The scientist’s investigation covers issues in Cell biology, Receptor, Molecular biology, T cell and T-cell receptor. His research in Cell biology is mostly concerned with Signal transduction. His Receptor research includes elements of Immune system, Immunology, Proinflammatory cytokine and Thymocyte.
His study looks at the relationship between Molecular biology and fields such as Antigen, as well as how they intersect with chemical problems. His study in T cell is interdisciplinary in nature, drawing from both Cell signaling, CD3 and Raft. His T-cell receptor study incorporates themes from Cell and CTLA-4.
His primary areas of study are Cell biology, Internal medicine, Molecular biology, Receptor and Immunology. His Cell biology research includes themes of T cell, Immunological synapse and T-cell receptor. The various areas that Takashi Saito examines in his T cell study include Cytotoxic T cell and Antigen.
His studies in T-cell receptor integrate themes in fields like CD28, CD3 and CD8. His Internal medicine research is multidisciplinary, incorporating elements of Endocrinology and Cardiology. Takashi Saito studies Immunology, focusing on Immune system in particular.
Takashi Saito mainly focuses on Neuroscience, Cell biology, Internal medicine, Disease and Amyloid precursor protein. The study incorporates disciplines such as Proteome and Microglia in addition to Neuroscience. His work carried out in the field of Cell biology brings together such families of science as Alzheimer's disease, Receptor, Amyloid and T-cell receptor.
His Internal medicine study integrates concerns from other disciplines, such as Gastroenterology, Endocrinology and Cardiology. Takashi Saito works mostly in the field of Disease, limiting it down to topics relating to Cognition and, in certain cases, Epilepsy. His work deals with themes such as Genetically modified mouse, Hippocampus and Presenilin, which intersect with Amyloid precursor protein.
Takashi Saito mainly investigates Neuroscience, Amyloid precursor protein, Cell biology, Internal medicine and Neurodegeneration. His Neuroscience research is multidisciplinary, relying on both Extracellular, Neuroinflammation and Genetically modified mouse. His studies deal with areas such as Mutation, Presenilin, Hippocampus and Transgene as well as Amyloid precursor protein.
His Cell biology research is multidisciplinary, incorporating perspectives in T cell, T-cell receptor and Receptor. T cell is a subfield of Immunology that he tackles. His work in Internal medicine covers topics such as Endocrinology which are related to areas like Voltage-dependent calcium channel, Thioflavin, Memantine and Glutathione.
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Induction of colonic regulatory T cells by indigenous Clostridium species
Koji Atarashi;Takeshi Tanoue;Tatsuichiro Shima;Akemi Imaoka.
Interaction of tyrosine-based sorting signals with clathrin-associated proteins
Hiroshi Ohno;Jay Stewart;Marie-Christine Fournier;Herbert Bosshart.
Fcγ Receptor–mediated Induction of Dendritic Cell Maturation and Major Histocompatibility Complex Class I–restricted Antigen Presentation after Immune Complex Internalization
Armelle Regnault;Danielle Lankar;Valérie Lacabanne;Ana Rodriguez.
Journal of Experimental Medicine (1999)
T cell receptor-proximal signals are sustained in peripheral microclusters and terminated in the central supramolecular activation cluster.
Rajat Varma;Gabriele Campi;Tadashi Yokosuka;Takashi Saito.
Design concepts for the Cherenkov Telescope Array CTA: An advanced facility for ground-based high-energy gamma-ray astronomy
M. Actis;G. Agnetta;F. Aharonian;A. Akhperjanian.
Experimental Astronomy (2011)
Functional annotation of a full-length mouse cDNA collection
J. Kawai;A. Shinagawa;K. Shibata;M. Yoshino.
Newly generated T cell receptor microclusters initiate and sustain T cell activation by recruitment of Zap70 and SLP-76.
Tadashi Yokosuka;Kumiko Sakata-Sogawa;Wakana Kobayashi;Michio Hiroshima.
Nature Immunology (2005)
Programmed cell death 1 forms negative costimulatory microclusters that directly inhibit T cell receptor signaling by recruiting phosphatase SHP2.
Tadashi Yokosuka;Masako Takamatsu;Wakana Kobayashi-Imanishi;Akiko Hashimoto-Tane.
Journal of Experimental Medicine (2012)
Dectin-2 Recognition of α-Mannans and Induction of Th17 Cell Differentiation Is Essential for Host Defense against Candida albicans
Shinobu Saijo;Satoshi Ikeda;Keiko Yamabe;Shigeru Kakuta.
Mincle is an ITAM-coupled activating receptor that senses damaged cells.
Sho Yamasaki;Eri Ishikawa;Machie Sakuma;Hiromitsu Hara.
Nature Immunology (2008)
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