The scientist’s investigation covers issues in Mitochondrial DNA, Molecular biology, Mitochondrion, Genetics and Gene. Specifically, his work in Mitochondrial DNA is concerned with the study of Non-Mendelian inheritance. His Molecular biology study combines topics in areas such as Cell culture, Mutation, Phenotype, Mutant and Respiratory function.
His Cell culture research includes elements of Tumor progression, Metastasis, Intracellular and Cytoplasmic hybrid. The concepts of his Mitochondrion study are interwoven with issues in Complementation, Human mitochondrial genetics, Point mutation and Somatic cell. His research investigates the link between Human mitochondrial genetics and topics such as Mitochondrial fission that cross with problems in Respiratory enzyme.
Jun-Ichi Hayashi mainly investigates Mitochondrial DNA, Molecular biology, Genetics, Mitochondrion and Gene. His work carried out in the field of Mitochondrial DNA brings together such families of science as Mutation, Phenotype and Mutant. Jun-Ichi Hayashi focuses mostly in the field of Phenotype, narrowing it down to topics relating to Metastasis and, in certain cases, Overproduction.
Jun-Ichi Hayashi combines subjects such as Cell culture, Genome, DNA, Respiratory function and Nuclear DNA with his study of Molecular biology. His work deals with themes such as Oxidative phosphorylation, Heteroplasmy and Somatic cell, which intersect with Mitochondrion. His research integrates issues of Endocrinology and Internal medicine in his study of Gene.
Jun-Ichi Hayashi focuses on Mitochondrial DNA, Mitochondrion, Genetics, Mutation and Gene. The study incorporates disciplines such as Phenotype, Molecular biology, Endocrinology and Somatic cell in addition to Mitochondrial DNA. His work is dedicated to discovering how Molecular biology, Missense mutation are connected with Mitochondrial respiratory chain and Cloning and other disciplines.
His research in Mitochondrion intersects with topics in Human mitochondrial genetics, Propofol, Pharmacology and Mitochondrial disease. His Mutation research integrates issues from Malignant transformation, Cancer research, Lewis lung carcinoma and Colorectal cancer. His studies in Gene integrate themes in fields like Cancer cell and Pathology.
Jun-Ichi Hayashi mostly deals with Mitochondrion, Mitochondrial DNA, Epigenetics, Cell biology and Gene. Jun-Ichi Hayashi interconnects Reactive oxygen species, Programmed cell death and Propofol, Pharmacology in the investigation of issues within Mitochondrion. The concepts of his Epigenetics study are interwoven with issues in Reprogramming and Somatic cell.
His research in Reprogramming intersects with topics in Phenotype, Human mitochondrial genetics and Induced pluripotent stem cell. His biological study focuses on Mutation. His Mutation study incorporates themes from Malignant transformation, Molecular biology, Lewis lung carcinoma and Cancer cell.
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ROS-generating mitochondrial DNA mutations can regulate tumor cell metastasis.
Kaori Ishikawa;Kaori Ishikawa;Keizo Takenaga;Miho Akimoto;Nobuko Koshikawa.
Introduction of disease-related mitochondrial DNA deletions into HeLa cells lacking mitochondrial DNA results in mitochondrial dysfunction.
Jun-Ichi Hayashi;Shigeo Ohta;Aiko Kikuchi;Masakazu Takemitsu.
Proceedings of the National Academy of Sciences of the United States of America (1991)
Inter-mitochondrial complementation: Mitochondria-specific system preventing mice from expression of disease phenotypes by mutant mtDNA
Kazuto Nakada;Kimiko Inoue;Kimiko Inoue;Tomoko Ono;Kotoyo Isobe.
Nature Medicine (2001)
Human cells are protected from mitochondrial dysfunction by complementation of DNA products in fused mitochondria.
Tomoko Ono;Kotoyo Isobe;Kazuto Nakada;Jun-Ichi Hayashi.
Nature Genetics (2001)
Generation of mice with mitochondrial dysfunction by introducing mouse mtDNA carrying a deletion into zygotes
Kimiko Inoue;Kazuto Nakada;Atsuo Ogura;Kotoyo Isobe.
Nature Genetics (2000)
The mitochondrial bottleneck occurs without reduction of mtDNA content in female mouse germ cells
Liqin Cao;Hiroshi Shitara;Takuro Horii;Yasumitsu Nagao.
Nature Genetics (2007)
Maternal inheritance of mouse mtDNA in interspecific hybrids: segregation of the leaked paternal mtDNA followed by the prevention of subsequent paternal leakage.
Hiroshi Shitara;Jun-Ichi Hayashi;Sumiyo Takahama;Hideki Kaneda.
Mitochondria-related male infertility
Kazuto Nakada;Akitsugu Sato;Kayo Yoshida;Takashi Morita.
Proceedings of the National Academy of Sciences of the United States of America (2006)
Selective and Continuous Elimination of Mitochondria Microinjected Into Mouse Eggs From Spermatids, but Not From Liver Cells, Occurs Throughout Embryogenesis
Hiroshi Shitara;Hiroshi Shitara;Hideki Kaneda;Akitsugu Sato;Kimiko Inoue;Kimiko Inoue.
Gene therapy for progeny of mito-mice carrying pathogenic mtDNA by nuclear transplantation
Akitsugu Sato;Tomohiro Kono;Kazuto Nakada;Kaori Ishikawa.
Proceedings of the National Academy of Sciences of the United States of America (2005)
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