Tanji;Takeshi Yamada.
Introduction of disease-related mitochondrial DNA deletions into HeLa cells lacking mitochondrial DNA results in mitochondrial dysfunction.
Jun-Ichi Hayashi;Shigeo Ohta;Aiko Kikuchi;Masakazu Takemitsu.
Proceedings of the National Academy of Sciences of the United States of America (1991)
Immunostaining of skeletal and cardiac muscle surface membrane with antibody against Duchenne muscular dystrophy peptide.
K Arahata;S Ishiura;T Ishiguro;T Tsukahara.
Mutations in the dystrophin-associated protein γ-sarcoglycan in chromosome 13 muscular dystrophy
Satoru Noguchi;Elizabeth M. McNally;Kamel Ben Othmane;Yasuko Hagiwara.
Inter-mitochondrial complementation: Mitochondria-specific system preventing mice from expression of disease phenotypes by mutant mtDNA
Kazuto Nakada;Kimiko Inoue;Kimiko Inoue;Tomoko Ono;Kotoyo Isobe.
Nature Medicine (2001)
Mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke‐like episodes (MELAS) A correlative study of the clinical features and mitochondrial DNA mutation
Y. Goto;S. Horai;T. Matsuoka;Y. Koga.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: