2015 - Fellow of the American Association for the Advancement of Science (AAAS)
2011 - Fellow of the MacArthur Foundation
The scientist’s investigation covers issues in Frontotemporal dementia, Pathology, Neuroscience, Alzheimer's disease and Frontotemporal lobar degeneration. Frontotemporal dementia is a subfield of Dementia that William W. Seeley investigates. His Neuroscience study integrates concerns from other disciplines, such as Disease and Audiology.
His Alzheimer's disease research includes elements of Psychiatry, Genome-wide association study, Pediatrics and Age of onset. His Frontotemporal lobar degeneration research integrates issues from Neurofilament, Cerebrospinal fluid, Disease severity, Charged multivesicular body protein 2B and Primary progressive aphasia. His Nerve net research incorporates elements of Functional connectivity and Brain mapping.
Frontotemporal dementia, Neuroscience, Pathology, Disease and Atrophy are his primary areas of study. His Frontotemporal dementia research focuses on Alzheimer's disease and how it connects with Positron emission tomography. In Neuroscience, William W. Seeley works on issues like Neurodegeneration, which are connected to Cell biology.
Disease is a subfield of Internal medicine that William W. Seeley studies. His Atrophy study combines topics from a wide range of disciplines, such as Voxel-based morphometry, Audiology, Semantic dementia, Primary progressive aphasia and Temporal lobe. His Primary progressive aphasia research includes elements of White matter and Aphasia.
His scientific interests lie mostly in Frontotemporal dementia, Disease, Neuroscience, Pathology and Frontotemporal lobar degeneration. His Frontotemporal dementia research incorporates themes from Neurodegeneration and Atrophy. William W. Seeley interconnects Sleep in non-human animals, Gene and Clinical psychology in the investigation of issues within Disease.
The study incorporates disciplines such as Degeneration and Empathy in addition to Neuroscience. His Frontotemporal lobar degeneration study integrates concerns from other disciplines, such as Tau protein, Corticobasal degeneration and Genome-wide association study. His research investigates the link between Primary progressive aphasia and topics such as Neuroimaging that cross with problems in Default mode network.
William W. Seeley mostly deals with Neuroscience, Disease, Frontotemporal dementia, Neurodegeneration and Frontotemporal lobar degeneration. When carried out as part of a general Disease research project, his work on Amyloid is frequently linked to work in Interpersonal communication, therefore connecting diverse disciplines of study. His research in Frontotemporal dementia intersects with topics in Tau protein, Audiology and Age of onset.
His Neurodegeneration study combines topics in areas such as Haploinsufficiency, Cell biology, Atrophy and Cognitive decline. The various areas that William W. Seeley examines in his Frontotemporal lobar degeneration study include Biomarker, Progressive supranuclear palsy and Tauopathy. His work deals with themes such as C9orf72 Protein and Amyotrophic lateral sclerosis, which intersect with C9orf72.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Dissociable Intrinsic Connectivity Networks for Salience Processing and Executive Control
William W. Seeley;Vinod Menon;Alan F. Schatzberg;Jennifer Keller.
The Journal of Neuroscience (2007)
Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS
Mariely DeJesus-Hernandez;Ian R. Mackenzie;Bradley F. Boeve;Adam L. Boxer.
Neuron (2011)
Sensitivity of revised diagnostic criteria for the behavioural variant of frontotemporal dementia.
Katya Rascovsky;John R. Hodges;David Knopman;Mario F. Mendez.
Brain (2011)
Neurodegenerative Diseases Target Large-Scale Human Brain Networks
William W. Seeley;Richard K. Crawford;Juan Zhou;Bruce L. Miller.
Neuron (2009)
Primary age-related tauopathy (PART): a common pathology associated with human aging
John F. Crary;John Q. Trojanowski;Julie A. Schneider;Jose F. Abisambra.
Acta Neuropathologica (2014)
Divergent network connectivity changes in behavioural variant frontotemporal dementia and Alzheimer's disease
Juan Zhou;Michael D. Greicius;Efstathios D. Gennatas;Matthew E. Growdon.
Brain (2010)
Acetylation of Tau Inhibits Its Degradation and Contributes to Tauopathy
Sang Won Min;Seo Hyun Cho;Yungui Zhou;Sebastian Schroeder.
Neuron (2010)
Distinct tau prion strains propagate in cells and mice and define different tauopathies
David W. Sanders;Sarah K. Kaufman;Sarah L. DeVos;Apurwa M. Sharma.
Neuron (2014)
Predicting regional neurodegeneration from the healthy brain functional connectome.
Juan Zhou;Efstathios D. Gennatas;Joel H. Kramer;Bruce L. Miller.
Neuron (2012)
ApoE4 markedly exacerbates tau-mediated neurodegeneration in a mouse model of tauopathy
Yang Shi;Kaoru Yamada;Shane Antony Liddelow;Shane Antony Liddelow;Scott T. Smith.
Nature (2017)
NeuroImage: Clinical
(Impact Factor: 4.891)
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