What is he best known for?
The fields of study he is best known for:
His scientific interests lie mostly in Genetics, Mitochondrial DNA, Mutation, Molecular biology and Gene.
Min-Xin Guan regularly links together related areas like Hearing loss in his Genetics studies.
His Mitochondrial DNA research is multidisciplinary, incorporating elements of Phenotype, Mitochondrial ribosome, Signal transduction and Sequence analysis.
His study explores the link between Molecular biology and topics such as RNA that cross with problems in Aminoglycoside.
His Mutation research incorporates elements of Penetrance, Neomycin and Haplogroup.
His Penetrance research includes themes of Expressivity and Haplotype.
His most cited work include:
- Observation of Electron-Antineutrino Disappearance at Daya Bay (1790 citations)
- Improved Measurement of Electron Antineutrino Disappearance at Daya Bay (284 citations)
- Maternally Inherited Aminoglycoside-Induced and Nonsyndromic Deafness Is Associated with the Novel C1494T Mutation in the Mitochondrial 12S rRNA Gene in a Large Chinese Family (284 citations)
What are the main themes of his work throughout his whole career to date?
Min-Xin Guan focuses on Genetics, Mitochondrial DNA, Mutation, Haplogroup and Penetrance.
While the research belongs to areas of Genetics, Min-Xin Guan spends his time largely on the problem of Hearing loss, intersecting his research to questions surrounding Nonsyndromic deafness.
Min-Xin Guan interconnects Molecular biology, Transfer RNA, Sequence analysis, Mitochondrion and Haplotype in the investigation of issues within Mitochondrial DNA.
His Mutation study frequently links to related topics such as Phenotype.
The various areas that Min-Xin Guan examines in his Haplogroup study include Proband, Point mutation, Homoplasmy, Age of onset and Human mitochondrial DNA haplogroup.
Penetrance is closely attributed to Expressivity in his research.
He most often published in these fields:
- Genetics (121.56%)
- Mitochondrial DNA (105.33%)
- Mutation (71.11%)
What were the highlights of his more recent work (between 2016-2021)?
- Argon (8.00%)
- Dark matter (8.89%)
- Mitochondrial DNA (105.33%)
In recent papers he was focusing on the following fields of study:
Min-Xin Guan spends much of his time researching Argon, Dark matter, Mitochondrial DNA, Mutation and Mitochondrion.
The study incorporates disciplines such as Neutrino and DarkSide in addition to Dark matter.
His Mitochondrial DNA study is concerned with the field of Genetics as a whole.
Within one scientific family, Min-Xin Guan focuses on topics pertaining to Hearing loss under Genetics, and may sometimes address concerns connected to Point mutation.
His Mutation research is multidisciplinary, relying on both Transfer RNA and Mutant.
As a member of one scientific family, he mostly works in the field of Mitochondrion, focusing on Retinal ganglion and, on occasion, ATP synthase.
Between 2016 and 2021, his most popular works were:
- Low-Mass Dark Matter Search with the DarkSide-50 Experiment (218 citations)
- DarkSide-20k: A 20 tonne two-phase LAr TPC for direct dark matter detection at LNGS (190 citations)
- Measurement of electron antineutrino oscillation based on 1230 days of operation of the Daya Bay experiment (150 citations)
In his most recent research, the most cited papers focused on:
His primary areas of study are Time projection chamber, Mutation, Mitochondrial DNA, Argon and Mitochondrion.
In Time projection chamber, Min-Xin Guan works on issues like Dark matter, which are connected to Nuclear physics and Cosmic ray.
The concepts of his Mitochondrial DNA study are interwoven with issues in Molecular biology and Mutant.
His Mitochondrion study is focused on Genetics in general.
His work on Mitochondrial disease, Gene, Atrophy and DNA repair as part of general Genetics study is frequently connected to Ferredoxin-NADP+ reductase activity, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
His Mitochondrial translation research incorporates themes from Penetrance, TRNA modification and Mutation.
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