D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 79 Citations 25,081 214 World Ranking 12302 National Ranking 370

Overview

What is he best known for?

The fields of study Marc Foretz is best known for:

  • Gene
  • Enzyme
  • Metabolism

His research on Endocrinology often connects related areas such as Skeletal muscle. His research ties Endocrinology and Skeletal muscle together. His research on Internal medicine frequently links to adjacent areas such as Insulin resistance. Borrowing concepts from Metformin, Marc Foretz weaves in ideas under Insulin resistance. His study deals with a combination of Metformin and Diabetes mellitus. Borrowing concepts from AMP-activated protein kinase, Marc Foretz weaves in ideas under Diabetes mellitus. He combines AMP-activated protein kinase and Phosphorylation in his research. Marc Foretz performs integrative study on Phosphorylation and Metabolism. In his study, he carries out multidisciplinary Metabolism and Gluconeogenesis research.

His most cited work include:

  • Cellular and molecular mechanisms of metformin: an overview (1295 citations)
  • Metformin inhibits hepatic gluconeogenesis in mice independently of the LKB1/AMPK pathway via a decrease in hepatic energy state (993 citations)
  • Metformin: From Mechanisms of Action to Therapies (875 citations)

What are the main themes of his work throughout his whole career to date

In the field of Glucose uptake and Insulin resistance Marc Foretz studies Insulin. In his research, Marc Foretz performs multidisciplinary study on Insulin resistance and Insulin. Marc Foretz conducts interdisciplinary study in the fields of Protein kinase A and Enzyme through his works. He regularly links together related areas like AMPK in his Enzyme studies. His work on Kinase expands to the thematically related AMPK. His Kinase study frequently draws connections between related disciplines such as Cell biology. Many of his studies on Cell biology apply to Signal transduction as well. Signal transduction and Protein kinase A are two areas of study in which he engages in interdisciplinary research. In his work, he performs multidisciplinary research in Biochemistry and Receptor.

Marc Foretz most often published in these fields:

  • Protein kinase A (70.19%)
  • AMPK (68.32%)
  • Biochemistry (66.46%)

What were the highlights of his more recent work (between 2019-2022)?

  • Endocrinology (71.43%)
  • Internal medicine (71.43%)
  • Biochemistry (71.43%)

In recent works Marc Foretz was focusing on the following fields of study:

In his papers, Marc Foretz integrates diverse fields, such as Endocrinology and Andrology. His work often combines Andrology and Endocrinology studies. Many of his studies on Internal medicine involve topics that are commonly interrelated, such as Resorption. Biochemistry is frequently linked to Beta oxidation in his study. While working on this project, he studies both Beta oxidation and Metabolism. In his work, Marc Foretz performs multidisciplinary research in Metabolism and Postprandial. AMPK is often connected to Protein kinase A in his work. His Protein kinase A study frequently links to related topics such as AMP-activated protein kinase. His AMP-activated protein kinase study frequently links to related topics such as Kinase.

Between 2019 and 2022, his most popular works were:

  • Inducible deletion of skeletal muscle AMPKα reveals that AMPK is required for nucleotide balance but dispensable for muscle glucose uptake and fat oxidation during exercise (27 citations)
  • The stress polarity signaling (SPS) pathway serves as a marker and a target in the leaky gut barrier: implications in aging and cancer (24 citations)
  • Transcriptional block of AMPK-induced autophagy promotes glutamate excitotoxicity in nutrient-deprived SH-SY5Y neuroblastoma cells (18 citations)

In his most recent research, the most cited works focused on:

  • Gene
  • Enzyme
  • Apoptosis

His Protein kinase A study frequently involves adjacent topics like AMP-activated protein kinase. Marc Foretz connects AMP-activated protein kinase with Phosphorylation in his research. Phosphorylation connects with themes related to AMPK in his study. His Kinase research extends to the thematically linked field of AMPK. He merges Kinase with Protein kinase A in his research. His Biochemistry study often links to related topics such as Beta oxidation. Many of his studies on Beta oxidation involve topics that are commonly interrelated, such as Biochemistry. His study connects Hypoglycemia and Endocrinology. He integrates Hypoglycemia with Diabetes mellitus in his study.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Cellular and molecular mechanisms of metformin: an overview

Benoit Viollet;Benoit Viollet;Benoit Viollet;Bruno Guigas;Nieves Sanz Garcia;Nieves Sanz Garcia;Nieves Sanz Garcia;Jocelyne Leclerc;Jocelyne Leclerc;Jocelyne Leclerc.
Clinical Science (2012)

1827 Citations

Metformin inhibits hepatic gluconeogenesis in mice independently of the LKB1/AMPK pathway via a decrease in hepatic energy state

Marc Foretz;Sophie Hébrard;Jocelyne Leclerc;Elham Zarrinpashneh.
Journal of Clinical Investigation (2010)

1302 Citations

Metformin: From Mechanisms of Action to Therapies

Marc Foretz;Bruno Guigas;Luc Bertrand;Michael Pollak.
Cell Metabolism (2014)

1020 Citations

Sterol regulatory element binding protein-1c is a major mediator of insulin action on the hepatic expression of glucokinase and lipogenesis-related genes

Marc Foretz;Colette Guichard;Pascal Ferré;Fabienne Foufelle.
Proceedings of the National Academy of Sciences of the United States of America (1999)

867 Citations

Biguanides suppress hepatic glucagon signalling by decreasing production of cyclic AMP

Russell A. Miller;Qingwei Chu;Jianxin Xie;Marc Foretz;Marc Foretz;Marc Foretz.
Nature (2013)

854 Citations

AMP-Activated Protein Kinase–Deficient Mice Are Resistant to the Metabolic Effects of Resveratrol

Jee Hyun Um;Sung-Jun Park;Hyeog Kang;Shutong Yang.
Diabetes (2010)

825 Citations

ADD1/SREBP-1c Is Required in the Activation of Hepatic Lipogenic Gene Expression by Glucose

Marc Foretz;Corinne Pacot;Isabelle Dugail;Patricia Lemarchand.
Molecular and Cellular Biology (1999)

642 Citations

Mitochondrial fission and remodelling contributes to muscle atrophy

Vanina Romanello;Eleonora Guadagnin;Ligia Gomes;Ira Roder.
The EMBO Journal (2010)

601 Citations

5'-AMP-activated protein kinase (AMPK) is induced by low-oxygen and glucose deprivation conditions found in solid-tumor microenvironments.

Keith R. Laderoute;Khalid Amin;Joy M. Calaoagan;Merrill Knapp.
Molecular and Cellular Biology (2006)

596 Citations

Characterization of the Role of AMP-Activated Protein Kinase in the Regulation of Glucose-Activated Gene Expression Using Constitutively Active and Dominant Negative Forms of the Kinase

Angela Woods;Dalila Azzout-Marniche;Marc Foretz;Silvie C. Stein.
Molecular and Cellular Biology (2000)

584 Citations

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