H-Index & Metrics Best Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Medicine D-index 160 Citations 107,483 690 World Ranking 271 National Ranking 175

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Enzyme

His primary scientific interests are in Alzheimer's disease, Pathology, Neurodegeneration, Cell biology and Neuroscience. His Alzheimer's disease research incorporates themes from Endocrinology, Genetically modified mouse and Degenerative disease. His studies deal with areas such as Central nervous system and In vivo as well as Genetically modified mouse.

His Neurodegeneration study combines topics from a wide range of disciplines, such as Alpha-synuclein, Glutamate receptor, Immunology, Amyloid precursor protein and Mitochondrion. His Cell biology course of study focuses on Molecular biology and Peptide sequence. His study on Neuroscience is mostly dedicated to connecting different topics, such as Cognitive decline.

His most cited work include:

  • Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis (4135 citations)
  • Physical basis of cognitive alterations in Alzheimer's disease: synapse loss is the major correlate of cognitive impairment. (3306 citations)
  • Alzheimer-type neuropathology in transgenic mice overexpressing V717F beta-amyloid precursor protein. (2313 citations)

What are the main themes of his work throughout his whole career to date?

Neuroscience, Pathology, Neurodegeneration, Cell biology and Alzheimer's disease are his primary areas of study. His Neuroscience study deals with Disease intersecting with Pathogenesis. His research in Pathology intersects with topics in Neocortex and Neuropil.

His Neurodegeneration research focuses on subjects like Immunology, which are linked to Central nervous system and Virology. Eliezer Masliah focuses mostly in the field of Cell biology, narrowing it down to topics relating to Alpha-synuclein and, in certain cases, Dementia with Lewy bodies. His Alzheimer's disease research is multidisciplinary, incorporating perspectives in Endocrinology and Degenerative disease.

He most often published in these fields:

  • Neuroscience (28.57%)
  • Pathology (21.84%)
  • Neurodegeneration (22.95%)

What were the highlights of his more recent work (between 2014-2021)?

  • Neuroscience (28.57%)
  • Disease (15.48%)
  • Neurodegeneration (22.95%)

In recent papers he was focusing on the following fields of study:

Eliezer Masliah mainly investigates Neuroscience, Disease, Neurodegeneration, Cell biology and Genetically modified mouse. His work in the fields of Neuroscience, such as Hippocampus, overlaps with other areas such as Context. The study incorporates disciplines such as Adiponectin, Immunology, Pathogenesis and Bioinformatics in addition to Disease.

His study focuses on the intersection of Neurodegeneration and fields such as Synucleinopathies with connections in the field of Dementia with Lewy bodies, Neuroinflammation and Atrophy. Cell biology connects with themes related to Autophagy in his study. His studies in Genetically modified mouse integrate themes in fields like Tau protein, Pharmacology, In vivo and Amyloid.

Between 2014 and 2021, his most popular works were:

  • NIA-AA Research Framework: Toward a biological definition of Alzheimer's disease (2145 citations)
  • Diagnosis and management of dementia with Lewy bodies Fourth consensus report of the DLB Consortium (1355 citations)
  • Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease (429 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Enzyme

His primary areas of study are Neuroscience, Cell biology, Neurodegeneration, Synucleinopathies and Alpha-synuclein. His Neuroscience research is multidisciplinary, relying on both Glutamate receptor, Dementia with Lewy bodies and Transgene. His research investigates the connection between Cell biology and topics such as Autophagy that intersect with problems in Neurotoxicity.

The Neurodegeneration study combines topics in areas such as Synapse, Biochemistry, Mitochondrion and Cognitive decline. His study explores the link between Genetically modified mouse and topics such as Amyloid precursor protein that cross with problems in Immunology and Synaptic plasticity. His Alzheimer's disease study introduces a deeper knowledge of Pathology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis

Manuela Neumann;Deepak M. Sampathu;Linda K. Kwong;Adam C. Truax.
Science (2006)

5207 Citations

Physical basis of cognitive alterations in Alzheimer's disease: synapse loss is the major correlate of cognitive impairment.

Robert D. Terry;Eliezer Masliah;David P. Salmon;Nelson Butters.
Annals of Neurology (1991)

4366 Citations

Alzheimer-type neuropathology in transgenic mice overexpressing V717F beta-amyloid precursor protein.

Dora Games;David Adams;Ree Alessandrini;Robin Barbour.
Nature (1995)

3118 Citations

NIA-AA Research Framework: Toward a biological definition of Alzheimer's disease

Clifford R. Jack;David A. Bennett;Kaj Blennow;Maria C. Carrillo.
Alzheimers & Dementia (2018)

2575 Citations

Dopaminergic Loss and Inclusion Body Formation in α-Synuclein Mice: Implications for Neurodegenerative Disorders

Eliezer Masliah;Edward Rockenstein;Isaac Veinbergs;Margaret Mallory.
Science (2000)

2549 Citations

High-level neuronal expression of abeta 1-42 in wild-type human amyloid protein precursor transgenic mice: synaptotoxicity without plaque formation.

Lennart Mucke;Eliezer Masliah;Gui-Qiu Yu;Margaret Mallory.
The Journal of Neuroscience (2000)

2447 Citations

Neuropathological Alterations in Alzheimer Disease

Alberto Serrano-Pozo;Matthew P. Frosch;Eliezer Masliah;Bradley T. Hyman.
Cold Spring Harbor Perspectives in Medicine (2011)

2244 Citations

|[alpha]|-Synuclein is phosphorylated in synucleinopathy lesions

Hideo Fujiwara;Masato Hasegawa;Naoshi Dohmae;Akiko Kawashima.
Nature Cell Biology (2002)

1687 Citations

National Institute on Aging-Alzheimer's Association Guidelines for the Neuropathologic Assessment of Alzheimer's Disease

Bradley T. Hyman;Creighton H. Phelps;Thomas G. Beach;Eileen H. Bigio.
Alzheimers & Dementia (2012)

1672 Citations

Molecular cloning of cDNA encoding an unrecognized component of amyloid in Alzheimer disease

Kenji Ueda;Hisashi Fukushima;Eliezer Masliah;Yu Xia.
Proceedings of the National Academy of Sciences of the United States of America (1993)

1589 Citations

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