His scientific interests lie mostly in Mitochondrial DNA, Genetics, Mitochondrion, Molecular biology and Mitochondrial myopathy. His Mitochondrial DNA research includes themes of Genome, Mutant and DNA. His biological study spans a wide range of topics, including Structural biology, Oxidative phosphorylation, Transgene and Homologous recombination.
His work deals with themes such as Oxidative stress and Cell biology, which intersect with Oxidative phosphorylation. His studies deal with areas such as Cytochrome c oxidase, Cytochrome c, Cytochrome aa3, Direct repeat and Respirasome as well as Molecular biology. His research integrates issues of Skeletal muscle, Myopathy, Internal medicine, Pathology and Mitochondrial disease in his study of Mitochondrial myopathy.
Mitochondrial DNA, Mitochondrion, Genetics, Molecular biology and Cell biology are his primary areas of study. Carlos T. Moraes has included themes like Genome and Mutant in his Mitochondrial DNA study. His Mitochondrion study integrates concerns from other disciplines, such as Oxidative phosphorylation, Internal medicine and Endocrinology.
His Molecular biology study incorporates themes from Cytochrome c, Respiratory chain, mtDNA control region and Transgene. His Cell biology research incorporates themes from Apoptosis and Cell culture. The study incorporates disciplines such as Myopathy, Pathology and Kearns–Sayre syndrome in addition to Mitochondrial myopathy.
The scientist’s investigation covers issues in Mitochondrial DNA, Mitochondrion, Cell biology, Genetics and Heteroplasmy. His Mitochondrial DNA research includes elements of Molecular biology and DNA, Nuclease. His Molecular biology study combines topics in areas such as Mutation, Cancer research, Transgene and Phenotype.
His Mitochondrion study is associated with Biochemistry. His Cell biology study integrates concerns from other disciplines, such as Cell culture and Oxidative phosphorylation. Carlos T. Moraes interconnects Transcription activator-like effector nuclease, Mutant, Germline and Genetic enhancement in the investigation of issues within Heteroplasmy.
Carlos T. Moraes mainly investigates Mitochondrion, Mitochondrial DNA, Cell biology, Molecular biology and Genetics. As part of one scientific family, Carlos T. Moraes deals mainly with the area of Mitochondrion, narrowing it down to issues related to the Oxidative phosphorylation, and often Kidney metabolism, Cellular respiration, Cytochrome c oxidase and Electron Transport Complex IV. Carlos T. Moraes works in the field of Mitochondrial DNA, focusing on Heteroplasmy in particular.
His biological study spans a wide range of topics, including Biogenesis and Biochemistry. His Molecular biology study combines topics from a wide range of disciplines, such as Oxidative stress, Transgene, Mutation, Cytochrome c and Gene isoform. His studies in Human mitochondrial genetics, Genome and Mitochondrial replacement therapy are all subfields of Genetics research.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Mitochondrial DNA Deletions in Progressive External Ophthalmoplegia and Kearns-Sayre Syndrome
Carlos T. Moraes;Salvatore Dimauro;Massimo Zeviani;Anne Lombes.
The New England Journal of Medicine (1989)
Glycolytic oligodendrocytes maintain myelin and long-term axonal integrity
Ursula Fünfschilling;Lotti Marianna Supplie;Don Mahad;Don Mahad;Susann Boretius.
Nature (2012)
Deletions of mitochondrial DNA in Kearns‐Sayre syndrome
M. Zeviani;C. T. Moraes;S. DiMauro;H. Nakase.
Neurology (1988)
Increased muscle PGC-1α expression protects from sarcopenia and metabolic disease during aging
Tina Wenz;Susana G. Rossi;Richard L. Rotundo;Bruce M. Spiegelman.
Proceedings of the National Academy of Sciences of the United States of America (2009)
A direct repeat is a hotspot for large-scale deletion of human mitochondrial DNA
Eric A. Schon;Rosario Rizzuto;Carlos T. Moraes;Hirofumi Nakase.
Science (1989)
mtDNA depletion with variable tissue expression: a novel genetic abnormality in mitochondrial diseases.
Carlos T. Moraes;Sara Shanske;Hans Jürgen Tritschler;June R. Aprille.
American Journal of Human Genetics (1991)
Depletion of muscle mitochondrial DNA in AIDS patients with zidovudine-induced myopathy
E. Arnaudo;S. Shanske;S. DiMauro;E.A. Schon.
The Lancet (1991)
MELAS: Clinical features, biochemistry, and molecular genetics
E. Ciafaloni;E. Ricci;S. Shanske;C. T. Moraes.
Annals of Neurology (1992)
The Qo site of the mitochondrial complex III is required for the transduction of hypoxic signaling via reactive oxygen species production
Eric L. Bell;Tatyana A. Klimova;James Eisenbart;Carlos T. Moraes.
Journal of Cell Biology (2007)
Respiratory Complex III Is Required to Maintain Complex I in Mammalian Mitochondria
Rebeca Acı́n-Pérez;Marı́a Pilar Bayona-Bafaluy;Marı́a Pilar Bayona-Bafaluy;Patricio Fernández-Silva;Raquel Moreno-Loshuertos.
Molecular Cell (2004)
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