D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 96 Citations 34,979 816 World Ranking 5847 National Ranking 170

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Gene
  • Mutation

Corrado Angelini mainly investigates Particle physics, Electron–positron annihilation, Nuclear physics, Branching fraction and Internal medicine. His Particle physics study frequently involves adjacent topics like Lepton. He has included themes like Particle decay, Invariant mass, Particle identification and Pi in his Electron–positron annihilation study.

The study incorporates disciplines such as Asymmetry and Photon in addition to Nuclear physics. His studies deal with areas such as Resonance, X, Isospin, Analytical chemistry and Semileptonic decay as well as Branching fraction. The various areas that Corrado Angelini examines in his Internal medicine study include Gastroenterology, Endocrinology and Cardiology.

His most cited work include:

  • Dysferlin, a novel skeletal muscle gene, is mutated in Miyoshi myopathy and limb girdle muscular dystrophy (722 citations)
  • Evidence for an excess of B̄→D( *)τ -ν ̄τ decays (550 citations)
  • Measurement of an excess of B̄→D(*) τ-ν̄τ decays and implications for charged Higgs bosons (513 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Particle physics, Nuclear physics, Electron–positron annihilation, Branching fraction and Internal medicine. Particle physics is represented through his B meson, Meson, CP violation, Hadron and B-factory research. His Nuclear physics research integrates issues from Resonance and Asymmetry.

His Electron–positron annihilation study combines topics in areas such as Pi, Dalitz plot, Branching and Atomic physics. As part of one scientific family, Corrado Angelini deals mainly with the area of Branching fraction, narrowing it down to issues related to the Particle decay, and often Pair production. Corrado Angelini interconnects Endocrinology and Cardiology in the investigation of issues within Internal medicine.

He most often published in these fields:

  • Particle physics (35.93%)
  • Nuclear physics (28.75%)
  • Electron–positron annihilation (24.15%)

What were the highlights of his more recent work (between 2013-2021)?

  • Pathology (10.13%)
  • Internal medicine (13.86%)
  • Myopathy (5.59%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Pathology, Internal medicine, Myopathy, Particle physics and Disease. His studies examine the connections between Pathology and genetics, as well as such issues in Limb-girdle muscular dystrophy, with regards to Muscular dystrophy and Sarcoglycan. His study in Internal medicine is interdisciplinary in nature, drawing from both Endocrinology, Weakness and Cardiology.

In his study, Muscle weakness is inextricably linked to Muscle biopsy, which falls within the broad field of Myopathy. The Particle physics study which covers Nuclear physics that intersects with Resonance. His Disease research is multidisciplinary, relying on both Pediatrics and Bioinformatics.

Between 2013 and 2021, his most popular works were:

  • Search for a dark photon in e(+)e(-) collisions at BABAR. (310 citations)
  • The Physics of the B Factories (258 citations)
  • The m.3243A>G mitochondrial DNA mutation and related phenotypes. A matter of gender? (70 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Gene
  • Disease

Corrado Angelini mostly deals with Nuclear physics, Pathology, Internal medicine, Particle physics and Disease. His work in Nuclear physics tackles topics such as Resonance which are related to areas like Invariant mass. His Pathology research incorporates themes from Limb-girdle muscular dystrophy and Neurology.

In Internal medicine, Corrado Angelini works on issues like Endocrinology, which are connected to Myopathy, Weakness and Duchenne muscular dystrophy. His research related to Hadron, Branching fraction, Meson, CP violation and Asymmetry might be considered part of Particle physics. Corrado Angelini focuses mostly in the field of Electron–positron annihilation, narrowing it down to topics relating to Photon and, in certain cases, Radiation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The BABAR detector

B. Aubert;A. Bazan;A. Boucham;D. Boutigny.
Nuclear Instruments & Methods in Physics Research Section A-accelerators Spectrometers Detectors and Associated Equipment (2002)

3317 Citations

ATLAS: Technical proposal for a general-purpose p p experiment at the Large Hadron Collider at CERN

W.W. Armstrong;W. Burris;D.M. Gingrich;P. Green.
(1994)

1238 Citations

Dysferlin, a novel skeletal muscle gene, is mutated in Miyoshi myopathy and limb girdle muscular dystrophy

Jing Liu;Masashi Aoki;Isabel Illa;Chenyan Wu.
Nature Genetics (1998)

1037 Citations

Assessment of mitochondrial respiratory chain enzymatic activities on tissues and cultured cells

Marco Spinazzi;Alberto Casarin;Vanessa Pertegato;Leonardo Salviati.
Nature Protocols (2012)

723 Citations

MELAS mutation in mtDNA binding site for transcription termination factor causes defects in protein synthesis and in respiration but no change in levels of upstream and downstream mature transcripts.

A. Chomyn;A. Martinuzzi;M. Yoneda;A. Daga.
Proceedings of the National Academy of Sciences of the United States of America (1992)

649 Citations

Carnitine Deficiency of Human Skeletal Muscle with Associated Lipid Storage Myopathy: A New Syndrome

Andrew G. Engel;Corrado Angelini.
Science (1973)

625 Citations

MELAS: Clinical features, biochemistry, and molecular genetics

E. Ciafaloni;E. Ricci;S. Shanske;C. T. Moraes.
Annals of Neurology (1992)

579 Citations

β–sarcoglycan (A3b) mutations cause autosomal recessive muscular dystrophy with loss of the sarcoglycan complex

Carsten G. Bönnemann;Raju Modi;Satoru Noguchi;Yuji Mizuno.
Nature Genetics (1995)

543 Citations

EXPLORING THE MOLECULAR-BASIS FOR VARIABILITY AMONG PATIENTS WITH BECKER MUSCULAR-DYSTROPHY - DYSTROPHIN GENE AND PROTEIN STUDIES

Alan H. Beggs;Eric P. Hoffman;Judith R. Snyder;Kiichi Arahata.
American Journal of Human Genetics (1991)

499 Citations

Gene for Chronic Proximal Spinal Muscular Atrophies Maps To Chromosome-5q

J. Melki;S. Abdelhak;Peter Sheth;M. F. Bachelot.
Nature (1990)

476 Citations

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