2023 - Research.com Best Female Scientist Award
2022 - Research.com Best Female Scientist Award
Rosa Rademakers performs integrative study on Frontotemporal dementia and Corticobasal degeneration in her works. She combines Corticobasal degeneration and Parkinsonism in her research. In most of her Parkinsonism studies, her work intersects topics such as Disease. Disease is closely attributed to Neurodegeneration in her research. She integrates many fields in her works, including Neurodegeneration and Alzheimer's disease. In her work, she performs multidisciplinary research in Alzheimer's disease and Temporal lobe. With her scientific publications, her incorporates both Temporal lobe and Hippocampal sclerosis. She integrates Hippocampal sclerosis and Epilepsy in her research. She regularly links together related areas like Psychiatry in her Epilepsy studies.
Her study connects Amyloid precursor protein and Disease. She integrates Amyloid precursor protein and Tau protein in her research. In her study, she carries out multidisciplinary Tau protein and Tauopathy research. Rosa Rademakers combines Tauopathy and Corticobasal degeneration in her research. Rosa Rademakers combines Corticobasal degeneration and Parkinsonism in her studies. She incorporates Parkinsonism and Progressive supranuclear palsy in her research. She undertakes interdisciplinary study in the fields of Progressive supranuclear palsy and Frontotemporal lobar degeneration through her research. Rosa Rademakers performs integrative study on Frontotemporal lobar degeneration and C9orf72 in her works. By researching both C9orf72 and Frontotemporal dementia, Rosa Rademakers produces research that crosses academic boundaries.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS
Mariely DeJesus-Hernandez;Ian R. Mackenzie;Bradley F. Boeve;Adam L. Boxer.
TREM2 Variants in Alzheimer's Disease
Rita Guerreiro;Rita Guerreiro;Aleksandra Wojtas;Jose Bras;Minerva Carrasquillo.
The New England Journal of Medicine (2013)
Mutations in progranulin cause tau-negative frontotemporal dementia linked to chromosome 17
Matt Baker;Ian R. Mackenzie;Stuart M. Pickering-Brown;Jennifer Gass.
Null mutations in progranulin cause ubiquitin-positive frontotemporal dementia linked to chromosome 17q21
Marc Cruts;Ilse Gijselinck;Julie van der Zee;Sebastiaan Engelborghs.
Mutations in prion-like domains in hnRNPA2B1 and hnRNPA1 cause multisystem proteinopathy and ALS
Hong Joo Kim;Nam Chul Kim;Yong Dong Wang;Emily A. Scarborough.
Unconventional Translation of C9ORF72 GGGGCC Expansion Generates Insoluble Polypeptides Specific to c9FTD/ALS
Peter E.A. Ash;Kevin F. Bieniek;Tania F. Gendron;Thomas Caulfield.
TDP-43 and FUS in amyotrophic lateral sclerosis and frontotemporal dementia
Ian R.A. Mackenzie;Rosa Rademakers;Manuela Neumann.
Lancet Neurology (2010)
A new subtype of frontotemporal lobar degeneration with FUS pathology.
Manuela Neumann;Rosa Rademakers;Sigrun Roeber;Matt Baker.
TDP-43 A315T mutation in familial motor neuron disease.
Michael A. Gitcho;Robert H. Baloh;Sumi Chakraverty;Kevin Mayo.
Annals of Neurology (2008)
Limbic-predominant age-related TDP-43 encephalopathy (LATE): consensus working group report.
Peter T. Nelson;Dennis W. Dickson;John Q. Trojanowski;Clifford R. Jack.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: