D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 115 Citations 68,413 570 World Ranking 2551 National Ranking 1474
Genetics D-index 111 Citations 64,747 495 World Ranking 306 National Ranking 160

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Genetics
  • Internal medicine

Philip L. De Jager mostly deals with Genetics, Genome-wide association study, Allele, Single-nucleotide polymorphism and Alzheimer's disease. His Genetics study combines topics from a wide range of disciplines, such as Disease and Immunology. He combines subjects such as Human genetics and Bioinformatics with his study of Disease.

The Genome-wide association study study combines topics in areas such as Quantitative trait locus, Expression quantitative trait loci, Genetic association, Heritability and Candidate gene. The concepts of his Allele study are interwoven with issues in Molecular genetics, Multiple sclerosis, Autoimmunity and Genotype. His work deals with themes such as PICALM, Apolipoprotein E, Case-control study and Cognitive decline, which intersect with Alzheimer's disease.

His most cited work include:

  • Integrative analysis of 111 reference human epigenomes (3776 citations)
  • Integrative analysis of 111 reference human epigenomes (3776 citations)
  • Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease (2614 citations)

What are the main themes of his work throughout his whole career to date?

Genetics, Genome-wide association study, Disease, Alzheimer's disease and Multiple sclerosis are his primary areas of study. His work on Genetics deals in particular with Gene, Single-nucleotide polymorphism, Allele, Locus and Haplotype. His Allele study combines topics in areas such as Odds ratio, Immunology and Genotype.

His Genome-wide association study study integrates concerns from other disciplines, such as Polymorphism, Quantitative trait locus, Genetic architecture, Expression quantitative trait loci and Genetic association. The study incorporates disciplines such as Computational biology, Neuroscience and Bioinformatics in addition to Disease. His Alzheimer's disease research includes themes of Apolipoprotein E, Oncology and Cognitive decline.

He most often published in these fields:

  • Genetics (75.61%)
  • Genome-wide association study (51.82%)
  • Disease (42.60%)

What were the highlights of his more recent work (between 2018-2021)?

  • Disease (42.60%)
  • Cognitive decline (27.79%)
  • Internal medicine (24.51%)

In recent papers he was focusing on the following fields of study:

Philip L. De Jager spends much of his time researching Disease, Cognitive decline, Internal medicine, Computational biology and Cognition. His biological study spans a wide range of topics, including Genome-wide association study, Single-nucleotide polymorphism, Pediatrics and Bioinformatics. His study in Genome-wide association study is interdisciplinary in nature, drawing from both Mendelian randomization and Endophenotype.

Philip L. De Jager has included themes like Multiple sclerosis, Endocrinology and Oncology in his Internal medicine study. His Computational biology research is multidisciplinary, relying on both Proteome, Transcriptome, Expression quantitative trait loci, Gene and Genetic association. His Gene study introduces a deeper knowledge of Genetics.

Between 2018 and 2021, his most popular works were:

  • Altered bile acid profile associates with cognitive impairment in Alzheimer's disease-An emerging role for gut microbiome. (119 citations)
  • Genetic architecture of subcortical brain structures in 38,851 individuals (71 citations)
  • Genetic architecture of subcortical brain structures in 38,851 individuals (71 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Genetics

Philip L. De Jager focuses on Internal medicine, Neuropathology, Oncology, Cognitive decline and Genome-wide association study. The various areas that Philip L. De Jager examines in his Neuropathology study include Genetics and Human brain. Philip L. De Jager has researched Genetics in several fields, including Inbreeding and Inbreeding depression.

His studies deal with areas such as Multiple sclerosis, Optic neuritis and Apolipoprotein E as well as Oncology. His Apolipoprotein E research is multidisciplinary, incorporating elements of Odds ratio and Allele. His work carried out in the field of Genome-wide association study brings together such families of science as Caudate nucleus, Nucleus accumbens, Putamen, Synaptic signaling and Genetic association.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Integrative analysis of 111 reference human epigenomes

Anshul Kundaje;Wouter Meuleman;Wouter Meuleman;Jason Ernst.
Nature (2015)

4957 Citations

Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease

Jean-Charles Lambert;Jean-Charles Lambert;Jean-Charles Lambert;Carla A Ibrahim-Verbaas;Denise Harold;Adam C Naj.
Nature Genetics (2013)

3630 Citations

Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis

Stephen Sawcer;Garrett Hellenthal;Matti Pirinen;Chris C. A. Spencer.
Nature (2011)

2667 Citations

Risk alleles for multiple sclerosis identified by a genomewide study.

David A. Hafler;Alastair Compston;Stephen Sawcer;Mark J. Daly.
The New England Journal of Medicine (2007)

2420 Citations

Genetics of rheumatoid arthritis contributes to biology and drug discovery

Yukinori Okada;Yukinori Okada;Di Wu;Di Wu;Di Wu;Gosia Trynka;Gosia Trynka;Towfique Raj;Towfique Raj.
Nature (2014)

2009 Citations

Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease.

Adam C. Naj;Gyungah Jun;Gary W. Beecham;Li-San Wang.
Nature Genetics (2011)

1885 Citations

Genetic and Epigenetic Fine-Mapping of Causal Autoimmune Disease Variants

Kyle Kai How Farh;Alexander Marson;Jiang Zhu;Markus Kleinewietfeld.
Nature (2015)

1617 Citations

Single-cell RNA-seq reveals new types of human blood dendritic cells, monocytes, and progenitors.

Alexandra Chloé Villani;Alexandra Chloé Villani;Rahul Satija;Rahul Satija;Gary Reynolds;Siranush Sarkizova.
Science (2017)

1560 Citations

Genome-wide association study meta-analysis identifies seven new rheumatoid arthritis risk loci

Eli A Stahl;Soumya Raychaudhuri;Soumya Raychaudhuri;Elaine F Remmers;Gang Xie.
Nature Genetics (2010)

1432 Citations

Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease.

Valentina Escott-Price;Céline Bellenguez;Li-San Wang;Seung-Hoan Choi.
PLOS ONE (2014)

1380 Citations

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