D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 102 Citations 33,930 407 World Ranking 3397 National Ranking 1945

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Disease
  • Gene

Keith A. Josephs focuses on Pathology, Frontotemporal dementia, Frontotemporal lobar degeneration, Dementia and Atrophy. His Pathology research incorporates elements of Magnetic resonance imaging and Neuroscience. Keith A. Josephs combines subjects such as Neuropathology, Amyotrophic lateral sclerosis and Frontal lobe with his study of Frontotemporal dementia.

The Frontotemporal lobar degeneration study combines topics in areas such as Genetics, Charged multivesicular body protein 2B, Age of onset, Primary progressive aphasia and Vascular dementia. His Dementia research includes themes of Differential diagnosis, Psychiatry, Neuroimaging and Degenerative disease. His biological study spans a wide range of topics, including Cerebellum, Central nervous system disease, Voxel-based morphometry and Grey matter.

His most cited work include:

  • Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS (3097 citations)
  • Sensitivity of revised diagnostic criteria for the behavioural variant of frontotemporal dementia. (2546 citations)
  • Criteria for the diagnosis of corticobasal degeneration (798 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Pathology, Atrophy, Frontotemporal dementia, Dementia and Progressive supranuclear palsy. His study explores the link between Pathology and topics such as Magnetic resonance imaging that cross with problems in Positron emission tomography. His Atrophy research is multidisciplinary, incorporating perspectives in Frontal lobe, Pittsburgh compound B, Temporal lobe and Voxel-based morphometry.

His Frontotemporal dementia study contributes to a more complete understanding of Internal medicine. His Dementia study combines topics from a wide range of disciplines, such as Psychiatry, Pediatrics and Audiology. The concepts of his Progressive supranuclear palsy study are interwoven with issues in Apraxia, Neuropathology, Neuroscience and Tauopathy.

He most often published in these fields:

  • Pathology (39.16%)
  • Atrophy (19.50%)
  • Frontotemporal dementia (18.66%)

What were the highlights of his more recent work (between 2019-2021)?

  • Pathology (39.16%)
  • Audiology (14.45%)
  • Disease (15.63%)

In recent papers he was focusing on the following fields of study:

Pathology, Audiology, Disease, Apraxia and Internal medicine are his primary areas of study. His work in Pathology is not limited to one particular discipline; it also encompasses Entorhinal cortex. His Disease research integrates issues from Phenotype, Pathological and Neuroscience.

His work focuses on many connections between Apraxia and other disciplines, such as Progressive supranuclear palsy, that overlap with his field of interest in Putamen, Physical medicine and rehabilitation, Striatum and Parkinsonism. As part of his studies on Frontotemporal lobar degeneration, Keith A. Josephs frequently links adjacent subjects like C9orf72. He is involved in the study of Frontotemporal dementia that focuses on Primary progressive aphasia in particular.

Between 2019 and 2021, his most popular works were:

  • Progressive dysexecutive syndrome due to Alzheimer’s disease: a description of 55 cases and comparison to other phenotypes (16 citations)
  • Longitudinal neuroimaging biomarkers differ across Alzheimer's disease phenotypes. (12 citations)
  • Truncated stathmin-2 is a marker of TDP-43 pathology in frontotemporal dementia. (10 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Disease
  • Gene

His scientific interests lie mostly in Pathology, Progressive supranuclear palsy, Disease, Dementia and Internal medicine. His Pathology research is multidisciplinary, incorporating elements of Entorhinal cortex and Grey matter. His work deals with themes such as Apraxia, Striatum, Physical medicine and rehabilitation and Putamen, which intersect with Progressive supranuclear palsy.

His study involves Frontotemporal dementia and TARDBP, a branch of Dementia. His studies deal with areas such as Precentral gyrus and Frontotemporal lobar degeneration as well as Pittsburgh compound B. The study incorporates disciplines such as Amyloid β and Genotype in addition to Frontotemporal lobar degeneration.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS

Mariely DeJesus-Hernandez;Ian R. Mackenzie;Bradley F. Boeve;Adam L. Boxer.
Neuron (2011)

3949 Citations

Sensitivity of revised diagnostic criteria for the behavioural variant of frontotemporal dementia.

Katya Rascovsky;John R. Hodges;David Knopman;Mario F. Mendez.
Brain (2011)

3292 Citations

Criteria for the diagnosis of corticobasal degeneration

Melissa J. Armstrong;Irene Litvan;Anthony E. Lang;Thomas H. Bak.
Neurology (2013)

1025 Citations

Clinical diagnosis of progressive supranuclear palsy: The movement disorder society criteria

Günter U Höglinger;Gesine Respondek;Maria Stamelou;Carolin Kurz.
Movement Disorders (2017)

729 Citations

Pathological gambling caused by drugs used to treat Parkinson disease.

M. Leann Dodd;Kevin J. Klos;James H. Bower;Yonas E. Geda.
JAMA Neurology (2005)

678 Citations

Mutations in progranulin are a major cause of ubiquitin-positive frontotemporal lobar degeneration

Jennifer Gass;Ashley Cannon;Ian R. Mackenzie;Bradley Boeve.
Human Molecular Genetics (2006)

575 Citations

Clinicopathologic analysis of frontotemporal and corticobasal degenerations and PSP

K. A. Josephs;R. C. Petersen;D. S. Knopman;B. F. Boeve.
Neurology (2006)

558 Citations

The spectrum of pathological involvement of the striatonigral and olivopontocerebellar systems in multiple system atrophy : clinicopathological correlations

Tetsutaro Ozawa;Dominic Paviour;Dominic Paviour;Niall P. Quinn;Keith A. Josephs.
Brain (2004)

426 Citations

Evidence that incidental Lewy body disease is pre-symptomatic Parkinson’s disease

Dennis W. Dickson;Hiroshige Fujishiro;Anthony DelleDonne;Joshua Menke.
Acta Neuropathologica (2008)

407 Citations

Neuropathological background of phenotypical variability in frontotemporal dementia

Keith A. Josephs;John R. Hodges;Julie S. Snowden;Ian R. Mackenzie.
Acta Neuropathologica (2011)

389 Citations

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