2023 - Research.com Medicine in United States Leader Award
2023 - Research.com Biology and Biochemistry in United States Leader Award
2023 - Research.com Neuroscience in United States Leader Award
2022 - Research.com Best Scientist Award
2014 - Member of the National Academy of Medicine (NAM)
2003 - Fellow of the American Association for the Advancement of Science (AAAS)
2001 - Metlife Foundation Award for Medical Research in Alzheimer's Disease
Member of the Association of American Physicians
The scientist’s investigation covers issues in Alzheimer's disease, Pathology, Neuroscience, Senile plaques and Dementia. His biological study spans a wide range of topics, including Neuropathology, Apolipoprotein E and Cell biology. In his research, Neurotoxicity is intimately related to Neurodegeneration, which falls under the overarching field of Neuroscience.
His research investigates the link between Senile plaques and topics such as Amyloid precursor protein that cross with problems in Endocrinology. His Dementia research includes elements of Biomarker and Gerontology. His Degenerative disease study deals with Neurofibrillary tangle intersecting with Tau protein.
His primary scientific interests are in Alzheimer's disease, Pathology, Neuroscience, Cell biology and Disease. The concepts of his Alzheimer's disease study are interwoven with issues in Apolipoprotein E, Dementia and Amyloid. His Apolipoprotein E research is multidisciplinary, incorporating elements of Endocrinology, LDL receptor, Receptor, Allele and Genotype.
His work focuses on many connections between Pathology and other disciplines, such as Genetically modified mouse, that overlap with his field of interest in In vivo. His Neuroscience research includes themes of Tau protein and Neurodegeneration. In his research on the topic of Cell biology, Presenilin is strongly related with Amyloid precursor protein.
Bradley T. Hyman focuses on Cell biology, Disease, Alzheimer's disease, Neuroscience and Apolipoprotein E. His Cell biology research incorporates themes from Neurotoxicity, Tau protein, Genetically modified mouse, Transgene and Tauopathy. His studies examine the connections between Disease and genetics, as well as such issues in Microglia, with regards to Immune system.
His Alzheimer's disease study is concerned with Pathology in general. His work deals with themes such as Calcium and Neurodegeneration, which intersect with Neuroscience. In Apolipoprotein E, Bradley T. Hyman works on issues like Allele, which are connected to Lower risk.
His main research concerns Alzheimer's disease, Cell biology, Neuroscience, Pathology and Disease. Bradley T. Hyman combines subjects such as Genome-wide association study, Extracellular, Genetic epidemiology, Cognitive decline and Biomarker with his study of Alzheimer's disease. His Cognitive decline study integrates concerns from other disciplines, such as Proportional hazards model, Microglia and Senile plaques.
His Cell biology research integrates issues from Neurotoxicity, Tau protein and Transgene. The various areas that Bradley T. Hyman examines in his Neuroscience study include Microvesicles, Genetically modified mouse and Pathogenesis. His studies deal with areas such as Vesicle and Extracellular vesicles as well as Disease.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The diagnosis of dementia due to Alzheimer’s disease: Recommendations from the National Institute on Aging-Alzheimer’s Association workgroups on diagnostic guidelines for Alzheimer's disease
Guy M. McKhann;Guy M. McKhann;David S. Knopman;Howard Chertkow;Bradley T. Hyman.
Alzheimers & Dementia (2011)
An automated labeling system for subdividing the human cerebral cortex on MRI scans into gyral based regions of interest.
Rahul S. Desikan;Florent Ségonne;Bruce Fischl;Bruce Fischl;Brian T. Quinn.
NeuroImage (2006)
The Consortium to Establish a Registry for Alzheimer's Disease (CERAD): Part II. Standardization of the neuropathologic assessment of Alzheimer's disease
Mirra Ss;Heyman A;McKeel D;Sumi Sm.
Neurology (1991)
Effects of Age, Sex, and Ethnicity on the Association Between Apolipoprotein E Genotype and Alzheimer Disease: A Meta-analysis
Lindsay A. Farrer;L. Adrienne Cupples;Jonathan L. Haines;Bradley T Hyman.
JAMA (1997)
Effects of age, sex, and ethnicity on the association between apolipoprotein E genotype and Alzheimer disease. A meta-analysis. APOE and Alzheimer Disease Meta Analysis Consortium.
L A Farrer;L A Cupples;J L Haines;B Hyman.
JAMA (1997)
Neurofibrillary tangles but not senile plaques parallel duration and severity of Alzheimer's disease
Arriagada Pv;Growdon Jh;Hedley-Whyte Et;Hyman Bt.
Neurology (1992)
Neuropathological Alterations in Alzheimer Disease
Alberto Serrano-Pozo;Matthew P. Frosch;Eliezer Masliah;Bradley T. Hyman.
Cold Spring Harbor Perspectives in Medicine (2011)
Alzheimer's disease: cell-specific pathology isolates the hippocampal formation.
BT Hyman;GW Van Hoesen;AR Damasio;CL Barnes.
Science (1984)
Editorial on Consensus Recommendations for the Postmortem Diagnosis of Alzheimer Disease from the National Institute on Aging and the Reagan Institute Working Group on Diagnostic Criteria for the Neuropathological Assessment of Alzheimer Disease
Bradley T. Hyman;John Q. Trojanowski.
Journal of Neuropathology and Experimental Neurology (1997)
Tau Suppression in a Neurodegenerative Mouse Model Improves Memory Function
K. SantaCruz;J. Lewis;T. Spires;J. Paulson.
Science (2005)
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