Genetics, Mitochondrial DNA, Mitochondrion, Mitochondrial myopathy and Molecular biology are his primary areas of study. He focuses mostly in the field of Mitochondrial DNA, narrowing it down to matters related to Phenotype and, in some cases, DNA. The various areas that Yu-ichi Goto examines in his Mitochondrion study include RNA interference and Transcription.
His Mitochondrial myopathy research includes themes of Lactic acidosis, Internal medicine, Heteroplasmy and Mutation. His Lactic acidosis study which covers Encephalopathy that intersects with Pathology. His studies deal with areas such as Chromatin immunoprecipitation, Myopathy, MECP2 and Gene, Rett syndrome as well as Molecular biology.
Yu-ichi Goto spends much of his time researching Genetics, Mitochondrial DNA, Internal medicine, Pathology and Molecular biology. His Gene, Mutation, Point mutation, Heteroplasmy and Mutation investigations are all subjects of Genetics research. His research in Mitochondrial DNA intersects with topics in Mitochondrion, Cytochrome c oxidase and Mutant.
In his study, Myopathy is strongly linked to Endocrinology, which falls under the umbrella field of Internal medicine. His studies in Pathology integrate themes in fields like Cortical dysplasia, White matter, Cerebellar ataxia and Kearns–Sayre syndrome. His Mitochondrial myopathy research is multidisciplinary, incorporating elements of Encephalopathy and Mitochondrial encephalomyopathy.
His main research concerns Genetics, Internal medicine, Gene, Cell biology and Mitochondrion. He integrates Genetics and Pelizaeus Merzbacher like disease in his studies. His biological study spans a wide range of topics, including Gastroenterology, STIM1 and Endocrinology.
His Cell biology research integrates issues from Muscle disorder, Mutation, Protein biosynthesis, Muscular dystrophy and TRNA modification. His Mitochondrion study also includes fields such as
His primary scientific interests are in Genetics, Internal medicine, Mitochondrion, Gastroenterology and Molecular biology. Yu-ichi Goto frequently studies issues relating to White matter and Internal medicine. His Mitochondrion research includes themes of Oxidative phosphorylation, Heteroplasmy and Mitochondrial disease.
Yu-ichi Goto has researched Gastroenterology in several fields, including Odds ratio, Taurine, Cohort study and Cohort. The Molecular biology study combines topics in areas such as Compound heterozygosity, Pyruvate dehydrogenase complex and Induced pluripotent stem cell. His work in Cell biology tackles topics such as Endocrinology which are related to areas like Mutation.
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A mutation in the tRNA Leu(UUR) gene associated with the MELAS subgroup of mitochondrial encephalomyopathies
Yu-Ichi Goto;Ikuya Nonaka;Satoshi Horai.
Mitochondrial fission factor Drp1 is essential for embryonic development and synapse formation in mice
Naotada Ishihara;Masatoshi Nomura;Akihiro Jofuku;Hiroki Kato.
Nature Cell Biology (2009)
Hydrogen sulfide increases glutathione production and suppresses oxidative stress in mitochondria.
Yuka Kimura;Yu-Ichi Goto;Hideo Kimura.
Antioxidants & Redox Signaling (2010)
A subtype of diabetes mellitus associated with a mutation of mitochondrial DNA
T Kadowaki;H Kadowaki;Y Mori;K Tobe.
The New England Journal of Medicine (1994)
Introduction of disease-related mitochondrial DNA deletions into HeLa cells lacking mitochondrial DNA results in mitochondrial dysfunction.
Jun-Ichi Hayashi;Shigeo Ohta;Aiko Kikuchi;Masakazu Takemitsu.
Proceedings of the National Academy of Sciences of the United States of America (1991)
Inter-mitochondrial complementation: Mitochondria-specific system preventing mice from expression of disease phenotypes by mutant mtDNA
Kazuto Nakada;Kimiko Inoue;Kimiko Inoue;Tomoko Ono;Kotoyo Isobe.
Nature Medicine (2001)
Generation of mice with mitochondrial dysfunction by introducing mouse mtDNA carrying a deletion into zygotes
Kimiko Inoue;Kazuto Nakada;Atsuo Ogura;Kotoyo Isobe.
Nature Genetics (2000)
Mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke‐like episodes (MELAS) A correlative study of the clinical features and mitochondrial DNA mutation
Y. Goto;S. Horai;T. Matsuoka;Y. Koga.
Mutations in the integrin alpha7 gene cause congenital myopathy.
Yukiko K. Hayashi;Fan-Li Chou;Eva Engvall;Megumu Ogawa.
Nature Genetics (1998)
A new mtDNA mutation associated with mitochondrial myopathy, encephalopathy, lactic acidosis and stroke-like episodes (MELAS).
Yu-ichi Goto;Ikuya Nonaka;Satoshi Horai.
Biochimica et Biophysica Acta (1991)
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