Matthew L. Senjem spends much of his time researching Alzheimer's disease, Pathology, Internal medicine, Dementia and Magnetic resonance imaging. Matthew L. Senjem has included themes like Positron emission tomography, Apolipoprotein E, Atrophy and Amyloidosis in his Alzheimer's disease study. His study in Atrophy is interdisciplinary in nature, drawing from both Frontal lobe, Neuroscience, Cohort and Grey matter.
His Neuroscience research incorporates themes from Voxel-based morphometry and Audiology. He has researched Pathology in several fields, including Biomarker, Neuroimaging and Brain mapping. His Internal medicine research incorporates elements of Oncology and Cardiology.
His primary scientific interests are in Pathology, Dementia, Internal medicine, Alzheimer's disease and Atrophy. As part of the same scientific family, Matthew L. Senjem usually focuses on Pathology, concentrating on Magnetic resonance imaging and intersecting with Aphasia. His research investigates the connection with Dementia and areas like Neuroscience which intersect with concerns in Primary progressive aphasia.
As a member of one scientific family, Matthew L. Senjem mostly works in the field of Internal medicine, focusing on Cardiology and, on occasion, Hyperintensity. His Alzheimer's disease research focuses on Pittsburgh compound B in particular. His work investigates the relationship between Atrophy and topics such as Grey matter that intersect with problems in Audiology.
Matthew L. Senjem focuses on Pathology, Internal medicine, Dementia, Standardized uptake value and Neuroimaging. His Pathology study integrates concerns from other disciplines, such as Temporal lobe and Grey matter. His study in Alzheimer's disease and Cohort study is done as part of Internal medicine.
His work deals with themes such as Biomarker, Dementia with Lewy bodies and Cohort, which intersect with Alzheimer's disease. The concepts of his Dementia study are interwoven with issues in Apolipoprotein E and Putamen. The Positron emission tomography study combines topics in areas such as Primary progressive aphasia and Magnetic resonance imaging.
His main research concerns Pathology, Temporal lobe, Standardized uptake value, Magnetic resonance imaging and Dementia. He regularly links together related areas like Hyperintensity in his Pathology studies. While the research belongs to areas of Magnetic resonance imaging, he spends his time largely on the problem of Frontotemporal dementia, intersecting his research to questions surrounding Tau protein, Aphasia, Age of onset and Neuropsychology.
To a larger extent, Matthew L. Senjem studies Internal medicine with the aim of understanding Dementia. His research in Atrophy intersects with topics in Neurodegeneration and Grey matter. He combines subjects such as Interquartile range, Cohort study, Dementia with Lewy bodies, Biomarker and Cohort with his study of Alzheimer's disease.
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Serial PIB and MRI in normal, mild cognitive impairment and Alzheimer's disease: implications for sequence of pathological events in Alzheimer's disease.
Clifford R. Jack Jr.;Val J. Lowe;Stephen D Weigand;Heather J. Wiste.
11C PiB and structural MRI provide complementary information in imaging of Alzheimer's disease and amnestic mild cognitive impairment.
Clifford R. Jack;Val J. Lowe;Matthew L. Senjem;Stephen D. Weigand.
An operational approach to National Institute on Aging–Alzheimer's Association criteria for preclinical Alzheimer disease
Clifford R. Jack;David S. Knopman;Stephen D. Weigand;Heather J. Wiste.
Annals of Neurology (2012)
Brain beta-amyloid measures and magnetic resonance imaging atrophy both predict time-to-progression from mild cognitive impairment to Alzheimer’s disease
Clifford R. Jack;Heather J. Wiste;Prashanthi Vemuri;Stephen D. Weigand.
Alzheimer's disease diagnosis in individual subjects using structural MR images: Validation studies
Prashanthi Vemuri;Jeffrey L. Gunter;Matthew L. Senjem;Jennifer L. Whitwell.
Defining imaging biomarker cut points for brain aging and Alzheimer's disease
Clifford R. Jack;Heather J. Wiste;Stephen D. Weigand;Terry M. Therneau.
Alzheimers & Dementia (2017)
Neuroimaging signatures of frontotemporal dementia genetics: C9ORF72, tau, progranulin and sporadics.
Jennifer L. Whitwell;Stephen D. Weigand;Bradley F. Boeve;Matthew L. Senjem.
MRI correlates of neurofibrillary tangle pathology at autopsy: a voxel-based morphometry study.
J. L. Whitwell;K. A. Josephs;M. E. Murray;K. Kantarci.
Non-Stationarity in the ``Resting Brain's'' Modular Architecture
David T. Jones;Prashanthi Vemuri;Matthew C. Murphy;Jeffrey L. Gunter.
PLOS ONE (2012)
Neuroimaging correlates of pathologically defined subtypes of Alzheimer's disease: a case-control study
Jennifer L Whitwell;Dennis W Dickson;Melissa E Murray;Stephen D Weigand.
Lancet Neurology (2012)
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