What is he best known for?
The fields of study he is best known for:
His primary areas of study are Cell biology, Biochemistry, Presenilin, Molecular biology and Amyloid precursor protein secretase.
Christian Haass has researched Cell biology in several fields, including Parkin, Protease and Neurodegeneration.
His studies in Biochemistry integrate themes in fields like Alpha secretase and Senile plaques.
His Presenilin research includes themes of Membrane protein and Notch signaling pathway.
His biological study spans a wide range of topics, including Genetics, Mutant, Gene and Transfection.
His Amyloid precursor protein secretase study integrates concerns from other disciplines, such as Neuroscience and Ectodomain.
His most cited work include:
- Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid beta-peptide. (3630 citations)
- Amyloid β-peptide is produced by cultured cells during normal metabolism (1763 citations)
- Mutation of the β-amyloid precursor protein in familial Alzheimer's disease increases β-protein production (1595 citations)
What are the main themes of his work throughout his whole career to date?
Christian Haass spends much of his time researching Cell biology, Biochemistry, Presenilin, Pathology and Neurodegeneration.
Christian Haass interconnects Transmembrane domain, Neuroscience and Amyloid precursor protein in the investigation of issues within Cell biology.
His work investigates the relationship between Biochemistry and topics such as Amyloid precursor protein secretase that intersect with problems in Gamma secretase.
His Presenilin study combines topics in areas such as Molecular biology, Mutant and Amyloid.
His Pathology research is multidisciplinary, relying on both Genetically modified mouse and In vivo.
The Neurodegeneration study combines topics in areas such as Frontotemporal lobar degeneration, Amyotrophic lateral sclerosis, TREM2, Microglia and Cognitive decline.
He most often published in these fields:
- Cell biology (41.44%)
- Biochemistry (28.10%)
- Presenilin (22.14%)
What were the highlights of his more recent work (between 2018-2021)?
- Microglia (18.68%)
- TREM2 (18.52%)
- Pathology (21.82%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Microglia, TREM2, Pathology, Disease and Internal medicine.
His Microglia research incorporates themes from Amyloid, Phenotype, Neuroinflammation, Neuroscience and Amyloidosis.
His Amyloid research is multidisciplinary, incorporating perspectives in Alzheimer's disease and Transgene.
His research integrates issues of Receptor, Innate immune system, Neurodegeneration and Cell biology in his study of TREM2.
The concepts of his Cell biology study are interwoven with issues in Frontotemporal lobar degeneration, Neurotransmission, Peptide and Amyloid precursor protein.
Christian Haass combines subjects such as Pi and In vivo with his study of Pathology.
Between 2018 and 2021, his most popular works were:
- Loss of TREM2 function increases amyloid seeding but reduces plaque-associated ApoE. (140 citations)
- Loss of TREM2 function increases amyloid seeding but reduces plaque-associated ApoE. (140 citations)
- Early increase of CSF sTREM2 in Alzheimer’s disease is associated with tau related-neurodegeneration but not with amyloid-β pathology (63 citations)
In his most recent research, the most cited papers focused on:
Christian Haass mainly investigates Microglia, TREM2, Cell biology, Neuroscience and Receptor.
In his study, which falls under the umbrella issue of Microglia, Functional studies, Aβ deposition, Potential biomarkers, Phagocytosis and Proteome is strongly linked to Phenotype.
His TREM2 research includes elements of Loss function, Neurodegeneration, Gene silencing, Monoclonal antibody and Amyloid.
His work carried out in the field of Amyloid brings together such families of science as Endocrinology, Cerebrospinal fluid and Neuroinflammation.
His research in Cell biology is mostly focused on Protein folding.
His Genetically modified mouse research extends to the thematically linked field of Neuroscience.
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