David S. Knopman mainly focuses on Alzheimer's disease, Dementia, Pathology, Internal medicine and Disease. His study in Alzheimer's disease is interdisciplinary in nature, drawing from both Neuroimaging, Neuroscience, Biomarker and Apolipoprotein E. His Dementia research includes elements of Gerontology, Psychiatry, Cognition, Prospective cohort study and Cohort.
His study looks at the relationship between Pathology and fields such as Magnetic resonance imaging, as well as how they intersect with chemical problems. His Internal medicine research incorporates elements of Oncology and Cardiology. His research integrates issues of Alzheimer's Disease Neuroimaging Initiative and Alzheimer's disease biomarkers in his study of Pittsburgh compound B.
His primary areas of study are Dementia, Internal medicine, Pathology, Alzheimer's disease and Disease. His Dementia research focuses on Cognition and how it relates to Audiology. His Internal medicine study incorporates themes from Oncology and Cardiology.
David S. Knopman regularly ties together related areas like Magnetic resonance imaging in his Pathology studies. His studies deal with areas such as Biomarker, Neuroimaging, Cognitive impairment and Amyloid as well as Alzheimer's disease. In most of his Disease studies, his work intersects topics such as Neuroscience.
David S. Knopman focuses on Internal medicine, Dementia, Cognition, Disease and Cognitive decline. His Alzheimer's disease and Cognitive impairment investigations are all subjects of Internal medicine research. His work carried out in the field of Alzheimer's disease brings together such families of science as Biomarker and Dementia with Lewy bodies.
His Dementia research is under the purview of Pathology. The concepts of his Cognition study are interwoven with issues in Neuroimaging, Association and Audiology. The various areas that David S. Knopman examines in his Cognitive decline study include White matter, Gerontology and Amyloid.
David S. Knopman mostly deals with Internal medicine, Dementia, Cognition, Alzheimer's disease and Cohort study. His biological study spans a wide range of topics, including Oncology and Cardiology. His Dementia study is related to the wider topic of Pathology.
His Pathology study combines topics from a wide range of disciplines, such as Hippocampal sclerosis and Temporal lobe. David S. Knopman interconnects Neuroimaging and Cognitive decline in the investigation of issues within Cognition. His studies in Alzheimer's disease integrate themes in fields like Biomarker, Dementia with Lewy bodies, Standardized uptake value and Psychiatry.
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The diagnosis of dementia due to Alzheimer’s disease: Recommendations from the National Institute on Aging-Alzheimer’s Association workgroups on diagnostic guidelines for Alzheimer's disease
Guy M. McKhann;Guy M. McKhann;David S. Knopman;Howard Chertkow;Bradley T. Hyman.
Alzheimers & Dementia (2011)
Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS
Mariely DeJesus-Hernandez;Ian R. Mackenzie;Bradley F. Boeve;Adam L. Boxer.
Neuron (2011)
Hypothetical model of dynamic biomarkers of the Alzheimer's pathological cascade
Clifford R Jack;David S Knopman;William J Jagust;Leslie M Shaw.
Lancet Neurology (2010)
Sensitivity of revised diagnostic criteria for the behavioural variant of frontotemporal dementia.
Katya Rascovsky;John R. Hodges;David Knopman;Mario F. Mendez.
Brain (2011)
Tracking pathophysiological processes in Alzheimer's disease: an updated hypothetical model of dynamic biomarkers.
Clifford R Jack;David S Knopman;William J Jagust;Ronald C Petersen.
Lancet Neurology (2013)
Introduction to the recommendations from the National Institute on Aging-Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease
Clifford R. Jack;Marilyn S. Albert;David S. Knopman;Guy M. McKhann.
Alzheimers & Dementia (2011)
Mild cognitive impairment: ten years later.
Ronald Carl Petersen;Rosebud O Roberts;David S Knopman;Bradley F Boeve.
JAMA Neurology (2009)
A 30-Week Randomized Controlled Trial of High-Dose Tacrine in Patients With Alzheimer's Disease
Margaret J. Knapp;David S. Knopman;Paul R. Solomon;William W. Pendlebury.
JAMA (1994)
Serial PIB and MRI in normal, mild cognitive impairment and Alzheimer's disease: implications for sequence of pathological events in Alzheimer's disease.
Clifford R. Jack Jr.;Val J. Lowe;Stephen D Weigand;Heather J. Wiste.
Brain (2009)
Primary age-related tauopathy (PART): a common pathology associated with human aging
John F. Crary;John Q. Trojanowski;Julie A. Schneider;Jose F. Abisambra.
Acta Neuropathologica (2014)
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