D-Index & Metrics Best Publications
Research.com 2022 Best Scientist Award Badge
Neuroscience
USA
2023

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Best Scientists D-index 189 Citations 157,928 851 World Ranking 309 National Ranking 208
Neuroscience D-index 182 Citations 155,251 746 World Ranking 27 National Ranking 19
Medicine D-index 191 Citations 164,481 800 World Ranking 128 National Ranking 84
Biology and Biochemistry D-index 189 Citations 161,377 785 World Ranking 34 National Ranking 31

Research.com Recognitions

Awards & Achievements

2023 - Research.com Medicine in United States Leader Award

2023 - Research.com Biology and Biochemistry in United States Leader Award

2023 - Research.com Neuroscience in United States Leader Award

2022 - Research.com Best Scientist Award

2014 - Member of the National Academy of Medicine (NAM)

2003 - Fellow of the American Association for the Advancement of Science (AAAS)

2001 - Metlife Foundation Award for Medical Research in Alzheimer's Disease

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Alzheimer's disease

The scientist’s investigation covers issues in Alzheimer's disease, Pathology, Neuroscience, Senile plaques and Dementia. His biological study spans a wide range of topics, including Neuropathology, Apolipoprotein E and Cell biology. In his research, Neurotoxicity is intimately related to Neurodegeneration, which falls under the overarching field of Neuroscience.

His research investigates the link between Senile plaques and topics such as Amyloid precursor protein that cross with problems in Endocrinology. His Dementia research includes elements of Biomarker and Gerontology. His Degenerative disease study deals with Neurofibrillary tangle intersecting with Tau protein.

His most cited work include:

  • The diagnosis of dementia due to Alzheimer’s disease: Recommendations from the National Institute on Aging-Alzheimer’s Association workgroups on diagnostic guidelines for Alzheimer's disease (9237 citations)
  • An automated labeling system for subdividing the human cerebral cortex on MRI scans into gyral based regions of interest. (6385 citations)
  • The Consortium to Establish a Registry for Alzheimer's Disease (CERAD): Part II. Standardization of the neuropathologic assessment of Alzheimer's disease (3949 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Alzheimer's disease, Pathology, Neuroscience, Cell biology and Disease. The concepts of his Alzheimer's disease study are interwoven with issues in Apolipoprotein E, Dementia and Amyloid. His Apolipoprotein E research is multidisciplinary, incorporating elements of Endocrinology, LDL receptor, Receptor, Allele and Genotype.

His work focuses on many connections between Pathology and other disciplines, such as Genetically modified mouse, that overlap with his field of interest in In vivo. His Neuroscience research includes themes of Tau protein and Neurodegeneration. In his research on the topic of Cell biology, Presenilin is strongly related with Amyloid precursor protein.

He most often published in these fields:

  • Alzheimer's disease (32.78%)
  • Pathology (25.60%)
  • Neuroscience (23.33%)

What were the highlights of his more recent work (between 2015-2021)?

  • Cell biology (20.45%)
  • Disease (15.91%)
  • Alzheimer's disease (32.78%)

In recent papers he was focusing on the following fields of study:

Bradley T. Hyman focuses on Cell biology, Disease, Alzheimer's disease, Neuroscience and Apolipoprotein E. His Cell biology research incorporates themes from Neurotoxicity, Tau protein, Genetically modified mouse, Transgene and Tauopathy. His studies examine the connections between Disease and genetics, as well as such issues in Microglia, with regards to Immune system.

His Alzheimer's disease study is concerned with Pathology in general. His work deals with themes such as Calcium and Neurodegeneration, which intersect with Neuroscience. In Apolipoprotein E, Bradley T. Hyman works on issues like Allele, which are connected to Lower risk.

Between 2015 and 2021, his most popular works were:

  • Analysis of shared heritability in common disorders of the brain (726 citations)
  • Tau positron emission tomographic imaging in aging and early Alzheimer disease (527 citations)
  • Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease (429 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Enzyme

His main research concerns Alzheimer's disease, Cell biology, Neuroscience, Pathology and Disease. Bradley T. Hyman combines subjects such as Genome-wide association study, Extracellular, Genetic epidemiology, Cognitive decline and Biomarker with his study of Alzheimer's disease. His Cognitive decline study integrates concerns from other disciplines, such as Proportional hazards model, Microglia and Senile plaques.

His Cell biology research integrates issues from Neurotoxicity, Tau protein and Transgene. The various areas that Bradley T. Hyman examines in his Neuroscience study include Microvesicles, Genetically modified mouse and Pathogenesis. His studies deal with areas such as Vesicle and Extracellular vesicles as well as Disease.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The diagnosis of dementia due to Alzheimer’s disease: Recommendations from the National Institute on Aging-Alzheimer’s Association workgroups on diagnostic guidelines for Alzheimer's disease

Guy M. McKhann;Guy M. McKhann;David S. Knopman;Howard Chertkow;Bradley T. Hyman.
Alzheimers & Dementia (2011)

11517 Citations

An automated labeling system for subdividing the human cerebral cortex on MRI scans into gyral based regions of interest.

Rahul S. Desikan;Florent Ségonne;Bruce Fischl;Bruce Fischl;Brian T. Quinn.
NeuroImage (2006)

9482 Citations

The Consortium to Establish a Registry for Alzheimer's Disease (CERAD): Part II. Standardization of the neuropathologic assessment of Alzheimer's disease

Mirra Ss;Heyman A;McKeel D;Sumi Sm.
Neurology (1991)

7423 Citations

Effects of Age, Sex, and Ethnicity on the Association Between Apolipoprotein E Genotype and Alzheimer Disease: A Meta-analysis

Lindsay A. Farrer;L. Adrienne Cupples;Jonathan L. Haines;Bradley T Hyman.
JAMA (1997)

4636 Citations

Effects of age, sex, and ethnicity on the association between apolipoprotein E genotype and Alzheimer disease. A meta-analysis. APOE and Alzheimer Disease Meta Analysis Consortium.

L A Farrer;L A Cupples;J L Haines;B Hyman.
JAMA (1997)

3145 Citations

Neurofibrillary tangles but not senile plaques parallel duration and severity of Alzheimer's disease

Arriagada Pv;Growdon Jh;Hedley-Whyte Et;Hyman Bt.
Neurology (1992)

3056 Citations

Neuropathological Alterations in Alzheimer Disease

Alberto Serrano-Pozo;Matthew P. Frosch;Eliezer Masliah;Bradley T. Hyman.
Cold Spring Harbor Perspectives in Medicine (2011)

2794 Citations

Alzheimer's disease: cell-specific pathology isolates the hippocampal formation.

BT Hyman;GW Van Hoesen;AR Damasio;CL Barnes.
Science (1984)

2576 Citations

Editorial on Consensus Recommendations for the Postmortem Diagnosis of Alzheimer Disease from the National Institute on Aging and the Reagan Institute Working Group on Diagnostic Criteria for the Neuropathological Assessment of Alzheimer Disease

Bradley T. Hyman;John Q. Trojanowski.
Journal of Neuropathology and Experimental Neurology (1997)

2361 Citations

Tau Suppression in a Neurodegenerative Mouse Model Improves Memory Function

K. SantaCruz;J. Lewis;T. Spires;J. Paulson.
Science (2005)

2052 Citations

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