What is he best known for?
The fields of study he is best known for:
His primary scientific interests are in Genetics, Mitochondrial DNA, Mitochondrion, Molecular biology and Mutation.
His is doing research in Gene, Multiple mitochondrial DNA deletions, Point mutation, Haplotype and Allele, both of which are found in Genetics.
His work carried out in the field of Mitochondrion brings together such families of science as Reactive oxygen species and Mitochondrial disease.
His research in Mitochondrial disease intersects with topics in Mitochondrial biogenesis and Pathology.
His Molecular biology research also works with subjects such as
- Mitochondrial myopathy that connect with fields like Genome,
- Skeletal muscle and Leigh disease most often made with reference to Cytochrome c oxidase.
His studies deal with areas such as Protein subunit, NDUFS1, Mutant and Myopathy as well as Mutation.
His most cited work include:
- Identification and characterization of a spinal muscular atrophy-determining gene (2770 citations)
- Mitochondrial DNA Deletions in Progressive External Ophthalmoplegia and Kearns-Sayre Syndrome (828 citations)
- Human mitochondrial DNA deletions associated with mutations in the gene encoding Twinkle, a phage T7 gene 4-like protein localized in mitochondria. (715 citations)
What are the main themes of his work throughout his whole career to date?
Massimo Zeviani focuses on Genetics, Mitochondrial DNA, Mitochondrial disease, Mitochondrion and Molecular biology.
His Genetics study frequently links to related topics such as Ataxia.
His Mitochondrial DNA research is multidisciplinary, relying on both Respiratory chain, Point mutation and Pathology.
His Mitochondrial disease study combines topics in areas such as Myopathy, Mitochondrial encephalomyopathy, Nuclear gene, Mitochondrial respiratory chain and Mitochondrial biogenesis.
His Mitochondrion research incorporates elements of Oxidative phosphorylation, Internal medicine and Endocrinology.
In his study, Leigh disease is strongly linked to Cytochrome c oxidase, which falls under the umbrella field of Molecular biology.
He most often published in these fields:
- Genetics (42.42%)
- Mitochondrial DNA (39.02%)
- Mitochondrial disease (28.41%)
What were the highlights of his more recent work (between 2014-2021)?
- Mitochondrial disease (28.41%)
- Cell biology (13.83%)
- Mitochondrion (26.14%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Mitochondrial disease, Cell biology, Mitochondrion, Mitochondrial DNA and Genetics.
He combines subjects such as Phenotype, Disease, Mitochondrial myopathy and Mitochondrial respiratory chain with his study of Mitochondrial disease.
Massimo Zeviani studied Mitochondrion and Endocrinology that intersect with Leigh disease.
His Mitochondrial DNA study incorporates themes from Translation and RNase P.
Genetics is closely attributed to Ataxia in his research.
Massimo Zeviani works mostly in the field of Mutation, limiting it down to topics relating to Molecular biology and, in certain cases, Mitochondrial translation and Mutant.
Between 2014 and 2021, his most popular works were:
- The Opa1-Dependent Mitochondrial Cristae Remodeling Pathway Controls Atrophic, Apoptotic, and Ischemic Tissue Damage (214 citations)
- Transcription Factor EB Controls Metabolic Flexibility during Exercise (139 citations)
- Opa1 Overexpression Ameliorates the Phenotype of Two Mitochondrial Disease Mouse Models (134 citations)
In his most recent research, the most cited papers focused on:
His primary areas of study are Mitochondrial disease, Mitochondrion, Cell biology, Genetics and Mitochondrial DNA.
He has researched Mitochondrial disease in several fields, including Phenotype, Mitochondrial myopathy, Myopathy, Disease and Mitochondrial respiratory chain.
His Mitochondrion study combines topics from a wide range of disciplines, such as mitochondrial fusion, Chronic progressive external ophthalmoplegia, Mitochondrial Encephalomyopathies and Parkinsonism.
His studies in Cell biology integrate themes in fields like Autophagy, Mitophagy, Downregulation and upregulation and Programmed cell death.
The study of Genetics is intertwined with the study of Molecular biology in a number of ways.
His Mitochondrial DNA research integrates issues from Penetrance and Respiratory chain.
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