2004 - Fellow of the Royal Society, United Kingdom
2002 - Fellow of the Royal Society of Canada Academy of Science
Peter St George-Hyslop mainly focuses on Genetics, Alzheimer's disease, Genome-wide association study, Presenilin and Cell biology. His work on Genetics is being expanded to include thematically relevant topics such as Apolipoprotein E. His Alzheimer's disease study results in a more complete grasp of Disease.
His work is dedicated to discovering how Genome-wide association study, Common disease-common variant are connected with TREM2 and other disciplines. His study in Presenilin is interdisciplinary in nature, drawing from both Mutation, Missense mutation, Endoplasmic reticulum, Biochemistry and Amyloid precursor protein. His study looks at the relationship between Cell biology and fields such as Amyloid, as well as how they intersect with chemical problems.
His primary areas of investigation include Genetics, Presenilin, Alzheimer's disease, Disease and Cell biology. His Presenilin research is multidisciplinary, relying on both Biochemistry and Amyloid precursor protein secretase, Amyloid precursor protein. The study incorporates disciplines such as Molecular biology and SORL1 in addition to Amyloid precursor protein.
His studies deal with areas such as Apolipoprotein E, Dementia and Amyloid as well as Alzheimer's disease. His research integrates issues of Neuroscience, Immunology and Bioinformatics in his study of Disease. His Genome-wide association study research incorporates themes from PICALM and Case-control study.
The scientist’s investigation covers issues in Disease, Genetics, Biophysics, Genome-wide association study and Neuroscience. His work carried out in the field of Disease brings together such families of science as Exome sequencing, SORL1 and Neurology. His Genetics study often links to related topics such as Agenesis of the corpus callosum.
His Genome-wide association study study is related to the wider topic of Single-nucleotide polymorphism. His Neuroscience research integrates issues from Alzheimer's disease, Frontotemporal dementia and Tauopathy. His research in Alzheimer's disease intersects with topics in Mutant and Cognitive decline.
Peter St George-Hyslop focuses on Genome-wide association study, Biophysics, Disease, Alzheimer's disease and Genetics. The Genome-wide association study study combines topics in areas such as SNP, Apolipoprotein E, Bipolar disorder and Genetic association. His Biophysics research includes elements of Calcium, Amyotrophic lateral sclerosis, Sequence motif, Stress granule and RNA-binding protein.
His study in the field of Dementia with Lewy bodies and Dementia also crosses realms of Psychological resilience. The various areas that Peter St George-Hyslop examines in his Alzheimer's disease study include Receptor, Neuroscience, Gene and Cognitive decline. His Genetics research is multidisciplinary, incorporating elements of Corpus callosum and Pathology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease
Jean-Charles Lambert;Jean-Charles Lambert;Jean-Charles Lambert;Carla A Ibrahim-Verbaas;Denise Harold;Adam C Naj.
Nature Genetics (2013)
Aβ peptide immunization reduces behavioural impairment and plaques in a model of Alzheimer's disease
Christopher Janus;Jacqueline Pearson;JoAnne McLaurin;Paul M. Mathews.
TREM2 Variants in Alzheimer's Disease
Rita Guerreiro;Rita Guerreiro;Aleksandra Wojtas;Jose Bras;Minerva Carrasquillo.
The New England Journal of Medicine (2013)
Common variants at MS4A4/MS4A6E , CD2AP , CD33 and EPHA1 are associated with late-onset Alzheimer's disease
Adam C. Naj;Gyungah Jun;Gary W. Beecham;Li-San Wang.
Nature Genetics (2011)
The neuronal sortilin-related receptor SORL1 is genetically associated with Alzheimer disease
Ekaterina Rogaeva;Ekaterina Rogaeva;Yan Meng;Joseph H. Lee;Yongjun Gu;Yongjun Gu.
Nature Genetics (2007)
Nicastrin modulates presenilin-mediated notch/glp-1 signal transduction and βAPP processing
Gang Yu;Masaki Nishimura;Shigeki Arawaka;Shigeki Arawaka;Diane Levitan.
Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease
Valentina Escott-Price;Céline Bellenguez;Li-San Wang;Seung-Hoan Choi.
PLOS ONE (2014)
Early-onset Amyloid Deposition and Cognitive Deficits in Transgenic Mice Expressing a Double Mutant Form of Amyloid Precursor Protein 695
M. Azhar Chishti;Dun-Shen Yang;Christopher Janus;Amie L. Phinney.
Journal of Biological Chemistry (2001)
Functional variants of OCTN cation transporter genes are associated with Crohn disease.
Vanya D Peltekova;Richard F Wintle;Laurence A Rubin;Laurence A Rubin;Christopher I Amos.
Nature Genetics (2004)
Presenilin-1 mutations downregulate the signalling pathway of the unfolded-protein response
Taiichi Katayama;Kazunori Imaizumi;Naoya Sato;Ko Miyoshi.
Nature Cell Biology (1999)
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