What is he best known for?
The fields of study he is best known for:
The scientist’s investigation covers issues in Mitochondrial DNA, Molecular biology, Mitochondrion, Genetics and Gene.
Specifically, his work in Mitochondrial DNA is concerned with the study of Non-Mendelian inheritance.
His Molecular biology study combines topics in areas such as Cell culture, Mutation, Phenotype, Mutant and Respiratory function.
His Cell culture research includes elements of Tumor progression, Metastasis, Intracellular and Cytoplasmic hybrid.
The concepts of his Mitochondrion study are interwoven with issues in Complementation, Human mitochondrial genetics, Point mutation and Somatic cell.
His research investigates the link between Human mitochondrial genetics and topics such as Mitochondrial fission that cross with problems in Respiratory enzyme.
His most cited work include:
- ROS-generating mitochondrial DNA mutations can regulate tumor cell metastasis. (962 citations)
- Introduction of disease-related mitochondrial DNA deletions into HeLa cells lacking mitochondrial DNA results in mitochondrial dysfunction. (466 citations)
- Inter-mitochondrial complementation: Mitochondria-specific system preventing mice from expression of disease phenotypes by mutant mtDNA (353 citations)
What are the main themes of his work throughout his whole career to date?
Jun-Ichi Hayashi mainly investigates Mitochondrial DNA, Molecular biology, Genetics, Mitochondrion and Gene.
His work carried out in the field of Mitochondrial DNA brings together such families of science as Mutation, Phenotype and Mutant.
Jun-Ichi Hayashi focuses mostly in the field of Phenotype, narrowing it down to topics relating to Metastasis and, in certain cases, Overproduction.
Jun-Ichi Hayashi combines subjects such as Cell culture, Genome, DNA, Respiratory function and Nuclear DNA with his study of Molecular biology.
His work deals with themes such as Oxidative phosphorylation, Heteroplasmy and Somatic cell, which intersect with Mitochondrion.
His research integrates issues of Endocrinology and Internal medicine in his study of Gene.
He most often published in these fields:
- Mitochondrial DNA (77.30%)
- Molecular biology (47.24%)
- Genetics (44.79%)
What were the highlights of his more recent work (between 2014-2019)?
- Mitochondrial DNA (77.30%)
- Mitochondrion (41.10%)
- Genetics (44.79%)
In recent papers he was focusing on the following fields of study:
Jun-Ichi Hayashi focuses on Mitochondrial DNA, Mitochondrion, Genetics, Mutation and Gene.
The study incorporates disciplines such as Phenotype, Molecular biology, Endocrinology and Somatic cell in addition to Mitochondrial DNA.
His work is dedicated to discovering how Molecular biology, Missense mutation are connected with Mitochondrial respiratory chain and Cloning and other disciplines.
His research in Mitochondrion intersects with topics in Human mitochondrial genetics, Propofol, Pharmacology and Mitochondrial disease.
His Mutation research integrates issues from Malignant transformation, Cancer research, Lewis lung carcinoma and Colorectal cancer.
His studies in Gene integrate themes in fields like Cancer cell and Pathology.
Between 2014 and 2019, his most popular works were:
- Epigenetic regulation of the nuclear-coded GCAT and SHMT2 genes confers human age-associated mitochondrial respiration defects (50 citations)
- Propofol induces a metabolic switch to glycolysis and cell death in a mitochondrial electron transport chain-dependent manner. (36 citations)
- Interleukin-33 enhances programmed oncosis of ST2L-positive low-metastatic cells in the tumour microenvironment of lung cancer (25 citations)
In his most recent research, the most cited papers focused on:
Jun-Ichi Hayashi mostly deals with Mitochondrion, Mitochondrial DNA, Epigenetics, Cell biology and Gene.
Jun-Ichi Hayashi interconnects Reactive oxygen species, Programmed cell death and Propofol, Pharmacology in the investigation of issues within Mitochondrion.
The concepts of his Epigenetics study are interwoven with issues in Reprogramming and Somatic cell.
His research in Reprogramming intersects with topics in Phenotype, Human mitochondrial genetics and Induced pluripotent stem cell.
His biological study focuses on Mutation.
His Mutation study incorporates themes from Malignant transformation, Molecular biology, Lewis lung carcinoma and Cancer cell.
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