D-Index & Metrics Best Publications
Research.com 2023 Best Female Scientist Award Badge

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 109 Citations 48,488 474 World Ranking 3364 National Ranking 1910
Best female scientists D-index 109 Citations 48,641 483 World Ranking 577 National Ranking 349

Research.com Recognitions

Awards & Achievements

2023 - Research.com Best Female Scientist Award

2022 - Research.com Best Female Scientist Award

Overview

What is she best known for?

The fields of study Rosa Rademakers is best known for:

  • Alzheimer's disease
  • Dementia
  • Neurodegeneration

Rosa Rademakers performs integrative study on Frontotemporal dementia and Corticobasal degeneration in her works. She combines Corticobasal degeneration and Parkinsonism in her research. In most of her Parkinsonism studies, her work intersects topics such as Disease. Disease is closely attributed to Neurodegeneration in her research. She integrates many fields in her works, including Neurodegeneration and Alzheimer's disease. In her work, she performs multidisciplinary research in Alzheimer's disease and Temporal lobe. With her scientific publications, her incorporates both Temporal lobe and Hippocampal sclerosis. She integrates Hippocampal sclerosis and Epilepsy in her research. She regularly links together related areas like Psychiatry in her Epilepsy studies.

Her most cited work include:

  • The role of tau (MAPT) in frontotemporal dementia and related tauopathies (293 citations)
  • Abnormal TDP-43 immunoreactivity in AD modifies clinicopathologic and radiologic phenotype (197 citations)

What are the main themes of her work throughout her whole career to date

Her study connects Amyloid precursor protein and Disease. She integrates Amyloid precursor protein and Tau protein in her research. In her study, she carries out multidisciplinary Tau protein and Tauopathy research. Rosa Rademakers combines Tauopathy and Corticobasal degeneration in her research. Rosa Rademakers combines Corticobasal degeneration and Parkinsonism in her studies. She incorporates Parkinsonism and Progressive supranuclear palsy in her research. She undertakes interdisciplinary study in the fields of Progressive supranuclear palsy and Frontotemporal lobar degeneration through her research. Rosa Rademakers performs integrative study on Frontotemporal lobar degeneration and C9orf72 in her works. By researching both C9orf72 and Frontotemporal dementia, Rosa Rademakers produces research that crosses academic boundaries.

Rosa Rademakers most often published in these fields:

  • Disease (100.00%)
  • Alzheimer's disease (69.23%)
  • Internal medicine (61.54%)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS

Mariely DeJesus-Hernandez;Ian R. Mackenzie;Bradley F. Boeve;Adam L. Boxer.
Neuron (2011)

4478 Citations

TREM2 Variants in Alzheimer's Disease

Rita Guerreiro;Rita Guerreiro;Aleksandra Wojtas;Jose Bras;Minerva Carrasquillo.
The New England Journal of Medicine (2013)

2446 Citations

Mutations in progranulin cause tau-negative frontotemporal dementia linked to chromosome 17

Matt Baker;Ian R. Mackenzie;Stuart M. Pickering-Brown;Jennifer Gass.
Nature (2006)

2009 Citations

Null mutations in progranulin cause ubiquitin-positive frontotemporal dementia linked to chromosome 17q21

Marc Cruts;Ilse Gijselinck;Julie van der Zee;Sebastiaan Engelborghs.
Nature (2006)

1520 Citations

Mutations in prion-like domains in hnRNPA2B1 and hnRNPA1 cause multisystem proteinopathy and ALS

Hong Joo Kim;Nam Chul Kim;Yong Dong Wang;Emily A. Scarborough.
Nature (2013)

1262 Citations

Unconventional Translation of C9ORF72 GGGGCC Expansion Generates Insoluble Polypeptides Specific to c9FTD/ALS

Peter E.A. Ash;Kevin F. Bieniek;Tania F. Gendron;Thomas Caulfield.
Neuron (2013)

1005 Citations

TDP-43 and FUS in amyotrophic lateral sclerosis and frontotemporal dementia

Ian R.A. Mackenzie;Rosa Rademakers;Manuela Neumann.
Lancet Neurology (2010)

982 Citations

A new subtype of frontotemporal lobar degeneration with FUS pathology.

Manuela Neumann;Rosa Rademakers;Sigrun Roeber;Matt Baker.
Brain (2009)

766 Citations

TDP-43 A315T mutation in familial motor neuron disease.

Michael A. Gitcho;Robert H. Baloh;Sumi Chakraverty;Kevin Mayo.
Annals of Neurology (2008)

730 Citations

Limbic-predominant age-related TDP-43 encephalopathy (LATE): consensus working group report.

Peter T. Nelson;Dennis W. Dickson;John Q. Trojanowski;Clifford R. Jack.
Brain (2019)

626 Citations

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Mayo Clinic

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